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Ординатура / Офтальмология / Английские материалы / Rapid Diagnosis in Ophthalmology Series Oculoplastic and Reconstructive Surgery_Nerad, Carter, Alford_2008

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Fig. 2.5 Chalazion of right lower eyelid. Note absence of infl ammation.

Hordeolum and Chalazion

Fig. 2.6 Hordeolum of left lower eyelid. Lesion is erythematous and tender.

Fig. 2.7 Everted eyelid with chalazion clamp in place.

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Lesions• 2 SECTIONEyelid: the of Disorders

Chalazion and Hordeolum (Continued)

Surgical

Drainage and curettage if medical therapy fails

Best performed via conjunctival surface with a chalazion clamp in place (Fig. 2.7): use a no. 11 blade to open chalazion with a vertical incision; scoop out contents with a curette (Fig. 2.8)

Intralesional injection of steroid can be effective

Complications:

gland dropout

eyelash loss

eyelid margin changes

ptosis

conjunctival scar

Prognosis

Most cases respond to medical and/or surgical treatment with no long-term complications

Patients with rosacea may have recurrent disease

Recurrence rate high if underlying blepharitis or meibomianitis not treated with chronic eyelid hygiene (see above)

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Fig. 2.8 Lipogranulomatous contents of a chalazion during excision.

Fig. 2.9 Hordeolum at eyelid margin.

(continued) Hordeolum and Chalazion

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Lesions• 2 SECTIONEyelid: the of Disorders

Seborrheic Keratosis

Key Facts

Most common skin lesion, common in periocular area

Benign neoplasm

Middle-aged to older patients

Clinical Findings

Lesions are brown to dark brown warty plaques

Lesion has a greasy, stuck-on appearance, especially away from the thin eyelid skin (Fig. 2.10)

Eyelid skin lesions tend to appear pedunculated or papillary rather than flat (Fig. 2.11)

Ancillary Testing

None

Differential Diagnosis

Malignant melanoma if darkly or asymmetrically pigmented

Epidermal nevus

Verruca vulgaris

Acrochordon (skin tag)

Treatment

Observation if sure of diagnosis

Biopsy if lesion is symptomatic, catching clothing

Shave biopsy works well

Cryotherapy can be effective

Prognosis

Excellent, low recurrence rate

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Fig. 2.10 Brown, warty-looking lesion with classic stuck-on appearance of seborrheic keratosis.

Keratosis Seborrheic

Fig. 2.11 Brownish black pedunculated papillary lesion of the right lower eyelid.

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Lesions• 2 SECTIONEyelid: the of Disorders

Epidermal Inclusion Cyst

Key Facts

Occurs when epithelial cells are turned inward, buried within the skin

The epidermal wall sheds keratin

Usually the result of entrapment of epithelial cells of the infundibulum of the hair follicle

May occur after trauma if the skin surface is interrupted

Common benign lesion in periocular area

Clinical Findings

Lesion appears smooth (Fig. 2.12), and a central pore may be present

Slowly enlarges as the cyst wall produces keratin (not sebaceous material)

Can be associated with minor skin trauma

Ancillary Testing

None

Differential Diagnosis

Neurofibroma

Treatment

Observation

Marsupialization of cyst is simple and effective

If complete excision of cyst is attempted, the entire cyst wall must be removed to prevent recurrence

Prognosis

Excellent with marsupialization or complete cyst excision

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Fig. 2.12 Smooth, elevated, whitish lesion in the right medial canthus.

Cyst Inclusion Epidermal

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Lesions• 2 SECTIONEyelid: the of Disorders

Xanthelasma

Key Facts

Common skin lesion of the medial aspect of upper and lower eyelids

Middle-aged to older patients

Usually bilateral

Usually not associated with hyperlipidemic condition

Clinical Findings

Yellowish dermal plaques of the eyelids (Fig. 2.13)

Lesion is composed of lipid-laden macrophages

Upper lids more common location

Ancillary Testing

Serum lipid levels if suspected metabolic disorder

Differential Diagnosis

Usually no other lesions look like this, but some other xanthematous lesions may appear yellow

Treatment

Full-thickness excision of lesion

Must be careful not to remove too much of the anterior lamella of the eyelid

Must avoid lagophthalmos and ectropion with skin closure

Carbon dioxide laser ablation or trichloroacetic acid is less likely to be effective, because the lesion extends deep into the dermis

Full-thickness skin grating may be required after excision of larger lesions

Prognosis

Excellent with surgical excision

Recurrence possible

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Fig. 2.13 A 49-year-old woman with yellowish plaques in all four eyelids.

Xanthelasma

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Lesions• 2 SECTIONEyelid: the of Disorders

Nevus

Key Facts

Common lesion of the eyelid

Appearance depends on skin level where nevus resides

Nevus cells arise from undifferentiated melanocytes

Nevi not normally present at birth

Nevi usually appear and can increase in size and change in pigmentation during puberty

Three stages of a nevus

1.Junctional: at junction of epidermis and dermis

2.Compound: in epidermis extending deeper into dermis

3.Dermal: solely in dermis

Clinical Findings

Children: nevi usually flat pigmented macules

This stage is junctional, usually appearing in adolescence

Middle-aged adults: elevated lesion, pigmented lesion compound stage. The pigment is slowly lost as the nevus ages (Fig. 2.14)

Older patients:

dermal stage

pigment usually lost

lesion appears elevated in a dome shape

A common location is the eyelid margin

Eyelid margin structures are not destroyed, as with carcinomas

Eyelashes grow out through the nevus but may lose their parallel orientation (Fig. 2.15)

Ancillary Testing

None. See below for indications for biopsy

Differential Diagnosis

Pigmented seborrheic keratosis

Melanoma (see below)

Treatment

Observation

Simple shave biopsy removes any elevated portion (may mean slowly)

Wedge resection of the eyelid margin or excisional biopsy at full thickness eyelid skin for complete removal

Prognosis

Excellent

If only superficial portion is removed, the nevus may recur over time

Biopsy recommended if melanoma suspected:

large lesions (>6 mm)

bleeding

highly irregular borders

change in pigmentation

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