- •Contents
- •1 History of Surgery for Retinal Detachment
- •FOUNDATIONS OF RETINAL DETACHMENT SURGERY
- •DEVELOPMENT OF MODERN SURGICAL PROCEDURES
- •TYPES OF RETINAL DETACHMENT
- •RETINAL BREAKS
- •EPIDEMIOLOGY OF RETINAL DETACHMENT
- •SYSTEMIC AND GENETIC CONDITIONS ASSOCIATED WITH RETINAL DETACHMENT
- •CLASSIFICATION OF RETINAL DETACHMENTS
- •PATHOLOGY OF THE DETACHED RETINA
- •NATURAL HISTORY OF UNTREATED DETACHMENT
- •SUMMARY
- •3 Ophthalmoscopy
- •CHARACTERISTICS OF INDIRECT AND DIRECT OPHTHALMOSCOPY
- •BASIC INDIRECT OPHTHALMOSCOPY TECHNIQUES
- •EXAMINATION THROUGH A SMALL PUPIL
- •SCLERAL DEPRESSION
- •SUMMARY
- •4 Evaluation and Management
- •OCULAR EVALUATION
- •RETINAL EXAMINATION
- •PREPARATION FOR SURGERY
- •POSTOPERATIVE MANAGEMENT
- •SUMMARY
- •5 Establishing the Diagnosis
- •FUNDUS CHANGES UNRELATED TO RETINAL DETACHMENT
- •NONRHEGMATOGENOUS RETINAL DETACHMENT
- •LESIONS SIMULATING RETINAL DETACHMENT
- •SUMMARY
- •6 Prevention of Retinal Detachment
- •RISK FACTORS FOR RETINAL DETACHMENT
- •SYMPTOMATIC EYES
- •ASYMPTOMATIC EYES
- •TREATMENT TO PREVENT RETINAL DETACHMENT
- •SUMMARY
- •7 Scleral Buckling
- •ANATOMICAL AND PHYSIOLOGICAL EFFECTS OF SCLERAL BUCKLES
- •PRINCIPLES OF SCLERAL BUCKLING
- •THE SCLERAL BUCKLING OPERATION
- •COMMON COMPLICATIONS OF SCLERAL BUCKLING
- •SUMMARY
- •8 Pneumatic Retinopexy
- •INTRAOCULAR GASES
- •PREOPERATIVE EVALUATION
- •INDICATIONS AND CONTRAINDICATIONS
- •OPERATIVE TECHNIQUE
- •SPECIAL PROCEDURES
- •SUMMARY OF PROCEDURE
- •POSTOPERATIVE MANAGEMENT
- •COMPLICATIONS
- •COMPARISON WITH SCLERAL BUCKLING
- •SUMMARY
- •VITRECTOMY TECHNIQUES FOR COMPLICATED CASES
- •RESULTS OF VITRECTOMY
- •COMPLICATIONS OF VITRECTOMY
- •SUMMARY
- •SURGERY FOR COMMON TYPES OF RETINAL DETACHMENT
- •TWELVE REPRESENTATIVE CASES
- •CONCLUSION
- •Index
7
Scleral Buckling
Inflammatory detachments are usually treated medically. Some serous detachments, such as choroidal hemangioma, respond to photocoagulation or photodynamic therapy (PDT). Selected traction detachments, such as diabetic or post-traumatic detachments, may be cured with intraocular microsurgery (vitrectomy). Radiation therapy is often used for detachments secondary to metastatic tumors. This chapter is confined to the surgical management of rhegmatogenous detachments with scleral buckling. Alternative methods of repair are discussed in
Chapters 8 and 9, and the three techniques are compared in Chapter 10. Controversy exists regarding the details of the surgical technique, but surgeons
generally agree on the three basic steps in closing retinal breaks and reattaching the retina:
1.Conducting thorough preoperative and intraoperative examinations with the goal of locating all retinal breaks and assessing any vitreous traction on the retina.
2.Creating a controlled injury to the retinal pigment epithelium and retina to produce a chorioretinal adhesion surrounding all retinal breaks so that intravitreal fluid can no longer reach the subretinal space.
3.Employing an appropriate technique, such as scleral buckling and/or intravitreal gas, to approximate the retinal breaks to the underlying treated retinal pigment epithelium.
If the surgeon follows these basics and applies modern surgical techniques, retinal reattachment may be expected following a single operation in more than 85% of uncomplicated primary detachments, and in more than 95% following additional procedures.
149
150 II: Practice
The traditional scleral buckle has served very well since the 1950’s. However, more recent developments have produced a more comprehensive menu for retinal reattachment surgery from which the surgeon may select the appropriate procedure for each case. By the turn of the millennium, surveys had demonstrated that scleral buckling alone was no longer the most popular means of repairing uncomplicated primary retinal detachments. Still, it is a valuable technique that is indicated in many situations.
Temporary scleral buckling can be performed with scleral infolding, gelatin, or orbital balloon. The term scleral buckling without a qualifying adjective is generally recognized as referring to a “permanent” scleral buckle with the implantation of a foreign material usually made of silicone.
Successful scleral buckling depends upon a thorough understanding of the anatomical and physiological effects of the procedure. These are substantially different from those associated with Pneumatic Retinopexy (Chapter 8) or Vitrectomy (Chapter 9).
ANATOMICAL AND PHYSIOLOGICAL EFFECTS OF SCLERAL BUCKLES
Anatomical changes in the vitreous gel and defects in the retina are of fundamental importance as causes of subsequent retinal detachment, as noted in Chapter 2. Scleral buckling of the eye wall and retina favorably alters pathoanatomy and allows normal physiological forces to reattach the retina.
