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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_2590_Библиотеки_им_академика_М_И_Перельмана

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CLIENT
TEACHING GUIDE
CERUMEN IMPACTION (EARWAX)
PROBLEM
temporary hearing loss.
CAUSE
tion and trauma. The wax is continuously being produced and removed from the ear via its own mechanism. Ears are self-cleaning organs. However, at times, an overproduction of wax may build up and remain in the external ear canal. With age, the normal mechanisms of the ear for removing earwax are decreased. This is called cerumen impaction. Use of cotton swabs to remove ear­wax can push the wax further into the ear and cause problems deeper into the ear canal.
PREVENTION/CARE
the ear canal and lead to an external ear infection and/or cause further impaction.
TREATMENT
A.
It is generally safe to clean the external ears with a wet washcloth. The external ear that is visible is the only part of the ear that
should be cleaned.
B.
In general, it is preferable to allow your healthcare provider or your otolaryngologist to clean your ears.
Activity:
Diet:
buildup of earwax in the external ear canal that may cause itching, pain, dizziness, ear ringing, aural fullness, and/or
Earwax
production is a normal, healthy process of the gland of the ear. Earwax is produced to protect the ear from infec-
not use cotton swabs, paper clips, bobby pins, or other objects to clean your ears. These can damage
PLAN
As tolerated.
As tolerated.
Medications:
You
Have Been Prescribed:
You
Need to Take:
You
Need to Notify the Office If:
A.
You are unable to hear.
B.
You have colored drainage or uid draining from your ears.
C.
You run a fever.
D.
You have dizziness.
E.
Other:
Phone:
From FAMILY PRACTICE GUIDELINES, Sixth Edition. Copyright Springer Publishing Company, LLC. All Rights Reserved.
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179
CLIENT
TEACHING GUIDE
TINNITUS
PROBLEM
CAUSE
strong association between tinnitus, stress, anxiety, and depression. Stressors include emotional, physical (comorbidities), envi­ronmental, and so forth. You do not have to have hearing loss to have tinnitus.
TREATMENT
A.
A hearing evaluation will be performed to determine if there is hearing loss.
B.
A CT or MRI may be performed to rule out rare worrisome causes of the tinnitus.
C.
Rest, exercise, and mindfulness meditation are encouraged to reduce stress, depression, and anxiety.
Activity:
Diet:
coffee, soft drinks) is encouraged.
Medications:
You
You
Tinnitus
Tinnitus
is the perception of sound heard in the ear and/or head with no external source. It may be in one or both ears.
is poorly understood and the cause is not known. It probably originates in the brain and not in the ear. There is a
PLAN
There is no activity restriction.
Some foods may make the tinnitus worse. Therefore, reducing the amount of sodium, nicotine, alcohol, and caffeine (tea,
Consider avoidance of aspirin. Aspirin may aggravate the severity of tinnitus.
Have Been Prescribed:
Need to Take:
You
Need to Notify the Office if You Have:
A.
Fever.
B.
Ear pain.
C.
Noticed a change in hearing loss or speech development.
D.
Dizziness.
E.
Any other new symptoms that occur.
F.
Other:
Phone:
From FAMILY PRACTICE GUIDELINES, Sixth Edition. Copyright Springer Publishing Company, LLC. All Rights Reserved.
https://t.me/med1917
C H A P T E R
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NASAL
Jill
C. Cash and Sarah Hendershott Taylor
ALLERGIC
RHINITIS
GUIDELINES
DEFINITION
A.
Allergic rhinitis is a chronic or recurring condition charac-
terized by nasal congestion, rhinorrhea, sneezing, pruritus of the nose, palate, and eyes, and watering and redness of the eyes. It can occur seasonally after exposure to allergens, such as tree pollen, grass pollen, and weed pollen. It can also occur perennially related to inhalants such as dust mites, molds, and pet dander. These classications can occur distinctly, or clients can have combined seasonal and perennial allergic rhinitis.
INCIDENCE
A.
Prevalence varies according to geographic region, affect-
ing 10% to 30% of children and adults in the United States and developed countries. This prevalence is increasing, mostly in urban areas. Allergic rhinitis accounts for six million missed workdays and two million missed school days per year.
PATHOGENESIS
A.
Allergic rhinitis is an immunoglobulin E (IgE)-mediated
inammation of the naso-ocular mucosa. Repeated exposure to aeroallergens leads to allergic sensitization. In subsequent exposure to the aeroallergen, IgE antibodies bind to mast cells in the respiratory epithelium and histamine is released. This results in naso-ocular inammation, leading to congestion, rhinorrhea, sneezing, and pruritus. The nasal allergic response has an immediate and late phase. The immediate phase peaks 15 to 30 minutes after allergen exposure, while the late phase peaks 6 to 12 hours after exposure.
PREDISPOSING
A.
Genetic predisposition to allergy (especially for individu-
FACTORS
als with eczema or asthma).
