- •Foreword
- •Erratum
- •Preface
- •The Physical Examination of the Eye
- •The orbit
- •The external eye
- •The eyeball
- •The conjunctiva
- •Tenon’s capsule
- •The sclera and episclera
- •The cornea
- •The anterior chamber
- •The uvea
- •The iris
- •The ciliary body
- •The choroid
- •The lens
- •The retina
- •The vitreous
- •The extraocular muscles
- •The rectus muscles
- •The oblique muscles
- •Innervation of the eye
- •The optic nerve (II)
- •The oculomotor nerve (III)
- •The trochlear nerve (IV)
- •The trigeminal nerve (V)
- •The abducens nerve (VI)
- •The blood supply of the eye
- •Physical examination of the eye
- •Vision
- •External examination
- •Extraocular movements
- •Examination of the conjunctiva
- •Examination of the sclera and episclera
- •Examination of the uvea
- •Intraocular pressure
- •Examination of the cornea
- •Examination of the pupils
- •Examination of the anterior chamber
- •Ophthalmoscopy
- •Summary
- •References
- •Visual acuity testing
- •Slit lamp examination
- •Flourescein examination
- •Tonometry
- •Lid eversion
- •Foreign body removal
- •Contact lens removal
- •Eye irrigation
- •Paracentesis
- •Lateral canthotomy
- •Ocular ultrasonography
- •Summary
- •References
- •Conjunctivitis
- •Subconjunctival hemorrhage
- •Episcleritis
- •Scleritis
- •Uveitis
- •Acute angle-closure glaucoma
- •Summary
- •References
- •Conjunctivitis
- •Viral conjunctivitis
- •Bacterial conjunctivitis
- •Neonatal conjunctivitis
- •Episcleritis
- •Keratitis
- •Viral keratitis
- •Bacterial keratitis
- •Keratitis due to light exposure
- •Uveitis
- •Anterior uveitis
- •Intermediate uveitis
- •Posterior uveitis and retinitis
- •Hordeolum and chalazion
- •Dacryocystitis
- •Periorbital and orbital cellulitis
- •References
- •Acute Monocular Visual Loss
- •Temporal arteritis
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Optic neuritis
- •Epidemiology
- •Etiology
- •Clinical features and diagnosis
- •Treatment
- •Central retinal artery occlusion
- •Epidemiology
- •Etiology
- •Cardiogenic embolism
- •Other causes
- •Clinical features
- •Diagnosis and treatment
- •Central retinal vein occlusion
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Retinal detachment
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Retinal vasculitis
- •Epidemiology and etiology
- •Clinical characteristics
- •Diagnosis and treatment
- •Summary
- •References
- •Trauma to the Globe and Orbit
- •History and physical examination
- •Imaging techniques
- •CT scan
- •Ultrasound
- •Blunt trauma to the orbit
- •Periorbital tissues
- •Orbital fractures
- •Retrobulbar hemorrhage
- •Anterior chamber
- •Traumatic hyphema
- •Subconjunctival hemorrhage
- •Injury to the iris and ciliary body
- •Traumatic iridocyclitis (uveitis)
- •Traumatic mydriasis and miosis
- •Iridodialysis
- •Acute glaucoma
- •Injury to the lens
- •Subluxation and dislocation
- •Cataract formation
- •Globe injury
- •Globe rupture
- •Globe luxation
- •Posterior segment
- •Vitreous hemorrhage
- •Chorioretinal injury
- •Commotio retina
- •Penetrating ocular injury
- •Periorbital tissues
- •Conjunctival lacerations
- •Laceration of the eyelid
- •Globe injury
- •Corneoscleral laceration and puncture wounds
- •Intraocular foreign body
- •Orbital foreign body
- •Delayed complications
- •Endophthalmitis
- •Sympathetic ophthalmia
- •Burns
- •Acid and alkali exposure
- •Miscellaneous irritants, solvents, and detergents
- •Thermal burns
- •UV keratitis
- •Prevention
- •Acknowledgment
- •References
- •Chemical burns
- •Pathophysiology
- •Alkali injury
- •Acid injury
- •Cyanoacrylate exposure
- •Treatment
- •Thermal injuries
- •Radiation injuries
- •Treatment
- •Biologic exposures
- •Treatment
- •Disposition
- •References
- •Neuro-Ophthalmology
- •Neuroanatomy and neuro-ophthalmologic examination
- •The visual pathway
- •The cranial nerves
- •Neuro-ophthalmologic examination
- •Visual acuity
- •Funduscopic examination
- •Testing ocular motility
- •Pupillary disorders
- •Pupil size and reactivity
- •Anisocoria
- •Horner syndrome
- •Tonic (Adie) pupil
- •Pharmacotherapy and pupils
- •Traumatic optic neuropathy
- •Optic neuritis
- •Oculomotor nerve palsy
- •Extraocular movement disorders
- •Cranial nerve palsies and binocular diplopia
- •Cranial nerve III
- •Cranial nerve IV
- •Cranial nerve VI
- •Nystagmus
- •Peripheral nystagmus
- •Central nystagmus
- •Myasthenia gravis
- •Multiple sclerosis
- •Stroke syndromes and gaze palsies
- •Stroke syndromes and the visual system
- •Anterior cerebral artery
- •Internal carotid artery
- •Middle cerebral artery
- •Posterior cerebral artery
- •Basilar artery
- •Vertebal arteries
- •Gaze palsies/conjugate gaze deviation
- •Hemispheric lesions
- •Midbrain lesions
- •Pontine lesions
- •Summary
- •References
- •Visual development
- •The eye examination in a child
- •Examination of the newborn and young infant
- •Older infants and preverbal children
- •Verbal children
- •Conjunctivitis
- •Ophthalmia neonatorum (neonatal conjunctivitis)
- •Childhood conjunctivitis
- •Orbital and periorbital cellulitis
- •Lacrimal system infections
- •Congenital
- •Nasal lacrimal duct obstruction
- •Congenital cataracts
- •Congenital glaucoma
- •Misalignment
- •Oncology
- •References
- •The Painful Eye
- •Acute angle closure glaucoma
- •Scleritis
- •Anterior uveitis (iritis)
- •HLA-B27-associated uveitis
- •Other noninfectious etiologies
- •Infectious etiologies
