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21 Extracorporeal Membrane Oxygenation
https://t.me/med1917
35. Sohn N, Marcoux J, Mycyk T, Krahn J, Meng Q.The impact of different biocompatible coated
cardiopulmonary bypass circuits on inammatory response and oxidative stress. Perfusion.
2009;24:231–7. https://doi.org/10.1177/0267659109351218.
36. Hirsh J, O'Donnell M, Weitz JI. New anticoagulants. Blood. 2005;105:453–63. https://doi.
org/10.1182/blood- 2003- 12- 4195.
37. DeLoughery EP, Olson SR, Puy C, McCarty OJT, Shatzel JJ.The safety and efcacy of novel
agents targeting factors XI and XII in early phase human trials. Semin Thromb Hemost.
2019;45:502–8.
38. Wallisch M, Lorentz CU, Lakshmanan HHS, Johnson J, Carris MR, Puy C, Gailani D, Hinds
MT, McCarty OJT, Gruber A, etal. Antibody inhibition of contact factor XII reduces platelet
deposition in a model of extracorporeal membrane oxygenator perfusion in nonhuman primates. Res Pract Thromb Haemost. 2020;4:205–16. https://doi.org/10.1002/rth2.12309.
39. Bembea MM, Annich G, Rycus P, Oldenburg G, Berkowitz I, Pronovost P. Variability in
anticoagulation management of patients on extracorporeal membrane oxygenation: an
international survey. Pediatr Crit Care Med. 2013;14:e77–84. https://doi.org/10.1097/
PCC.0b013e31827127e4.
40. Rajsic S, Breitkopf R, Bachler M, Treml B.Diagnostic modalities in critical care: point-ofcare approach. Diagnostics (Basel). 2021;11:2202.
41. Goldberg RJ, Spencer FA, Gore JM, Lessard D, Yarzebski J.Thirty-year trends (1975 to 2005)
in the magnitude of, management of, and hospital death rates associated with cardiogenic
shock in patients with acute myocardial infarction: a population-based perspective. Circulation.
2009;119:1211–9. https://doi.org/10.1161/circulationaha.108.814947.
42. Napp LC, Kühn C, Bauersachs J. ECMO in cardiac arrest and cardiogenic shock. Herz.
2017;42:27–44.
43. Sayer GT, Baker JN, Parks KA.Heart rescue: the role of mechanical circulatory support in the
management of severe refractory cardiogenic shock. Curr Opin Crit Care. 2012;18:409–16.
https://doi.org/10.1097/MCC.0b013e328357f1e6.
44. Wilson-Smith AR, Bogdanova Y, Roydhouse S, Phan K, Tian DH, Yan TD, Loforte
A.Outcomes of venoarterial extracorporeal membrane oxygenation for refractory cardiogenic
shock: systematic review and meta-analysis. Ann Cardiothorac Surg. 2019;8:1–8. https://doi.
org/10.21037/acs.2018.11.09.
45. Cheng R, Hachamovitch R, Kittleson M, Patel J, Arabia F, Moriguchi J, Esmailian F, Azarbal
B.Complications of extracorporeal membrane oxygenation for treatment of cardiogenic shock
and cardiac arrest: a meta-analysis of 1,866 adult patients. Ann Thorac Surg. 2014;97:610–6.
https://doi.org/10.1016/j.athoracsur.2013.09.008.
46. Chen Y-C, Tsai F-C, Fang J-T, Yang C-W. Acute kidney injury in adults receiving extracorporeal membrane oxygenation. J Formos Med Assoc. 2014;113:778–85. https://doi.
org/10.1016/j.jfma.2014.04.006.
47. Villa G, Katz N, Ronco C.Extracorporeal membrane oxygenation and the kidney. Cardiorenal
Med. 2015;6:50–60. https://doi.org/10.1159/000439444.
48. Fisser C, Reichenbächer C, Müller T, Schneckenpointner R, Malfertheiner MV, Philipp
A, Foltan M, Lunz D, Zeman F, Lubnow M.Incidence and risk factors for cannula-related
venous thrombosis after venovenous extracorporeal membrane oxygenation in adult patients
with acute respiratory failure. Crit Care Med. 2019;47:e332–9. https://doi.org/10.1097/
ccm.0000000000003650.