B.
Exposure to indoor allergens.
C.
Birth during pollen season.
D.
Maternal smoking exposure (during rst year of life).
COMMON
A. B. C. D. E. F.
OTHER
A. B.
COMPLAINTS
Nasal congestion/obstruction. Sneezing. Clear rhinorrhea. Cough from postnasal drip. Pruritus of the nose, eyes, oral mucosa, or face. Watering and redness of eyes.
SIGNS AND SYMPTOMS
Dry mouth from mouth-breathing. Snoring.
C.
Eczema.
D.
Hyposmia.
E.
Shortness of breath, difculty breathing, and wheezing.
F.
Headache.
G.
Halitosis.
H.
Fatigue.
SUBJECTIVE
A.
Onset, pattern, and duration of symptoms.
B.
Characteristics of nasal discharge.
C.
Exposure to people with similar symptoms or family his-
DATA
tory of similar symptoms.
D.
Seasonal impact on symptoms.
E.
Inquire about other diseases caused by allergens, such as
asthma, eczema, and urticaria.
F.
Rule out pregnancy.
G.
Ask about systemic medications that can induce nasal
symptoms (e.g., contraceptives, antihypertensives, erectile dysfunction drugs, and psychiatric drugs).
H.
Exposure to irritants.
I.
Prior nasal trauma.
PHYSICAL
A.
Check vitals: Make sure to include temperature as fever
EXAMINATION
would indicate an alternate etiology for the client’s symptoms.
B.
Inspect:
1.
Examine face:
a.
Note Dennie–Morgan lines (skin folds below lower
eyelids), allergic crease (“allergic salute,” transverse crease on nose from chronic rubbing), and allergic shiners (infraorbital edema and darkening from subcu­taneous vasodilation).
2.
Examine eyes:
a.
Note conjunctival injection, excess lacrimation, and
discharge.
3.
Examine ears, nose, and throat.
a.
Ears: Red, dull, bulging, perforated tympanic mem-
brane is seen with otitis media.
b.
Nose: Look for enlarged and pale (boggy) inferior
turbinates, rhinorrhea, septal deviation, septal perfora­tion, and polyps. Mucosa will appear pale (blue) and boggy with clear discharge in chronic allergy. Findings such as nasal redness, inammation, enlarged turbi­nates, and thick/discolored drainage are seen with upper respiratory infection (URI).
c.
Throat: Cobblestone appearance (“cobblestoning”)
in posterior pharynx is seen in chronic allergies. Look for tonsillar hypertrophy and postnasal drip.
The
contributions of Moya Cook to this chapter in prior editions are acknowledged here.
182
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C.
Palpate:
1.
2.
D.
Percuss:
1.
2.
E.
Auscultate:
1.
DIAGNOSTIC
A.
Diagnosis may be made from history and physical.
B.
Best test: skin prick-puncture method to detect allergen
7: NASAL GUIDELINES
Frontal and maxillary sinuses for tenderness. Head and neck for enlarged lymph nodes.
Frontal and maxillary sinuses for tenderness. Chest for consolidation.
Heart and lungs.
TESTS
sensitization.
C.
Serum tests for allergen-specic IgE antibodies (e.g., IgE
immunoassay).
DIFFERENTIAL
A.
Rhinitis caused by systemic medications.
B.
Rhinitis caused by nasal decongestant or cocaine abuse
DIAGNOSES
(“rhinitis medicamentosa”).
C.
Rhinitis of pregnancy.
D.
Rhinosinusitis.
E.
Work-related (irritant, corrosive).
F.
URI.
TREATMENT
A.
General interventions:
1.
The most effective is allergy avoidance.
2.
Implement changes in work and home environment
to reduce contact with allergens, such as removing uphol­stery and carpets to reduce reservoirs for dust mites.
3.
Nasal saline sprays and irrigation can be used to wash
allergens from nasal passages. These should be used prior to application of topical medications.
B.
Client teaching: See Client Teaching Guide for this chapter,
“Allergic Rhinitis.”
C.
Pharmaceutical therapy:
1.
Topical therapy:
a.
Topical corticosteroids are the single most effec-
tive maintenance therapy for allergic rhinitis. They have been proven more effective than oral or topical antihistamines.
i.
Fluticasone propionate (Flonase), 50 mcg/spray:
1)
Not recommended for children under
4 years.
2)
Children 4 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare daily.
ii.
Mometasone (Nasonex), 50 mcg/spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare
daily.
iii.
Beclomethasone (Beconase) 42 mcg/spray or
Qnasl 80 mcg/spray:
1)
Not recommended for children under
6 years.
Children 6 to 11 years, start with one spray
2)
BID with 42-mcg product.
3)
12 years to adult, two sprays each nare daily
with 80-mcg product.
iv.
Budesonide (Rhinocort), 32 mcg/spray:
1)
Not recommended for children under
6 years.
