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Ординатура / Офтальмология / Английские материалы / Illustrated Tutorials in Ophthalmology Kanski, Bolton 2001

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Clinical features of full-thickness macular hole

Typically affects elderly females

Eventually bilateral in 10%

VA about 6/60

 

 

 

 

Round punched-out area at fovea

Multiple yellow deposits within crater

Surrounding halo of sub-retinal fluid

Positive Watzke-Allen sign

 

 

 

 

FA of full-thickness macular hole

Hyperfluorescence due to RPE ‘window defect’

Treatment of Macular Hole

1.Indications

Full-thickness macular hole

Visual acuity < 6/18

Duration < 1 year

2.Technique

Vitrectomy and fluid-gas exchange

3.Results

Closure in about 60%

40% regain 2 or more lines of VA

Idiopathic premacular fibrosis

Cellophane maculopathy

Macular pucker

 

 

 

 

 

 

Translucent epiretinal

Severe retinal wrinkling and

Opaque epiretinal membrane

 

membrane

 

vascular distortion

 

 

Fine retinal striae and mild

Pucker emanating from

May be associated with

 

macular pseudo-hole

 

vascular distortion

 

epicentre

 

 

 

 

 

 

 

 

 

 

 

Cystoid macular oedema ( CMO )

Fluid-filled microcysts in outer plexiform and inner nuclear layer

May lead to lamellar hole formation if longstanding

Important causes of CMO

Retinal vein occlusion

Background diabetic retinopathy

Post-cataract surgery

Intermediate uveitis

Clinical diagnosis of CMO

 

 

 

 

Loss of foveal depression

Retinal thickening

Yellow spot at foveola

Multiple cystoid areas

 

 

 

 

FA of cystoid macular oedema

 

 

 

Early parafoveal leakage

Coalescence of leaking points

Late pooling with

‘flower-petal’ pattern

 

 

 

 

 

Treatment Options of CMO

1.No treatment

Very mild - good VA

Too early - wait for spontaneous improvement

Too late - poor VA (lamellar hole)

Treatment not beneficial - CRVO

2.Laser photocoagulation

Diabetic retinopathy

Branch retinal vein occlusion

3.Periocular steroids

Intermediate uveitis

Post cataract

4.Systemic carbonic anhydrase inhibitors

Intermediate uveitis

Post cataract

Myopic maculopathy

 

Atrophic

 

‘Lacquer cracks’

 

 

 

 

 

 

 

 

Progressive chorioretinal atrophy

Large breaks in Bruch membrane

May be associated with macular hole

Develop in about 5% of highly myopic eyes

 

 

 

 

 

Macular haemorrhage

 

Fuchs spot

 

 

 

 

 

 

 

 

From CNV with lacquer cracks

Secondary pigment proliferation

From lacquer cracks alone

Follows absorption of blood

 

 

 

 

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