- •Foreword
- •Preface
- •Acknowledgments
- •Contents
- •Contributors
- •References
- •References
- •Introduction
- •Eyelid Anatomy
- •Eyelid Skin
- •The Orbicularis Muscle
- •The Orbital Septum
- •The Preaponeurotic Fat Pockets
- •The Major Eyelid Retractors
- •The Sympathetic Eyelid Retractors
- •The Tarsal Plates
- •The Canthal Tendons
- •The Conjunctiva
- •Nerves to the Eyelids
- •Vascular Supply to the Eyelids
- •Upper Eyelid Physiology
- •References
- •Introduction
- •Ocular Surface Disease
- •Medications
- •Prior Ocular and Periocular Surgery
- •Contact Lens Use
- •Miscellaneous Conditions
- •Congenital Ptosis
- •Conclusion
- •References
- •Recognise the Ptosis!
- •Unsafe Ptosis
- •Safe Ptosis
- •Distinguishing Safe Ptosis from Unsafe Ptosis
- •Lifting the Ptotic Lid
- •Contralateral Lid Retraction
- •Proptosis and Enophthalmos
- •Frontalis Overaction
- •The Dry Eye Patient
- •Definitive Examination of the Ptosis
- •Special Diagnostic Tests for Ptosis
- •References
- •Introduction
- •History
- •Physical Exam
- •Determination of Procedure
- •Blood Thinners
- •Patient Expectations
- •When Not to Operate
- •References
- •References
- •Pathogenesis
- •Myogenic Causes
- •Aponeurotic Causes
- •Mechanical Causes
- •Neurogenic Causes
- •Pseudoptosis
- •Examination
- •Preoperative Considerations
- •Surgical Repair
- •Müllerectomy
- •Levator Resection
- •Frontalis Suspension
- •Conclusions
- •References
- •Introduction
- •Background
- •Etiology
- •Clinical Findings
- •Ophthalmic Exam
- •Treatment
- •Conclusions
- •References
- •Introduction
- •Congenital Myogenic Ptosis
- •Acquired Myogenic Ptosis
- •Evaluation of the Patient
- •Treatment
- •References
- •Overview
- •Diagnosis: Clinical
- •Diagnosis: Testing
- •Medical Therapy
- •Surgical Therapy
- •References
- •Introduction
- •Third Nerve (Oculomotor) Palsy
- •Diagnosis
- •Localization of a Third Nerve Palsy
- •Common Etiologies for Third Nerve Palsy
- •Horner Syndrome
- •Diagnosis
- •Pharmacologic Evaluation
- •Localization of Horner Syndrome
- •Radiographic Evaluation
- •Horner Syndrome in Children
- •Treatment
- •References
- •Introduction
- •Iatrogenic Causes of Ptosis
- •Ptosis Postintraocular Surgery
- •Ptosis Posteyelid and Adnexal Procedures
- •Contact Lens Wear
- •Ptosis Following Systemic Interventions
- •Birth Trauma
- •Blunt Trauma
- •Lacerating Trauma
- •Traumatic Ptosis Secondary to Restrictive Scarring
- •Traumatic Ptosis Following Facial Fractures
- •Neurogenic Ptosis Secondary to Trauma
- •Traumatic Superior Orbital Fissure Syndrome
- •Blepharoptosis Secondary to Traumatic Third Nerve Palsy
- •Isolated Neurogenic Ptosis
- •Traumatic “Ptosis” Secondary to Facial Synkinesis
- •References
- •Etiology
- •Evaluation
- •Solutions
- •Conclusion
- •References
- •Introduction
- •Pathophysiology
- •Clinical Evaluation
- •Surgical Management
- •Minimal Lash Ptosis
- •Moderate to Severe Lash Ptosis
- •Conclusion
- •References
- •Introduction
- •Mechanical Measures
- •Lid Crutches
- •Eyelid Taping
- •Glues
- •Apraclonidine
- •Medical Measures: Botulinum Toxin
- •References
- •Introduction
- •Procedure
- •Conclusion
- •Suggested Reading
- •Introduction
- •Principle of the Procedure
- •Methodology of the Procedure
- •References
- •Technique [5]
- •References
- •Introduction
- •Surgical Technique
- •Preservation of the Conjunctiva
- •Discussion
- •References
- •History