2)
Children 6 to 11 years, one spray each nare
BID.
3)
12 years to adult, two sprays each nare
daily.
v.
Triamcinolone (Nasacort), 55 mcg/spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 5 years, one spray each nare
daily.
3)
6 years to adult, two sprays each nare daily.
vi.
Flonase furoate (Flonase Sensimist), 27.5 mcg/
spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare
daily.
b.
Topical antihistamine:
i.
Azelastine hydrochloride (Astelin) 0.1% or
0.15% spray:
1)
Only lower strength can be used in children
6 months to 6 years, one spray per nare BID.
2)
6 to 12 years can use either strength, one
spray per nare BID.
3)
12 years to adult can be administered one to
two sprays each nare, one or two times daily.
ii.
Olopatadine (Patanase), 665 mcg/spray:
1)
Dose for children 6 to 11 years is one spray
each nare BID.
2)
Dose for 12 years to adult is two sprays each
nare BID.
c.
Other agents:
i.
Nasal saline:
1)
Nasal spray or irrigations help lavage irri-
tants from the mucosa.
2)
Saline gel can help lubricate nasal mucosa
to help prevent dryness.
ii.
Ipratropium bromide (Atrovent Nasal) 0.03%
spray:
1)
Useful in reducing rhinorrhea.
2)
6 years to adult, two sprays each nare, two
to three times daily.
iii.
Oxymetazoline (Afrin) 0.05% spray:
1)
6 years to adult, one to two sprays each
nare, one to two times daily.
2)
Must not use longer than 3 days due to
rebound congestion.
3)
Decongestants are NOT recommended as
monotherapy in chronic treatment, but can help with severe nasal congestion.
iv.
Phenylephrine (Neo-Synephrine) 1% spray or
drops:
1)
12 years to adult, two sprays/drops in each
nare Q4H.
2)
Therapy should not last longer than 3
days.
3)
Decongestant NOT recommended as
monotherapy in chronic treatment, but can help with severe nasal congestion.
2.
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Oral therapy:
a.
Antihistamines (H1 receptor antagonists) are the
preferred agent. Several may need to be tried before an effective one is found. Drugs may also need to be switched occasionally to prevent tolerance.
i.
Cetirizine (Zyrtec):
1)
6 months to 2 years, 2.5 mg daily.
2)
Children 2 to 5 years, 5 mg daily.
3)
6 years to adult, 10mg daily.
ii.
Levocetirizine (Xyzal):
1)
Children 6 to 11 years, 2.5 mg daily.
2)
12 years to adult, 5 mg daily.
iii.
Loratadine (Claritin):
1)
Children 2 to 5 years, 5 mg daily.
2)
6 years to adult, 10mg daily.
iv.
Fexofenadine (Allegra):
1)
6 months to 2 years, 15mg BID.
2)
Children 2 to 11 years, 30mg BID.
3)
12 years to adult, 180mg daily.
FOLLOW-UP
A.
Client should return for follow-up visit in 4 to 6 weeks,
earlier if symptoms worsen after 3 days of treatment.
CONSULTATION/REFERRAL
A.
Refer the client to an allergist if symptoms continue and
interfere with daily activities.
B.
The allergist may prescribe immunotherapy following
identication of offending allergens.
INDIVIDUAL
A.
Pregnancy:
1.
2.
3.
CONSIDERATIONS
Skin testing should be deferred until after delivery. Implement nonpharmacologic therapy rst. Cromolyn sodium nasal spray is rst line for mild
allergic rhinitis during pregnancy/breastfeeding.
4.
Budesonide (Rhinocort) is the only pregnancy category
B nasal corticosteroid.
5.
Immunotherapy should not be initiated during preg-
nancy but can be continued at regular dose during pregnancy.
BIBLIOGRAPHY
deShazo, R., & Kemp, S. (2021). Pharmacotherapy of allergic rhinitis. In J.
Corren (Ed.), UpToDate. Wolters Kluwer. https://www.uptodate.com /contents/pharmacotherapy-of-allergic-rhinitis?search=allergic%20r hinitis%20treatment&source=search_result&selectedTitle=1~150&us age_type=default&display_rank=1#H104439168
deShazo, R. D., & Kemp, S. F. (2020). Allergic rhinitis: Clinical
manifes-tations, epidemiology, and diagnosis. In J. Corren (Ed.), UpToDate. Wolters Kluwer. https://www.uptodate.com/contents/ allergic-rhinitis-clinical-manifestations-epidemiology-and-diagnosis# H3018812
Peden, D. (2020). An overview of rhinitis. In J. Corren (Ed.), UpToDate.
Wolters Kluwer. https://www.uptodate.com/contents/an-overview
-of-rhinitis#H2176292974
EPISTAXIS
DEFINITION
A.