49. Sohail S, Fan E, Foroutan F, Ross HJ, Billia F, Alba AC.Predictors of mortality in patients
treated with veno-arterial ECMO for cardiogenic shock complicating acute myocardial infarction: a systematic review and meta-analysis. J Cardiovasc Transl Res. 2022;15:227–38. https://
doi.org/10.1007/s12265- 021- 10140- w.
50. Connors JM, Levy JHJB.COVID-19 and its implications for thrombosis and anticoagulation.
Blood. 2020;135:2033–40.
https://doi.org/10.1055/s- 0039- 1692439.
https://doi.org/10.1007/s00059- 016- 4523- 4.
351

352
https://t.me/med1917
51. Lim MS, Mcrae S. COVID-19 and immunothrombosis: pathophysiology and therapeutic
implications. Crit Rev Oncol Hematol. 2021;168:103529.
52. Ranucci M, Ballotta A, Di Dedda U, Baryshnikova E, Dei Poli M, Resta M, Falco M, Albano
G, Menicanti L.The procoagulant pattern of patients with COVID-19 acute respiratory distress syndrome. J Thromb Haemost. 2020;18:1747–51. https://doi.org/10.1111/jth.14854.
53. Weir-McCall JR, Galea G, Mun Mak S, Joshi K, Agrawal B, Screaton N, Toshner M, Ruggiero
A, Benedetti G, Brozik J, etal. Vascular thrombosis in severe COVID-19 requiring extracorporeal membrane oxygenation: a multicenter study. Crit Care Med. 2022;50:624–32. https://
doi.org/10.1097/ccm.0000000000005322.
54. Yusuff H, Zochios V, Brodie D.Thrombosis and coagulopathy in COVID-19 patients requiring extracorporeal membrane oxygenation. ASAIO J. 2020;66:844–6. https://doi.org/10.1097/
mat.0000000000001208.
55. Peek GJ, Mugford M, Tiruvoipati R, Wilson A, Allen E, Thalanany MM, Hibbert CL,
Truesdale A, Clemens F, Cooper N, etal. Efcacy and economic assessment of conventional
ventilatory support versus extracorporeal membrane oxygenation for severe adult respiratory failure (CESAR): a multicentre randomised controlled trial. Lancet (London, England).
2009;374:1351–63.
56. Pappalardo F, Pieri M, Arnaez Corada B, Ajello S, Melisurgo G, De Bonis M, Zangrillo
A. Timing and strategy for weaning from Venoarterial ECMO are complex issues. J
Cardiothorac Vasc Anesth. 2015;29:906–11. https://doi.org/10.1053/j.jvca.2014.12.011.
57. Aissaoui N, Luyt CE, Leprince P, Trouillet JL, Léger P, Pavie A, Diebold B, Chastre J, Combes
A. Predictors of successful extracorporeal membrane oxygenation (ECMO) weaning after
assistance for refractory cardiogenic shock. Intensive Care Med. 2011;37:1738–45. https://
doi.org/10.1007/s00134- 011- 2358- 2.
58. Cavarocchi NC, Pitcher HT, Yang Q, Karbowski P, Miessau J, Hastings HM, Hirose H.Weaning
of extracorporeal membrane oxygenation using continuous hemodynamic transesophageal
echocardiography. J Thorac Cardiovasc Surg. 2013;146:1474–9. https://doi.org/10.1016/j.
jtcvs.2013.06.055.
59. Aissaoui N, El-Banayosy A, Combes A.How to wean a patient from veno-arterial extracorporeal membrane oxygenation. Intensive Care Med. 2015;41:902–5. https://doi.org/10.1007/
s00134- 015- 3663- y.
60. Santise G, Panarello G, Ruperto C, Turrisi M, Pilato G, Giunta A, Sciacca S, Pilato
M.Extracorporeal membrane oxygenation for graft failure after heart transplantation: a multidisciplinary approach to maximize weaning rate. Int J Artif Organs. 2014;37:706–14. https://
doi.org/10.5301/ijao.5000353.
61. Ortuno S, Delmas C, Diehl JL, Bailleul C, Lancelot A, Naili M, Cholley B, Pirracchio R,
Aissaoui N.Weaning from veno-arterial extra-corporeal membrane oxygenation: which strategy to use? Ann Cardiothorac Surg. 2019;8:E1–e8. https://doi.org/10.21037/acs.2018.08.05.