- •Treatment of anterior uveitis
- •Optic neuritis
- •Keratitis
- •Noninfectious keratitis
- •Ulcerative keratitis
- •Infectious keratitis
- •Bacterial
- •Viral
- •Fungal
- •Amoebic
- •Corneal abrasion
- •References
- •Acquired syphilis
- •Varicella-zoster virus infection
- •Lyme disease
- •Reiter’s syndrome
- •Infectious endocarditis
- •Kawasaki’s disease
- •Temporal arteritis
- •Hypertension
- •Diabetes
- •Summary
- •References
- •Emergency ophthalmology consultation caveats
- •Emergency diagnoses requiring emergency ophthalmology consultation
- •Trauma
- •Endophthalmitis
- •Acute angle closure glaucoma
- •Severe uveitis
- •Corneal ulceration
- •Acute visual loss
- •Optic neuritis
- •Central retinal artery occlusion
- •Retinal detachment
- •Orbital cellulitis
- •References
234 |
MAGAURAN |
important for emergency medicine practitioners who find themselves in such a situation to have referral hospitals that can be contacted to accept patients in transfer.
Emergency diagnoses requiring emergency ophthalmology consultation
The following box contains a list of diagnoses or conditions that generally warrant an emergency ophthalmologic consultation (Box 1).
There are also cases where emergency ophthalmologic consultation is reasonable, and these include patients with one good eye experiencing vision changes, patients with a complicated ocular history, as well as cases where the ED practitioner is unsure of a diagnosis and is concerned about vision loss. A telephone discussion to determine the timing of the consultation with the ophthalmologist will usually answer such questions.
Trauma
Emergency physicians are well versed in the management of trauma. The basic ABC principle (Airway, Breathing, Circulation) in ED trauma management takes precedence over eye injuries. Evaluation for life and limbthreatening injuries is the first priority. Most patients with multiple trauma and an eye injury will require ophthalmologic consultation, but the timing of this consultation is dependent on the severity of other injuries. Most often, this consultation occurs at some point during the in-patient stay after life threatening conditions have been addressed. This article reviews the conditions that usually require ophthalmologic consultation from the ED.
Box 1. Conditions that generally warrant an emergency ophthalmologic consultation
Trauma
Ruptured globe
Lid laceration through margin, nasolacrimal system, or cannaliculus
Endophthalmitis
Angle closure glaucoma Severe uveitis
Corneal ulceration Acute vision loss
Central retinal artery occlusion Temporal arteritis
Retinal detachment Optic Neuritis
Orbital cellulitis
OPHTHALMOLOGIC CONSULTATION |
235 |
CT scan is the imaging modality of choice in defining the extent of blunt and penetrating trauma to the eye and orbit. Orbital fractures are not an emergency unless there is vision loss or globe injury. Retrobulbar hemorrhage may occur with nondisplaced orbital fractures and may result in acute vision loss. Penetrating injuries with globe rupture also require CT scanning for retained foreign bodies, but sensitivity is only 75% [3]. These entities are discussed in detail in the article by Linden and Bord elsewhere in this issue.
Direct trauma to the eyelid involving the nasolacrimal system, lid margin, or tarsal plate requires specialized repair by either a plastic surgeon or an ophthalmologist, depending on institutional preference and availability. The timing of this repair should occur after any life or limb-threatening injuries are addressed.
Endophthalmitis
Endophthalmitis represents inflammation of the aqueous or vitreous humor. It may result from infection, trauma, or as a postoperative complication. This is generally a di cult diagnosis to make in the ED. Suspicion of the diagnosis requires emergent ophthamologic consultation for diagnostic evaluation, hospital admission, and surgery as necessary.
Acute angle closure glaucoma
Acute angle closure glaucoma (AACG) is generally painful and occasionally associated with headache, nausea, vomiting, and abdominal pain. The diagnosis may be di cult to establish when abdominal complaints are prominent [4]. Intraocular pressure generally rises beyond 50 mm Hg and may induce optic nerve atrophy if untreated. The emergency physician can certainly initiate therapy and consult an ophthalmologist. The issue is then response to treatment and monitoring of the intraocular pressure. Generally, patients with a new diagnosis of AACG are admitted to the hospital and an ophthalmologist oversees medical therapy. Cases refractory to medical therapy may require surgical intervention. AACG is discussed in detail in the article by Lowenstein and Dargin elsewhere in this issue.
Severe uveitis
Anterior uveitis (iridocyclitis) is inflammation of the iris (iritis) and ciliary body (cyclitis) and accounts for the majority of uveitis in Western countries [5]. It typically occurs in patients between the ages of 20 and 50 [5]. Inflammatory cells and flare in the anterior chamber associated with conjunctival injection primarily involving the limbus help confirm the diagnosis. The etiology of anterior uveitis is broad and may include infection, rheumatologic disease (HLA-B27 particularly), idiopathic, or even a malignancy which may mimic anterior uveitis. Given the broad di erential diagnosis associated with this condition, consultations with specialists in rheumatology,