Epistaxis is a nosebleed or hemorrhage from the nose. It
may be classied as anterior or posterior, depending on the source of bleeding. Up to 90% of nosebleeds are anterior and can often be managed denitely in the primary care setting.
EPISTAXIS
183
INCIDENCE
A.
Epistaxis occurs in up to 60% of the general population;
only 10% seek medical attention. Most cases occur before the age of 10 or between the age of 45 and 65.
PATHOGENESIS
A.
Epistaxis is caused by disruption of the nasal mucosa.
Up to 90% of nosebleeds occur within the vascular area of the anterior nasal septum at the site of anastomosis of three primary vessels (known as the Kiesselbach plexus). Anterior epistaxis is mostly self-limited or managed in the primary care setting. Posterior bleeds arise most commonly from the sphenopalatine artery and result in signicant hemorrhage. Posterior nosebleeds require prompt referral to ED for care by an otolaryngologist and possible nasal packing, cautery, and/ or hospital admission.
PREDISPOSING
A.
Local trauma, usually from nose-picking (digital trauma)
FACTORS
or vigorous nose-blowing.
B.
Acute inammation, often from URI, acute sinusitis, aller-
gic rhinitis, or common cold.
C.
Low moisture, causing nasal dryness and irritation.
D.
Chronic use of nasal corticosteroid spray.
E.
Inhalation of chemical irritants.
F.
Presence of foreign body.
G.
Trauma.
H.
Cocaine use.
I.
Pregnancy.
J.
Neoplasm.
K.
Systemic causes:
1.
Bleeding disorders (most common).
2.
Hypertension.
3.
Heart failure.
4.
Anticoagulation.
COMMON
A.
COMPLAINT
Common complaint is unusually severe or recurrent
nosebleeds.
OTHER
SIGNS AND SYMPTOMS
A.
Anterior epistaxis:
1.
Unilateral.
2.
Continuous, mild/moderate bleeding from nasal
septum.
B.
Posterior epistaxis:
1.
Brisk (arterial) hemorrhage.
2.
Blood owing into pharynx (indicates a more serious
problem).
SUBJECTIVE
A.
Onset (with trigger or spontaneous), frequency, and dura-
DATA
tion of bleeding.
B.
Use of oral anticoagulants, aspirin, or aspirin-containing
products (e.g., Pepto-Bismol and Excedrin).
C.
Recent or current URI, acute sinusitis, and so forth.
D.
Family history of abnormal bleeding, bleeding disorders
(e.g., hemophilia or von Willebrand disease).
E.
Recent surgery or trauma.
F.
Rule out pregnancy.
G.
Ask about possible foreign body in the nose.
H.
Ask about cocaine use or occupational exposure to irri-
tants or chemicals.
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I.
If the client has a history of nosebleeds, how did the client
7: NASAL GUIDELINES
treat previous nosebleeds? Has prior cautery or nasal packing been performed?
J.
Has the client ever been evaluated for a blood clot-
ting abnormality, such as thrombocytopenia or platelet dysfunction?
K.
Does the client complain of bruising easily, melena, or
heavy menstrual periods?
PHYSICAL
A.
Check vitals: temperature, blood pressure (check for ortho-
EXAMINATION
static hypertension), pulse, and respirations. Look for signs of airway compromise or hypovolemic shock. If nasal packing is required, take precaution and monitor the client closely for vasovagal episode during insertion of nasal packing.
B.
Inspect:
1.
Check airway patency with the client sitting and lean-
ing forward.
2.
Encourage client to expectorate clots and posterior pha-
ryngeal blood into an emesis basin; this will help reduce risk of emesis and aspiration.
3.
Observe skin, mucous membranes, and conjunctiva
for rash, pallor, purpura, petechiae, telangiectasias, or ecchymosis.
4.
The nasal cavity should be anesthetized prior to nasal
exam with topical lidocaine.
5.
Using nasal speculum, examine nose closely for sep-
tal perforation, ulcerations, or erosion. This may indicate cocaine use or can occasionally be associated with certain autoimmune diseases (such as lupus).
6.
Examine nasal discharge. Unilateral bleeding accom-
panied by purulent discharge indicates a foreign body in the nose. If the discharge is bilateral, sinusitis is also in the differential diagnosis.
7.
After bleeding has stopped:
a.
Inspect nasal mucosa; look closely at the nasal vesti-
bule, septum, and turbinates for bleeding source.
b.
Inspect nasal septum for deviation, perforation,
and/or crusting.
C.
Palpate:
1.
Check for enlarged lymph nodes or masses in the neck
to rule out sarcoidosis, tuberculosis, or malignancy.
D.
Auscultate:
1.
Heart.
2.
Lungs.
DIAGNOSTIC
A.
None required unless the client has recurrent epistaxis
TESTS
or severe blood loss.
B.
Drug screen or pregnancy test if indicated.
C.
Hematocrit and hemoglobin if bleeding is severe.
D.
Complete blood count with differential.