62. Rajsic S, Breitkopf R, Treml B, Jadzic D, Oberleitner C, Oezpeker UC, Innerhofer N,
Bukumiric Z.Association of aPTT-guided anticoagulation monitoring with thromboembolic
events in patients receiving V-A ECMO support: a systematic review and meta-analysis. J Clin
Med. 2023;12:3224.
63. Rajsic S, Treml B, Jadzic D, Breitkopf R, Oberleitner C, Bachler M, Bösch J, Bukumiric
Z. aPTT-guided anticoagulation monitoring during ECMO support: a systematic review and
meta-analysis. J Crit Care. 2023;77:154332. https://doi.org/10.1016/j.jcrc.2023.154332.
64. Guervil DJ, Rosenberg AF, Winterstein AG, Harris NS, Johns TE, Zumberg MS.Activated
partial thromboplastin time versus antifactor Xa heparin assay in monitoring unfractionated
heparin by continuous intravenous infusion. Ann Pharmacother. 2011;45:861–8. https://doi.
org/10.1345/aph.1Q161.
https://doi.org/10.1016/s0140- 6736(09)61069- 2.
S. Rajsic et al.

21 Extracorporeal Membrane Oxygenation
https://t.me/med1917
65. Swayngim R, Preslaski C, Burlew CC, Beyer J.Comparison of clinical outcomes using activated partial thromboplastin time versus antifactor-Xa for monitoring therapeutic unfractionated heparin: a systematic review and meta-analysis. Thromb Res. 2021;208:18–25. https://
doi.org/10.1016/j.thromres.2021.10.010.
66. Willems A, Roeleveld PP, Labarinas S, Cyrus JW, Muszynski JA, Nellis ME, Karam
O. Anti-Xa versus time-guided anticoagulation strategies in extracorporeal membrane
oxygenation: a systematic review and meta-analysis. Perfusion. 2021;36:501–12. https://doi.
org/10.1177/0267659120952982.
67. Zhang M, Pauls JP, Bartnikowski N, Haymet AB, Chan CHH, Suen JY, Schneider B, Ki KK,
Whittaker AK, Dargusch MS, et al. Anti-thrombogenic surface coatings for extracorporeal
membrane oxygenation: a narrative review. ACS Biomater Sci Eng. 2021;7:4402–19. https://
doi.org/10.1021/acsbiomaterials.1c00758.
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Part VI
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Perioperative Surgical Challenges

Chapter 22
https://t.me/med1917
Damage Control Laparotomy
DavidRayVelez andAnthonyJosephDuncan
Abbreviations
ATLS Advanced Trauma Life Support
BAT Blunt abdominal trauma
CT Computerized tomography
DCL Damage control laparotomy
DPL Diagnostic peritoneal lavage
FAST Focused Assessment with Sonography for Trauma
NPWT Negative pressure wound therapy
PAT Penetrating abdominal trauma
REBOA Resuscitative Endovascular Balloon Occlusion of the Aorta
Introduction
History of the traumatic abdomen extends through antiquity. Although management
has evolved over time, the traumatic abdomen in a crashing patient can still, as
described by Hippocrates, “mortify imperatively.”
Damage control is not a novel modality for therapeutic interventions. The term
itself comes from the US Navy and refers to the ability of a ship to undergo damage
D. R. Velez (*)
Department of Surgery, University of Nevada, Las Vegas, Las Vegas, NV, USA
e-mail: david.velez@unlv.edu
A. J. Duncan
University of North Dakota, Grand Forks, ND, USA
e-mail: Anthony.Duncan@ndus.edu
Switzerland AG 2024
J. Faintuch, S. Faintuch (eds.), Recent Strategies in High Risk Surgery,
https://doi.org/10.1007/978-3-031-56270-9_22
357© The Author(s), under exclusive license to Springer Nature

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but still maintain mission integrity. The term was brought into the mainstream surgical community in 1993, when Rotondo etal. proposed it as an approach for managing penetrating abdominal injuries [1]. Before the adoption of damage control as a
therapeutic intervention, several methodologies were implemented to reduce surgical time at the index procedure and improve outcomes of critically ill patients.
These included peri-hepatic packing and Feliciano etal.’s rapid conservative operative techniques, which were published in 1993 [2, 3].
