- •Foreword
- •Erratum
- •Preface
- •The Physical Examination of the Eye
- •The orbit
- •The external eye
- •The eyeball
- •The conjunctiva
- •Tenon’s capsule
- •The sclera and episclera
- •The cornea
- •The anterior chamber
- •The uvea
- •The iris
- •The ciliary body
- •The choroid
- •The lens
- •The retina
- •The vitreous
- •The extraocular muscles
- •The rectus muscles
- •The oblique muscles
- •Innervation of the eye
- •The optic nerve (II)
- •The oculomotor nerve (III)
- •The trochlear nerve (IV)
- •The trigeminal nerve (V)
- •The abducens nerve (VI)
- •The blood supply of the eye
- •Physical examination of the eye
- •Vision
- •External examination
- •Extraocular movements
- •Examination of the conjunctiva
- •Examination of the sclera and episclera
- •Examination of the uvea
- •Intraocular pressure
- •Examination of the cornea
- •Examination of the pupils
- •Examination of the anterior chamber
- •Ophthalmoscopy
- •Summary
- •References
- •Visual acuity testing
- •Slit lamp examination
- •Flourescein examination
- •Tonometry
- •Lid eversion
- •Foreign body removal
- •Contact lens removal
- •Eye irrigation
- •Paracentesis
- •Lateral canthotomy
- •Ocular ultrasonography
- •Summary
- •References
- •Conjunctivitis
- •Subconjunctival hemorrhage
- •Episcleritis
- •Scleritis
- •Uveitis
- •Acute angle-closure glaucoma
- •Summary
- •References
- •Conjunctivitis
- •Viral conjunctivitis
- •Bacterial conjunctivitis
- •Neonatal conjunctivitis
- •Episcleritis
- •Keratitis
- •Viral keratitis
- •Bacterial keratitis
- •Keratitis due to light exposure
- •Uveitis
- •Anterior uveitis
- •Intermediate uveitis
- •Posterior uveitis and retinitis
- •Hordeolum and chalazion
- •Dacryocystitis
- •Periorbital and orbital cellulitis
- •References
- •Acute Monocular Visual Loss
- •Temporal arteritis
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Optic neuritis
- •Epidemiology
- •Etiology
- •Clinical features and diagnosis
- •Treatment
- •Central retinal artery occlusion
- •Epidemiology
- •Etiology
- •Cardiogenic embolism
- •Other causes
- •Clinical features
- •Diagnosis and treatment
- •Central retinal vein occlusion
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Retinal detachment
- •Epidemiology
- •Etiology
- •Clinical features
- •Diagnosis and treatment
- •Retinal vasculitis
- •Epidemiology and etiology
- •Clinical characteristics
- •Diagnosis and treatment
- •Summary
- •References
- •Trauma to the Globe and Orbit
- •History and physical examination
- •Imaging techniques
- •CT scan
- •Ultrasound
- •Blunt trauma to the orbit
- •Periorbital tissues
- •Orbital fractures
- •Retrobulbar hemorrhage
- •Anterior chamber
- •Traumatic hyphema
- •Subconjunctival hemorrhage
- •Injury to the iris and ciliary body
- •Traumatic iridocyclitis (uveitis)
- •Traumatic mydriasis and miosis
- •Iridodialysis
- •Acute glaucoma
- •Injury to the lens
- •Subluxation and dislocation
- •Cataract formation
- •Globe injury
- •Globe rupture
- •Globe luxation
- •Posterior segment
- •Vitreous hemorrhage
- •Chorioretinal injury
- •Commotio retina
- •Penetrating ocular injury
- •Periorbital tissues
- •Conjunctival lacerations
- •Laceration of the eyelid
- •Globe injury
- •Corneoscleral laceration and puncture wounds
- •Intraocular foreign body
- •Orbital foreign body
- •Delayed complications
- •Endophthalmitis
- •Sympathetic ophthalmia
- •Burns
- •Acid and alkali exposure
- •Miscellaneous irritants, solvents, and detergents
- •Thermal burns
- •UV keratitis
- •Prevention
- •Acknowledgment
- •References
- •Chemical burns
- •Pathophysiology
- •Alkali injury
- •Acid injury
- •Cyanoacrylate exposure
- •Treatment
- •Thermal injuries
- •Radiation injuries
- •Treatment
- •Biologic exposures
- •Treatment
- •Disposition
- •References
- •Neuro-Ophthalmology
- •Neuroanatomy and neuro-ophthalmologic examination
- •The visual pathway
- •The cranial nerves
- •Neuro-ophthalmologic examination
- •Visual acuity
- •Funduscopic examination
- •Testing ocular motility
- •Pupillary disorders
- •Pupil size and reactivity
- •Anisocoria
- •Horner syndrome
- •Tonic (Adie) pupil
- •Pharmacotherapy and pupils
- •Traumatic optic neuropathy
- •Optic neuritis
- •Oculomotor nerve palsy
- •Extraocular movement disorders
- •Cranial nerve palsies and binocular diplopia
- •Cranial nerve III
- •Cranial nerve IV
- •Cranial nerve VI
- •Nystagmus
- •Peripheral nystagmus
- •Central nystagmus
- •Myasthenia gravis
- •Multiple sclerosis
- •Stroke syndromes and gaze palsies
- •Stroke syndromes and the visual system
- •Anterior cerebral artery
- •Internal carotid artery
- •Middle cerebral artery
- •Posterior cerebral artery
- •Basilar artery
- •Vertebal arteries
- •Gaze palsies/conjugate gaze deviation
- •Hemispheric lesions