E.
Platelets, prothrombin time, and partial thromboplas-
tin time if bleeding disorder is suspected or if the client is anticoagulated.
F.
Sinus lms if recurrent sinus pain, tenderness, bleeding, or
if nasal mass is suspected.
DIFFERENTIAL
A.
Foreign body.
B.
Septal deviation or perforation.
C.
Nasal tumors or mass.
D.
Coagulation disorder (von Willebrand disease).
DIAGNOSES
E.
Drug-induced coagulopathy.
F.
Hypertension.
G.
Pregnancy.
PLAN
A.
General interventions:
1.
Main goal is control of bleeding or reduction of
recurrence.
B.
Client teaching: See Client Teaching Guide for this chapter,
“Nosebleeds.”
C.
Treatment:
1.
Anterior epistaxis:
a.
Have client sit and lean forward to prevent swal-
lowing of blood.
b.
Initial tamponade:
i.
Soak a cotton pledget in phenylephrine
(Neo-Synephrine) or oxymetazoline (Afrin); place this plug into bleeding nostril and hold pressure over nasal alae for 10 to 15 minutes without releas­ing pressure.
ii.
Remove and check for bleeding.
iii.
If this fails, chemical cautery may be needed.
c.
Cautery:
i.
Only do cautery if site of bleeding is visualized.
ii.
Warn client that this may be a little painful.
Anesthetize mucous membrane by applying cotton soaked with 2% lidocaine, lidocaine with epineph­rine, or 4% cocaine.
iii.
Apply silver nitrate by applying applicator tip
to the site of bleeding for no longer than 10 seconds until a white precipitate forms. While silver nitrate needs moisture to work, it will only work on a rela­tively bloodless surface.
iv.
Avoid overzealous cautery, which can lead to
ulceration and perforation.
v.
Avoid cautery of both sides of the septum,
which can lead to tissue necrosis.
vi.
If bleeding continues, repeat initial tamponade
and cautery, then place a small amount of oxidized regenerated cellulose (Surgicel) against the bleeding artery. If client continues to have high-ow epi-
staxis, nasal packing may need to be performed in emergency setting.
vii.
If bleeding stops, give client antibiotic ointment
to apply gently with ngertip or cotton swab TID for 3 days. Saline gel or Vaseline can also be used.
2.
Posterior epistaxis:
a.
Have client sit and lean forward to prevent swal-
lowing of blood.
b.
As it can be difcult to determine the source of epi-
staxis, try to control bleeding rst with the previous steps of topical vasoconstrictor and anesthetic and then apply pressure.
c.
Consult a physician. The client needs ED care
immediately due to rapid blood loss.
d.
Monitor vital signs (blood pressure and pulse);
order hematocrit; blood type and crossmatch may be needed.
FOLLOW-UP
A.
Anterior epistaxis: as needed.
B.
Posterior epistaxis: need admission to hospital and referral
to an otolaryngologist.
NONALLERGIC RHINITIS
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CONSULTATION/REFERRAL
A.
Anterior epistaxis: Referral to an otolaryngologist is rec-
ommended for unsuccessful cessation of hemorrhage or per­sistent, recurrent epistaxis despite interventions.
B.
Posterior epistaxis: Refer to a physician and/or otolaryn-
gologist immediately.
INDIVIDUAL
A.
Pregnancy:
1.
CONSIDERATIONS
Nosebleeds are common, possibly due to hyperemia of
the nasal tissue.
2.
Suggest use of saline spray to keep the mucous mem-
branes moist throughout the day.
3.
Use Vaseline or saline gel applied gently with ngertip
or cotton-tipped applicator before bed; use humidier in the bedroom.
B.
Pediatrics:
1.
Nosebleeds commonly occur due to trauma from
nose-picking or rubbing. Nosebleeds in children are rarely severe.
2.
Advise parents to keep children’s ngernails short.
3.
Apply saline gel or spray (avoid Vaseline in children)
to rims of nostrils to maintain mucosal moisture.
4.
If child is using topical corticosteroid, it should be
discontinued.
C.
Geriatrics:
1.
Monitor older adults clients very closely for airway
compromise or hypovolemic shock.
2.
Anticoagulated clients are at high risk of nosebleeds.
3.
Suggest use of saline spray to keep the mucous mem-
branes moist throughout the day.
4.
Use Vaseline or saline gel applied gently with ngertip
or cotton-tipped applicator before bed; use humidier in bedroom.