Damage control surgery is a surgical technique that involves a brief initial operation aimed at quickly controlling severe bleeding and/or contamination in critically
ill patients [4]. Following the initial surgery, patients are admitted to the intensive
care unit where their condition is stabilized with the objective of returning them to
their pre-injury state before denitive repair can be carried out [4]. While advancements in technology have led to signicant improvements in this technique over the
years, the fundamental principles of damage control surgery have remained
unchanged.
D. R. Velez and A. J. Duncan
Epidemiology andEtiology
There are 5.8million traumatic deaths every year across the globe [5]. Trauma is the
third leading cause of death in the United States with 150,000 deaths per year [6].
Trauma is largely a disease of young males with over 50% between the ages of 10
and 24years and over 66% male [7]. Trauma is the most common cause of death
between ages 1 and 44years [8].
The most common causes of traumatic death in the United States are motor vehicle collision (37,991 deaths/year), falls (37,455 deaths/year), and gunshot wounds
with suicide (24,432 deaths/year) accounting for almost twice that of homicide
(13,958 deaths/year) [9]. Blunt abdominal trauma (BAT) accounts for 90% of
patients presenting but only 67% of those requiring laparotomy [10]. Penetrating
abdominal trauma (PAT) accounts for only 10% of patients presenting but 33% of
those requiring laparotomy [9, 10]. Of penetrating mechanisms, stab wounds are the
most common, although gunshot wounds are approximately eight times more
lethal [11].
Among patients with injury due to BAT, the most commonly injured organs are
the spleen (40–55%), liver (35–45%), and small bowel (5–10%) [9]. The most common injuries after PAT are small bowel (50%), colon (40%), liver (30%), and vascular injury (25%) [9]. After the decision is made to proceed with trauma laparotomy,
the most common procedures performed are enterectomy (23%), hepatorrhaphy
(20%), enterorrhaphy (16%), splenectomy (16%), and colectomy (15%) [10].
Damage control procedures are used in 38% of cases [10]. Overall mortality is 21%
which doubles to 46% in hypotensive patients [10]. Surprisingly, despite major
advancements in the eld of trauma, these high mortality rates have seen minimal
change over the last two decades [10].

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359
General Management ofAbdominal Trauma
As with any trauma, abdominal trauma begins with a thorough primary and secondary survey [12]. Advanced Trauma Life Support (ATLS) algorithms start with the
management of airway, breathing, circulation, disability, exposure, and then a headto- toe injury inventory and exam [12]. Diagnosis then diverges depending on the
patient’s condition and mechanism of injury.
All patients with overt peritonitis or evisceration mandate trauma laparotomy [6,
13]. Regarding patients without peritonitis or evisceration, management will then
differ by the mechanism of injury. In an unstable patient after PAT, a trauma laparotomy is mandated. In an unstable patient after BAT, the abdomen must rst be
conrmed as the source of instability by a Focused Assessment with Sonography
for Trauma (FAST) prior to laparotomy. Some surgeons do elect for diagnostic peritoneal lavage (DPL), although it has almost entirely been replaced since the advent
of FAST and is now primarily used when FAST results are equivocal. A positive
FAST requires trauma laparotomy but a negative FAST should instead prompt further evaluation for other sources of instability with chest X-ray or computerized
tomography (CT) scan as indicated. A stable patient allows more time for diagnosis
in a comprehensive manner. As such, a stable patient after BAT should be evaluated
by CT scan. A stable patient after PAT should undergo local wound exploration.
The primary goal of local wound exploration is to evaluate for violation of posterior fascia, not peritoneum. If no fascial violation is found in a PAT that is stable
with no other indication for admission, the patient may be discharged home [13].
Fascial violation requires further evaluation to determine peritoneal violation or
damage to intrabdominal structures. This can be accomplished by CT scan, FAST,
DPL, or diagnostic laparoscopy dependent on surgeon preference. A patient without
any evidence of peritoneal violation should be monitored for 24h with serial clinical exams. Peritoneal violation mandates surgical evaluation with either laparotomy
or diagnostic laparoscopy. Of note, when evaluating anterior abdominal stab
wounds, only 50–75% will penetrate the peritoneum and of those, only 50–75% will
require repair of intraabdominal injury [13].