PATHOPHYSIOLOGY OF RHEGMATOGENOUS RETINAL DETACHMENT
Rhegmatogenous retinal detachment occurs when factors maintaining attachment are overwhelmed by the volume of intravitreal fluid entering the potential subretinal space through retinal breaks, as noted in Chapter 2. Isolated defects in the retina are usually not solely responsible for clinical retinal detachments. In the vast majority of cases, vitreoretinal traction forces upon the retina near the location of the break(s) are required for a progressive accumulation of fluid in the subretinal space. Liquid currents in front of retinal breaks and in the subretinal space contribute secondarily to progression of retinal detachment.
REATTACHMENT FORCES INFLUENCED BY SCLERAL BUCKLES
Localized indentation of the sclera, choroid, and pigment epithelium beneath a retinal break alters the anatomical and physiological factors associated with the production of a retinal detachment. The fundamental goal of scleral buckling is the functional closure of all retinal breaks, so that normal physiological forces can maintain a permanent state of attachment. Drainage of subretinal fluid and scleral buckling will usually close the responsible break(s) immediately.
In a non-drainage procedure, functional closure of retinal breaks can result from several beneficial effects of a scleral buckle, including:
7: Scleral Buckling |
151 |
1.reduction of vitreoretinal traction by displacing the eye wall and retina centrally;
2.displacement of subretinal fluid away from the location of the retinal break and scleral buckle;
3.postoperative increase in the height of the scleral buckle;
4.approximation of the retinal break and adjacent vitreous gel;
5.increase in resistance to fluid flow through the retinal break, with consequent increase in the relative reattachment forces;
6.alteration in the concave shape of the eyeball, resulting in a change in the effect of intraocular currents that encourage liquid vitreous to enter the subretinal space.
These effects are probably synergistic, and are also important in drainage cases. Although contemporary scleral buckling procedures routinely include the creation of a chorioretinal burn, such an adhesion is not always necessary to maintain retinal reattachment.
PRINCIPLES OF SCLERAL BUCKLING
The most important skill required in surgery for retinal detachment is the ability to detect all retinal breaks and additional areas of vitreoretinal pathology. Scleral buckling is performed to produce functional closure of retinal breaks responsible for retinal detachment and to reduce the chances of recurrent detachment. Various kinds and shapes of silicone are used, including segments of silicone sponge as well as solid silicone shaped into bands for encircling the eye and into additional forms to augment the width and height of the buckle in selected areas (Figure 7–1). The specific configuration of the scleral buckle depends upon a number of factors.
Following localization and treatment of retinal breaks and areas of vitreoretinal degeneration, the silicone buckling element is sutured to the scleral surface. Drainage of subretinal fluid is performed in the majority of cases. Intravitreal gas injection is sometimes employed in conjunction with scleral buckling. Problems encountered at any point of the procedure may require modifications in technique.
SCLERAL BUCKLE CONFIGURATION
The location, number, size, and types of retinal breaks are important variables affecting the selection of a specific buckling technique. Similarly, the presence of vitreoretinal degeneration, with or without retinal breaks, and of significant vitreoretinal traction unassociated with retinal breaks should be considered in the preoperative assessment (Figure 7–2).
If retinal breaks, vitreoretinal degenerative disorders, and significant vitreoretinal traction are present in multiple quadrants, a circumferential buckle is probably favored. A single retinal break unassociated with additional significant problems is usually managed with an isolated segmental buckle, if not with pneumatic retinopexy.
152 II: Practice
BANDS AND STRIPS
2
40 |
0.75 |
20
5.7
4.0
31
7.2
4.5
32
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9.2 |
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5.0 |
41 |
0.75 |
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4.8 |
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3.5 |
42 |
1.25 |
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5.9 |
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4 |
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2.5 |
240 |
0.6 |
219
6
4.5
220
7.5
6.0
225
8
5.0
HEMISPHERE
77G
26.0
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TIRES |
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IMPLANTS |
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275 |
276 |
Band |
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6.7 |
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240 |
6.2 |
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278 |
9 |
Clip |
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7.0 |
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250 |
10.0 |
277 |
50 |
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8.5 |
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286 |
9 |
51 |
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52 |
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7.0 |
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6.6 |
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Boat |
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260 |
6.0 |
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60 |
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287 |
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Sleeve |
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9 |
70 |
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270 |
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7.0 |
ID = 1.0 |
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ID = .76 |
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OD = 2.1 |
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OD = 1.65 |
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289 |
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71 |
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72 |
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ID = 1.0 |
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ID = 1.6 |
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12 |
OD = 2.2 |
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OD = 2.4 |
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10.0 |
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IMPLANTS |
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279 |
11 |
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103 |
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22 |
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9.0 |
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280 |
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4.0 |
15.0 |
5.0 |
5.0 |
12 |
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10.0 |
106 |
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80 |
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280LG |
4.0 |
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4.0 |
8.0 |
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6.0 |
10.5 |
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12 |
112 |
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190 |
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10.0 |
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78G |
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12.0 |
12.0 |
5.0 |
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12 |
10.0 |
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10.0 |
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RADIAL WEDGES |
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281 |
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16 |
135 |
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16 |
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12.5 |
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14.0 |
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287WG |
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7.0 |
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288 |
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137 |
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14 |
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2.0 |
10.0 |
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12.0 |
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Figure 7–1. A wide variety of solid silicone and silicone sponge materials are available for scleral buckling. (Courtesy of MIRA, Inc.)
The anterior–posterior dimensions of retinal break(s) and areas of significant vitreoretinal degeneration and vitreoretinal traction are also important considerations in planning a buckling procedure. Scleral buckles should support all edges of the retinal breaks and associated areas of vitreoretinal degeneration. In general, the buckling effect should extend into the zone of the vitreous base to eliminate current and future traction forces.