BIBLIOGRAPHY
Alter, H. (2021). Approach to the adult with epistaxis. In A. B. Wolfson&
D. G. Deschler (Eds.), UpToDate. Wolters Kluwer. https://www.uptod ate.com/contents/approach-to-the-adult-with-epistaxis?search=epist axis&source=search_result&selectedTitle=1~150&usage_type=default &display_rank=1#H18
Lockwood, C. J., & Magriples, U. (2021). Prenatal care: Patient education,
health promotion, and safety of commonly used drugs. In V. Berghella (Ed.), UpToDate. Wolters Kluwer. https://www.uptodate.com/cont ents/prenatal-care-patient-education-health-promotion-and-safety­of-commonly-used-drugs?search=epistaxis%20in%20pregnancy&sou rce=search_result&selectedTitle=1~150&usage_type=default&display _rank=1
Messner, A. H. (2020). Management of epistaxis in children. In A. M.
Stack& G. C. Isaacson (Eds.), UpToDate. Wolters Kluwer. https:// www.uptodate.com/contents/management-of-epistaxis-in­children?search=epistaxis%20in%20pediatrics&source=search_result &selectedTitle=1~150&usage_type=default&display_rank=1
NONALLERGIC
RHINITIS
DEFINITION
A.
Nonallergic rhinitis is rhinitis without an identiable eti-
ology and without an immunoglobulin E-dependent com­ponent. It is characterized by nasal congestion, postnasal drainage, rhinorrhea, and sneezing. It is associated with some degree of autonomic dysregulation. It is classied in various ways: idiopathic, vasomotor, atrophic, geriatric, drug-induced, gustatory, occupational, or rhinitis of pregnancy.
INCIDENCE
A.
Nonallergic rhinitis may be responsible for 17% to 52% of
all rhinitis cases in adults.
PATHOGENESIS
A.
No single theory of pathogenesis for chronic nonallergic
rhinitis has been determined, but it can be divided into nonin-
ammatory and inammatory. It is a diagnosis of exclusion.
B.
Clients exhibit clinical hypersensitivity of nasal mucosa to
certain odors and irritants.
C.
Atrophic and geriatric rhinitis results from progressive
atrophy of nasal mucosa.
D.
Overuse of topical nasal decongestants or cocaine and
some systemic medications can result in nasal dysfunction.
E.
Rhinitis of pregnancy results from hormonal changes;
symptoms abate with delivery.
PREDISPOSING
A.
Age: onset of nonallergic rhinitis usually after the age
FACTORS
of 20 (whereas allergic rhinitis usually presents before 20 years).
B.
Abrupt changes in temperature, odors, and emotional stress.
C.
Other predisposing factors depending on type (e.g., over-
use of medications, side effects of systemic medications, and occupational environment).
COMMON
A. B. C. D. E.
COMPLAINTS
Nasal congestion. Postnasal drainage. Rhinorrhea. Sneezing. Perennial symptoms, often exacerbated by weather
conditions.
SUBJECTIVE
A.
Clinical history is the most important tool used to make
DATA
distinction between allergic and nonallergic rhinitis.
B.
Ask about the onset, duration, and pattern of symptoms.
C.
Inquire about the characteristics of nasal discharge and if it
is bilateral or unilateral.
D.
Ask about potential triggers, including cigarette smoke,
strong scents, or fragrances.
E.
Ask about exposure to other people with similar symptoms.
F.
Inquire about seasonal or weather impact on symptoms,
previous treatments, and results.
G.
Rule out pregnancy.
H.
Ask about use of prescription drugs, over-the-counter
(OTC) drugs (Afrin), and illicit drugs (cocaine).
I.
Review medical history for other respiratory problems,
most importantly asthma.
J.
Pediatric consideration: Determine possibility of a foreign
object in the nostrils.
PHYSICAL
A.
Check vitals: temperature and blood pressure.
B.
Inspect:
EXAMINATION
1.
Examine general appearance.
2.
Examine eyes.
a.
Inspect conjunctivae for “allergic shiners” (dark
circles under eyes), tearing, and eyelid swelling. This would indicate possible allergic rhinitis; nonallergic rhinitis is NOT associated with ocular symptoms.
3.
Examine the ears, nose, and throat.
a.
Inspect ears for signs of otitis media (red, bulging, per-
forated tympanic membrane, and purulent drainage).
b.
Inspect nose for redness, swelling, polyps (soft,
pedunculated, nontender, pale, smooth structures), enlarged turbinates, foreign objects, septal deviation, septal perforation, ischemia, mucosal injury, atrophy,
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7: NASAL GUIDELINES
and “cobblestoned” pharyngeal mucosa. Nasal turbi­nates can appear boggy and edematous in both aller­gic and nonallergic rhinitis. In nonallergic rhinitis, the mucosa is often more erythematous compared with pale blue in allergic rhinitis. However, none of these
ndings are diagnostic and the mucosa may appear relatively normal in nonallergic rhinitis.
C.
Palpate:
1.
Head and neck.
D.
Percuss:
1.
Sinuses.
E.
Auscultate:
1.
Heart.
2.
Lungs.
DIAGNOSTIC
A.
Nonallergic rhinitis is a diagnosis of exclusion. Diagnosis
TESTS
is based on characteristic history and physical exam, as well as absence of clinical allergy to aeroallergens.