Trauma Laparotomy: ASystematic Approach
Upon arrival in the operating room, skin should be prepped from chin to knees with
arms extended allowing adequate access for the worst-case scenarios. Understand
that sterility is a luxury in hemorrhagic shock. Rather than disappearing for a prolonged relaxing scrub, if necessary the surgeon should throw on gloves and dive-in
if needed to save the patient’s life.

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The trauma laparotomy “Operative Sequence” was outlined by Hirshberg and
Mattox in the book “Top Knife” published two decades ago in 2004 [14]. It follows
four basic steps: access and exposure, temporary bleeding control, exploration, and
then decision of denitive repair versus damage control [14]. There have been many
descriptions of how to approach a trauma laparotomy, and although they do not
always use the same terminology, they all at their core follow the same format
[13–15].
D. R. Velez and A. J. Duncan
Access andExposure
The rst steps of an efcient trauma laparotomy are access and exposure to facilitate the ensuing procedure. The typical access is through a long midline incision
from xiphoid to pubis. However, every effort should be made to enter through virgin
territory to avoid scars and internal adhesions. This may be accomplished by extending the incision superiorly or inferiorly if able. If unable, an alternative incision can
be chosen such as the chevron incision which resembles the military chevron insignia just under the rib cage bilaterally. This is also known as a bilateral subcostal,
double Kocher, or rooftop incision. For additional access, a Mercedes Incision can
be made by adding a small midline extension toward the xiphoid from the chevron
incision, therefore, resembling a Mercedes-Benz automobile emblem. Immediately
upon entry into the abdomen, the bowel should be eviscerated early. Incision should
be done rapidly but in a controlled manner as a major pitfall during this step would
be iatrogenic injury to the liver, bowel, or bladder.
Temporary Bleeding Control
Once inside the abdomen, the rst step should be temporary bleeding control. BAT
begins with empiric packing. Packing should be placed over and under the liver,
over and medial to the spleen, along the right gutter, along the left gutter, and in the
pelvis. Although it will not arrest major hemorrhage, it will give time to organize
efforts and systematically explore the abdomen. PAT begins by directly approaching
the bleed. Although some have recommended empiric packing in all trauma laparotomies, the consensus appears to be in favor of directly approaching the bleed.
The exsanguinating patient should be approached by obtaining supraceliac control of the Aorta. This is done through division of the Gastrohepatic ligament.
Although normally avascular, care should be taken to watch for a replaced left
hepatic artery. The stomach and esophagus are then reected to the left to allow
access to the aorta. Division of the diaphragmatic crura may be required. The aorta
can then be bluntly dissected and occluded. Aortic occlusion is typically with a
vascular clamp although it can also be compressed against the spine either manually
or with an aortic root compressor.

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Other described methods for aortic control include suprahepatic control through
the triangular ligaments, inframesocolic control through the inferior mesentery, or a
left-sided medial visceral rotation (Mattox maneuver). These other methods, however, have fallen out-of-favor as suprahepatic control is technically more difcult,
inframesocolic control does not halt bleeding supplied by the proximal aortic
branches, and the Mattox maneuver, although providing more extensive exposure,
takes more time and should be reserved for exploration after temporary bleeding
control. A left anterolateral thoracotomy can also provide excellent control of the
distal thoracic aorta. However, it may produce unnecessary morbidity if the aorta
can instead be accessed directly from the abdomen. Resuscitative Endovascular
Balloon Occlusion of the Aorta (REBOA) is another technique being utilized at
some centers although its use is evolving and is not available in all centers.
361
Exploration
After bleeding has been temporarily controlled, time is allowed to systematically
explore the abdomen for signs of injury. Newly identied sites of bleeding should
be controlled with clamps or packing. Gastrointestinal perforations should be temporarily controlled with bowel, vascular, Babcock, or Allis clamps. The order of
exploration is not as important as making sure that the surgeon performs the same
steps the same way every time so that injuries are not missed. The most commonly
described order starts with inframesocolic exploration followed by supramesocolic,
lesser sac, and then nally retroperitoneum.
For inframesocolic exposure, the transverse colon is lifted cranially and the
bowel is run from the ligament of Treitz down to the rectum. Each loop of bowel is
systematically ipped between hands inspecting both sides as well as the associated
mesentery. Notorious sites of missed injury include the proximal jejunum near the
ligament of Treitz, posterior transverse colon, hepatic exure, and splenic exure.