- •Midbrain lesions
- •Pontine lesions
- •Summary
- •References
- •Visual development
- •The eye examination in a child
- •Examination of the newborn and young infant
- •Older infants and preverbal children
- •Verbal children
- •Conjunctivitis
- •Ophthalmia neonatorum (neonatal conjunctivitis)
- •Childhood conjunctivitis
- •Orbital and periorbital cellulitis
- •Lacrimal system infections
- •Congenital
- •Nasal lacrimal duct obstruction
- •Congenital cataracts
- •Congenital glaucoma
- •Misalignment
- •Oncology
- •References
- •The Painful Eye
- •Acute angle closure glaucoma
- •Scleritis
- •Anterior uveitis (iritis)
- •HLA-B27-associated uveitis
- •Other noninfectious etiologies
- •Infectious etiologies
- •Treatment of anterior uveitis
- •Optic neuritis
- •Keratitis
- •Noninfectious keratitis
- •Ulcerative keratitis
- •Infectious keratitis
- •Bacterial
- •Viral
- •Fungal
- •Amoebic
- •Corneal abrasion
- •References
- •Acquired syphilis
- •Varicella-zoster virus infection
- •Lyme disease
- •Reiter’s syndrome
- •Infectious endocarditis
- •Kawasaki’s disease
- •Temporal arteritis
- •Hypertension
- •Diabetes
- •Summary
- •References
- •Emergency ophthalmology consultation caveats
- •Emergency diagnoses requiring emergency ophthalmology consultation
- •Trauma
- •Endophthalmitis
- •Acute angle closure glaucoma
- •Severe uveitis
- •Corneal ulceration
- •Acute visual loss
- •Optic neuritis
- •Central retinal artery occlusion
- •Retinal detachment
- •Orbital cellulitis
- •References
TRAUMA TO THE GLOBE AND ORBIT |
121 |
ocular pain, foreign body sensation, red eye, tearing, photophobia, and blurred vision. Examination reveals decreased visual acuity and di use punctate lesions on fluorescein staining, with a sharp demarcation at the lower lid where the eye was protected. Treatment consists of cycloplegic drops, topical antibiotic ointment, and oral analgesics.
Prevention
Wearing protective eyewear often can prevent injuries to the globe. A study of patients in the US military who had sustained eye injury found that only a small percentage of them were wearing protective glasses at the time of injury [37,42]. In addition, many sports-related eye injuries could be prevented by wearing the recommended protective equipment. Air bag deployment, although associated with a 20% reduction in the incidence of fatal and severe injuries after frontal and near-frontal automobile collisions, increases the risk of orbital fractures or other ocular trauma [44].
Acknowledgment
The authors thank Dr. John Lee for his expert manuscript review and comments.
References
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Emerg Med Clin N Am
26 (2008) 125–136
Chemical, Thermal, and Biological
Ocular Exposures
Jordan Spector, MDa,b,
William G. Fernandez, MD, MPHa,*
aBoston Medical Center, Department of Emergency Medicine, Boston University School of Medicine, One BMC Place, Dowling 1 South, Boston, MA 02118, USA
bDepartment of Emergency Medicine, Albert Einstein Medical Center, 5501 Old York Road,
Philadelphia, PA, USA
Chemical or radiant energy injuries to the eyes are considered ocular burns. The majority of these injuries are occupation-related [1]. Chemical burns are by far more common and represent a true emergency. Thermal and UV injuries are associated with severe pain, but often result in less long-term sequelae than chemical injuries do. The term ‘‘biologic exposure’’ refers to an exposure to human blood or other body fluid. This article describes patterns of these injuries and exposures, with particular emphasis on emergent management and including acute diagnostic and treatment considerations.
Chemical burns
Chemical burns to the eye are common, particularly in industrial settings, and constitute an ocular emergency. In fact, chemical burns were the second leading cause of work-related eye injury treated in United States emergency departments in 1999 [2]. A burn may occur with exposure of the eye to any chemical, solid, liquid, or aerosol. Household cleaning supplies and cosmetics are common o enders. The potent alkaline or acidic substances contained within these products cause the burn injury. Accidents involving industrial materials in the workplace are a frequent cause of eye burns.
Chemical exposures to the eye can result in significant damage to the ocular surface epithelium, the cornea, and the anterior segment. Permanent unilateral or bilateral visual impairment may result. Alkaline substances can
* Corresponding author.
E-mail address: william.fernandez@bmc.org (W.G. Fernandez).
0733-8627/08/$ - see front matter 2008 Elsevier Inc. All rights reserved. |
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doi:10.1016/j.emc.2007.11.002 |
emed.theclinics.com |