B.
Skin testing for allergies may be done to exclude allergic
rhinitis.
DIFFERENTIAL
A.
Allergic rhinitis.
B.
Upper respiratory infection.
C.
Foreign body. D. E.
Tumor. F.
Deviated septum. G.
Nasal polyps. H.
Endocrine conditions, such as hypothyroidism and
DIAGNOSES
pregnancy.
I.
Drug use: oral contraceptives, aspirin, alpha-adrenergic
blockers, cocaine, and nasal decongestant overuse (rhinitis medicamentosa).
J.
Ciliary dysfunction syndrome. K.
Cerebral spinal uid leak. L.
Laryngopharyngeal reux.
PLAN
A.
General interventions:
1.
Identify triggers and address alleviating triggers with
avoidance/minimization.
B.
Client teaching:
1.
Teach the client the signicance of individual triggers
for nonallergic rhinitis. Encourage use of a journal to learn personal triggers.
2.
Avoid triggers such as smoking, smoke-lled rooms,
wood-burning stoves/replaces, sprays, and perfumes.
3.
Other triggers may include weather changes, hor-
monal changes, and medications.
4.
Teach methods of treatment and identify treatments
that work best for the client.
5.
Encourage use of saline irrigation daily to cleanse nasal
and sinus mucosa. Cleansing the mucosa daily will help remove foreign materials inhaled and will also help with tis­sue edema. Clean device after each use and allow to air-dry.
C.
Pharmaceutical therapy:
1.
Monotherapy versus combination therapy:
Clients with mild disease may be adequately treated
a.
with intranasal corticosteroid or intranasal antihista­mine spray. For clients who are more symptomatic, clinical evidence has shown combination therapy to be more effective, with both daily intranasal corticoste­roid and antihistamine spray.
b.
Topical corticosteroids:
i.
Fluticasone propionate (Flonase), 50 mcg/spray:
1)
Not recommended for children under 4 years.
2)
Children 4 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare
daily.
ii.
Mometasone (Nasonex), 50 mcg/spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare
daily.
iii.
Beclomethasone (Beconase) 42 mcg/spray or
Qnasl 80 mcg/spray:
1)
Not recommended for children under
6 years.
2)
Children 6 to 11 years, start with one spray
BID with 42-mcg product.
3)
12 years to adult, two sprays each nare daily
with 80-mcg product.
iv.
Budesonide (Rhinocort), 32 mcg/spray:
1)
Not recommended for children under
6 years.
2)
Children 6 to 11 years, one spray each nare
BID.
3)
12 years to adult, two sprays each nare
daily.
v.
Triamcinolone (Nasacort), 55 mcg/spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 5 years, one spray each nare
daily.
3)
6 years to adult, two sprays each nare daily.
vi.
Flonase furoate (Flonase Sensimist), 27.5 mcg/
spray:
1)
Not recommended for children under
2 years.
2)
Children 2 to 11 years, one spray each nare
daily.
3)
12 years to adult, two sprays each nare daily.
c.
Topical antihistamine:
i.
Azelastine hydrochloride (Astelin) 0.1% or
0.15% spray:
1)
Only lower strength can be used
in children 6 months to 6 years, one spray per nare BID.
2)
Children 6 to 12 years can use either strength,
one spray per nare BID.
3)
12 years to adult, one to two sprays each
nare, one to two times daily.
ii.
Olopatadine (Patanase), 665 mcg/spray:
1)
Dose for children 6 to 11 years is one spray
each nare BID.
2)
Dose for 12 years to adult is two sprays each
nare BID.
2.
If rhinorrhea is the dominant or only symptom (such
as in gustatory rhinitis or rhinitis of older adults), recom­mend anticholinergic nasal spray.
a.
Ipratropium (Atrovent) 0.03% or 0.06%:
i.
Not recommended for children under 6 years.
ii.
6 years to adult, two sprays each nare, two to
three times daily.
3.
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Oral decongestants:
a.
Oral decongestants can be added to the treatment
regimen, with dosing of pseudoephedrine 30 or 60mg PO up to TID on symptomatic days. Do not use in cli­ents with prostatic hyperplasia or hypertension.
FOLLOW-UP
A.
Clients receiving chronic therapy for rhinitis should be seen
every 3 to 12 months, depending on severity of symptoms.
CONSULTATION/REFERRAL
A.
Consult with a physician if symptoms continue despite
treatment.
B.
If treatment fails, refer the client to an allergist for testing,
and/or an ear, nose, and throat specialist to consider surgical approaches for clients with severe, chronic nonallergic rhinitis.
INDIVIDUAL
A.
Pregnancy:
1.
CONSIDERATIONS
Reassure pregnant clients that rhinitis is a common
hormonal response. Nonallergic rhinitis is not contagious and cannot cross the placenta.