Once nished, the bladder and pelvis are inspected. The transverse colon is then
pulled caudally for supramesocolic exposure. Typically, structures are inspected
from the patient’s right-to-left as if reading a book. First the liver and gallbladder
are palpated, the right kidney is palpated, the stomach is evaluated from the gastroesophageal junction down through the duodenal loop, and then the spleen and left
kidney are palpated. The gastroesophageal junction and retroperitoneal duodenum
are also at higher risk for missed injury and should allocate special attention. Next
the lesser sac is accessed by blunt dissection through the greater omentum, allowing
inspection of the posterior stomach and pancreas.
Finally, the retroperitoneum is evaluated. Exploration should be kept targeted
and limited based on missile trajectory and presence of hematoma. Hematoma
exploration is based on the retroperitoneal zones. Zone I is midline between the
kidneys from the aortic hiatus to the sacral promontory. Zone II exists lateral to the
kidneys. Zone III encompasses the pelvis with everything inferior to the sacral
promontory. All Zone I hematomas should be explored with risk for major vascular

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D. R. Velez and A. J. Duncan
injury. Penetrating Zone II hematomas should be selectively explored. Blunt Zone
II hematomas are further directed by associated structures. Peri-Colonic and PeriDuodenal hematomas should all be explored [16]. Peri-Renal hematomas should be
explored only if expanding, pulsatile or with active hemorrhage [16]. Exploration of
Zone II should be kept limited to exclude injury with the rst step being palpation
of the contralateral kidney to determine the need for heroic measures. Needless
exploration of Zone II may result in unnecessary loss of the kidney. All Zone III
hematomas due to penetrating trauma should be explored. Zone III hematomas due
to blunt trauma should be explored only if expanding, pulsatile, or with active hemorrhage. Unnecessary exploration of blunt Zone III hematomas can result in uncontrolled bleeding.
Retroperitoneal exposure is generally accomplished through any of four primary
maneuvers: the Mattox Maneuver, inframesocolic division, Kocher Maneuver, or
Cattell-Braasch Maneuver. In general, left-sided supramesocolic injuries should be
approached by a Mattox Maneuver, while isolated left-sided inframesocolic injuries
can be approached through an inframesocolic division. A right-sided suprarenal
injury can be approached through a simple Kocher Maneuver, although a rightsided infrarenal injury would require a Cattell-Braasch Maneuver. These maneuvers, however, have signicant overlap, and the exploration should be based on the
patient’s presentation, keeping it limited to avoid unnecessary morbidity yet extensive enough to allow adequate exposure.
A Mattox Maneuver, also known as a left-sided medial visceral rotation, allows
for visualization of the entire abdominal aorta as well as the primary arterial
branches. The descending colon is mobilized by incising along the Line of Toldt
from the sigmoid colon up to the splenic exure and then extending laterally around
the spleen. A hand is then used to sweep from inferior to superior with the dissection
plane directly against the posterior abdominal wall. All structures are then rotated
medially. To visualize the left renal artery, the kidney may be left behind during the
rotation. Major pitfalls to avoid would be injury to the spleen or risk for avulsion of
the left descending lumbar vein as it connects to the renal vein.
Inframesocolic division allows visualization of the inframesocolic aorta. It is
commonly used outside of trauma as the preferred operative approach for a transperitoneal open abdominal aortic aneurysm repair. Although a Mattox Maneuver
allows visualization of the same structures, the inframesocolic division allows for a
more targeted and limited dissection if access to the supramesocolic aorta is unnecessary. In practice the transverse mesocolon is reected cephalad and the small
bowel is eviscerated to the right. A retroperitoneal incision is then made starting at
the Ligament of Treitz to the left of the aorta. Avoid injury to the inferior mesenteric
vein with this initial cut. The incision is then carried inferiorly and should course to
the right of the aortic midline preventing the major pitfall of injury to the inferior
mesenteric artery or sigmoid mesentery.
The Kocher Maneuver provides visualization of the suprarenal inferior vena
cava, right renal hilum, duodenum, and pancreas. It is performed by incising the
posterior peritoneum lateral to the duodenum. A hand is then placed behind to
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