- •Mechanism of Action
- •Indications
- •Procedure
- •Description of the Procedure
- •Complications
- •Discussion
- •References
- •Indications
- •Techniques
- •Lamellar Technique
- •En Bloc Technique
- •Challenges and Solutions
- •Pearls
- •References
- •Indications
- •Autologous Tissue for Frontalis Suspension
- •Autogenous Fascia Lata
- •Harvesting Fascia Lata
- •Temporalis Fascia
- •Harvesting Deep Temporalis Fascia
- •Palmaris Longus Tendon
- •Harvesting Palmaris Longus Tendon
- •Frontalis Muscle Flap Advancement
- •Allografts for Frontalis Suspension
- •Preserved Fascia Lata
- •Other Processed Tissues
- •Synthetic Materials for Frontalis Suspension
- •Techniques for Frontalis Suspension
- •Double Triangle or Rhomboid Frontalis Sling
- •Single Pentagonal Frontalis Sling
- •References
- •The Transition to Office-Based Surgery
- •Reasons to Transition
- •Surgical Space and Equipment
- •State Regulations
- •Procedure Selection
- •Patient Selection
- •Evaluating Patients at Risk for Anxiety
- •Nonmedical Prevention of Anxiety
- •Medical Prevention of Anxiety
- •Postoperative Nausea and Vomiting
- •Anesthesia for Surgery
- •Topical Anesthetics
- •Injectable Anesthetics
- •Postoperative Pain Control
- •Conclusion
- •References
- •References
- •References
- •Etiology and Evaluation
- •Treatment
- •Surgical Technique
- •Aporneurotic Ptosis Repair
- •Frontalis Sling
- •Complications
- •Summary
- •Tarsal Switch
- •Severe Horizontal Eyelid Laxity
- •Inadequate Tarsus
- •Neurofibromatosis
- •References
- •Involutional/Aponeurotic ptosis
- •Levator Advancement/Plication
- •Congenital Myogenic Ptosis
- •Frontalis Suspension
- •Levator Resection
- •Maximal Levator Resection
- •Whitnall’s Sling
- •Summary
- •References
- •Introduction
- •Preoperative Factors
- •Intraoperative Factors
- •Postoperative Factors
- •Surgical Approach to Ptosis Reoperation
- •Summary
- •References
- •Entropion
- •Symblepharon
- •Ectropion
- •Contour Deformity
- •Lagophthalmos
- •Eyelid Fold and Crease
- •Conjunctival Prolapse
- •Hemorrhage/Hematoma
- •Infection
- •Conclusion
- •References
- •Twelve Steps to a Successful Surgical Encounter
- •Index
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Minimal Lash Ptosis
The lash ptosis is addressed after the excess skin is removed (to address concurrent dermatochalasis), and the levator is advanced in cases of blepharoptosis. Interrupted bites of 6–0 chromic sutures are used to attach skin to the deep levator aponeurosis just above the superior tarsal border. It is important to note that as the sutures are tightened, often the subsequent sutures are more difficult to place; therefore, we often preplace the sutures prior to tying them down to ensure that each one is appropriately placed. The number of sutures placed depends on the size of the incision; however, we typically place five to six sutures in each eyelid. The preplaced sutures are then tied down and any remaining skin gaps are closed with 6–0 fast-absorbing gut suture. The careful attention to closure, by ensuring the incorporation of the levator aponeurosis, accomplishes two goals. First, it reforms the lid crease, and second, it causes enough of a vertical traction on the anterior lamella to improve the angle of the eyelash orientation. We aim for slight overcorrection, as the lashes are expected to drop slightly in the postoperative period.