BIBLIOGRAPHY
Lieberman, P. L. (2020). Chronic nonallergic rhinitis. In J. Corren (Ed.),
UpToDate. Wolters Kluwer. https://www.uptodate.com/contents/ chronic-nonallergic-rhinitis?search=nonallergic%20rhinitis&source=se arch_result&selectedTitle=1~63&usage_type=default&display_rank= 1#H19
Peden, D. (2020). An overview of rhinitis. In J. Corren (Ed.), UpToDate.
Wolters Kluwer. https://www.uptodate.com/contents/an-overview
-of-rhinitis?search=nonallergic%20rhinitis&source=search_result&sel ectedTitle=2~63&usage_type=default&display_rank=2
SINUSITIS
DEFINITION
A.
Sinusitis (often referred to as rhinosinusitis) is the symp-
tomatic inammation of the mucous membrane lining of one or more paranasal sinuses. It may be acute, subacute, or chronic. Diagnosis is based on subjective and objective nd­ings and depends on duration of symptoms.
1.
Acute sinusitis: symptoms lasting up to 4 weeks.
2.
Subacute sinusitis: symptoms lasting 4 to 12 weeks.
3.
Chronic sinusitis: symptoms lasting longer than 12 weeks.
INCIDENCE
A.
Sinusitis is very prevalent, affecting millions of people in the
United States each year. It is estimated that one in seven indi­viduals in the United States will experience an episode of acute sinusitis each year. Incidence is higher in females, and among all adults it is highest in those aged 45 to 64 years. Chronic sinusitis affects between 5% and 12% of the general population.
PATHOGENESIS
A.
The etiology of acute rhinosinusitis involves factors such
as environmental, anatomic, and systemic. These factors can occur at the same time and predispose clients not only to acute sinusitis but also to the development of chronic sinusitis.
B.
The most common cause of acute sinusitis is viral infection,
with the most common causes being rhinovirus, inuenza virus, and parainuenza virus. Acute bacterial rhinosinusitis occurs in 0.5% to 2% of all sinusitis episodes. The most common bacterial organisms are Streptococcus pneumoniae, Haemophilus inuenzae, and Moraxella catarrhalis. If acute sinusitis infections are not resolved or become recurrent, Staphylococcus aureus or Pseudomonas aeruginosa can become dominant.
SINUSITIS
C.
The transition from viral to bacterial has been studied exten-
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sively and it is thought to be secondary to decreased muco­ciliary clearance and consequent colonization of bacteria on retained mucus secretion from the obstruction of outow. The sinonasal mucosa inammation from the triggering agent stuns the cilia and mucus remains trapped in the sinuses, and bacte­ria proliferate. Anatomic abnormalities can also interfere with normal mucociliary clearance and can prolong illness.
D.
Other causes of acute sinusitis are fungi, dental abscess,
and autoimmune reactions. Fungal sinusitis tends to occur in immunosuppressed clients.
PREDISPOSING
A.
Recent upper respiratory infection (URI).
B.
Allergic rhinitis.
C.
Cigarette smoke (rsthand or secondhand).
D.
Air pollutant exposure.
E.
Sinonasal anatomy (septal deviation, septal spurs, tur-
FACTORS
binate hypertrophy, Haller cells, adenoidal hypertrophy, or obstructive mass).
F.
Ciliary dysfunction (as in Kartagener syndrome and cystic
brosis).
G.
Dental anatomy/abscess.
H.
Asthma.
I.
Neoplasms.
J.
Immunodeciency (often associated with recurrent acute
or chronic sinusitis).
K.
Trauma.
L.
Diving and swimming.
M.
Flying or rapid changes in altitude.
COMMON
A. B. C. D. E.
OTHER
A. B. C. D. E. F. G.
POTENTIAL
A.
COMPLAINTS
Mucopurulent rhinorrhea. Headache and/or facial pain. Dental or ear pain. Loss of smell (anosmia). Nasal congestion.
SIGNS AND SYMPTOMS
Fever (mostly associated with acute sinusitis). Malaise or fatigue. Cough. Periorbital edema. Halitosis. Snoring and mouth-breathing. Hyponasal speech.
COMPLICATIONS
The following requires immediate ear, nose, and throat
referral.
1.
Meningitis (symptoms are increased fever and stiff
neck).
2.
Preseptal or orbital cellulitis.
3.
Subperiosteal or intracranial abscess.
4.
Epidural abscess.
5.
Cavernous sinus thrombosis (acute thrombophlebitis
due to infection in the area where veins drain into the cav­ernous sinus).
6.
Osteomyelitis of the sinus bones.
SUBJECTIVE
A.
Ask about onset, duration, and course of symptoms. Have
DATA
symptoms lasted longer than 10 days or longer than 12 weeks?
B.
Inquire whether seasons affect symptoms.
C.
Ask the client about recent URI and how it was treated.
Did the client receive antibiotics or steroids? Did the client n­ish the full course? Any relief from symptoms?