Moderate to Severe Lash Ptosis
These cases can be challenging to correct and therefore require a more powerful procedure to improve the vertical vector forces on the lash base. Again, in cases of either acquired or congenital blepharoptosis, we prefer to address this first. Once we have the lid height in the desired position and just prior to closure, we address the lashes. For moderate cases, we like to use a polygalactin double-armed 5–0 or 6–0 horizontal mattress suture through the upper third of the tarsus and exit just above the lash base. These sutures are placed along the same level of the tarsus across the eyelid and exit in the same position immediately above the lash base to ensure symmetric elevation across the lashes. Once tied down, the closure of the skin is completed with a running 6–0 fast-absorbing gut suture.
For more severe cases of lash ptosis, we employ a similar technique as described above with a couple of modifications. Instead of using a dissolvable suture, we like to use a permanent suture, such as silk and remove it around postoperative week 6 to minimize the chance of postoperative descent. In addition, we reinforce the vertical traction by closing the skin similar to that described for minimal lash ptosis.
In patients with FES, the lash ptosis is corrected with a horizontal tightening procedure. The authors’ preference is to use a full-thickness pentagonal wedge resection at the junction of the lateral ¼ and medial ¾ of the eyelid. The eyelid is closed using standard marginal and layered closure techniques. In our experience, rarely will floppy eyelid patients need additional rotational suture techniques as described above. Lash ptosis secondary to cicatricial entropion needs to be corrected by treatment of the underlying etiology having caused, or causing, the cicatricial changes. Once the cicatrix has stabilized, or if protection of the ocular surface warrants sooner intervention, surgery can be undertaken. The approach depends on the severity of the cicatrix, but usually requires incisional relaxation of the cicatrix and placement of a spacer graft. If the anterior lamellar anatomy has not been chronically disrupted, the lash ptosis may resolve; however, residual lash ptosis may need to be addressed secondarily using one of the above procedures.
In all of our techniques we aim for mild to moderate overcorrection, as the lash angle is expected to drop slightly in the postoperative period.
Conclusion
Lash ptosis is associated with various conditions, and the etiology of the lash ptosis may be secondary to the underlying condition, i.e., congenital or acquired blepharoptosis, FES, etc. Preoperatively, it is important to document the severity of the lash ptosis and grade it accordingly in order to determine the best surgical technique to employ.
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The combined approach of addressing the underlying condition along with the severity of the lash ptosis will lead to an improved outcome and the greater likelihood of a satisfied patient.
References
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2.Guimarães FC, Cruz AV. Eyelid changes in longstanding leprosy. Ophthal Plast Reconstr Surg. 1998;14(4):239–43.
3.Casson RJ, Selva D. Lash ptosis caused by latanoprost. Am J Ophthalmol. 2005;139(5):932–3.
4.McNab AA. Floppy eyelid syndrome. Ophthalmology. 1998;105(11):1977–8.
5.Singh AJ, Atkinson PL. Ocular manifestations of congenital lamellar ichthyosis. Eur J Ophthalmol. 2005;15(1):118–22.
6.Mulhern MG, Aduriz-Lorenzo PM, Rawluk D, Viani L, Eustace P, Logan P. Ocular complications of acoustic neuroma surgery. Br J Ophthalmol. 1999;83(12): 1389–92.
7.McNab AA. Floppy eyelid syndrome and obstructive sleep apnea. Ophthal Plast Reconstr Surg. 1997;13(2): 98–114.
8.Culbertson WW, Ostler HB. The floppy eyelid syndrome. Am J Ophthalmol. 1981;92(4):568–75.
9.Netland PA, Sugrue SP, Albert DM, Shore JW. Histopathologic features of the floppy eyelid syndrome: involvement of tarsal elastin. Ophthalmology. 1994;101(1):174–81.
10.Dutton JJ. Surgical management of floppy eyelid syndrome. Am J Ophthalmol. 1985;99(5):557–60.
11.Malik KJ, Lee MS, Park DJJ, Harrison AR. Lash ptosis in congenital and acquired blepharoptosis. Arch Ophthalmol. 2007;125(12):1613–5.
Part IV
Treatment
