- •Chapter 1
- •Ocular Adnexal Lymphoproliferative
- •1.1 Pathogenesis
- •1.2 Chronic Antigen Stimulation
- •1.3 Immunosuppression
- •1.4 Pathology
- •1.5 Cytogenetics
- •1.6 Clinical Features
- •1.7 Imaging Findings
- •1.8 Staging
- •1.9 Positron Emission Tomography
- •1.10 Treatment
- •1.11 Follicular Lymphoma
- •1.12 Mantle Cell Lymphoma
- •1.13 Radiotherapy
- •1.14 Chemotherapy
- •1.15 Immunotherapy
- •1.16 Radioimmunotherapy
- •1.17 Outcome
- •1.18 The Future
- •References
- •Chapter 2
- •2.1 General Introduction
- •2.2 The Aging Process and Facial Analysis
- •2.3 Endoscopic Brow Lift
- •2.3.1 Introduction
- •2.3.2 Endoscopic Browlift Anesthesia Pearls
- •2.3.4 Endoscopic Browlift Postoperative Care Pearls
- •2.4 Upper Blepharoplasty
- •2.4.1 Introduction
- •2.4.2 Patient Evaluation
- •2.4.3 Upper Blepharoplasty Anesthesia Pearls
- •2.4.4 Upper Blepharoplasty Surgical Procedure Pearls
- •2.5 Lower Blepharoplasty, Fillers, and Midface Augmentation
- •2.5.1 Introduction
- •2.5.2 Patient Evaluation
- •2.5.3 Lower Blepharoplasty Anesthesia Pearls
- •2.5.4 Lower Blepharoplasty Surgical Procedure Pearls
- •References
- •Chapter 3
- •3.1 Introduction
- •3.2 What Is the Diagnosis?
- •3.2.1 Pitfalls of Diagnosis
- •3.2.2 A Diagnostic Corticosteroid Trial?
- •3.2.3 The Question of Biopsy
- •3.3 Treatment
- •3.3.1 Corticosteroids
- •3.3.2 Radiation
- •3.3.3 Other Agents
- •3.4 Special Circumstances
- •3.4.1 Pediatric IOIS
- •3.4.2 Sclerosing Pseudotumor
- •3.4.3 Tolosa–Hunt Syndrome
- •References
- •Chapter 4
- •4.1 Introduction
- •4.2 Embryology, Anatomy, Physiology, and Pathophysiology of the Canalicular System
- •4.3 Infective Causes
- •4.3.1 Periocular Herpes Simplex Infection
- •4.3.2 Bacterial Canaliculitis
- •4.4.1 Lichen Planus
- •4.4.2 Ocular Cicatricial Pemphigoid
- •4.5 Iatrogenic Causes
- •4.5.1 Systemic Drugs
- •4.5.1.2 Docetaxel (Taxotere)
- •4.5.2 Radiotherapy
- •4.5.3 Topical Ophthalmic Treatments
- •4.5.3.2 Mitomycin C (MMC) Therapy
- •4.5.4 Lacrimal Stents and Plugs
- •4.6 The Surgical Approach to Managing Canalicular Disease
- •4.6.1 Surgical Technique for Dacryocystorhinostomy with Retrograde Canaliculostomy
- •References
- •Chapter 5
- •5.1 Introduction
- •5.2 Nomenclature
- •5.3 Clinical Manifestations of NF1
- •5.4 Orbitofacial Tumors in NF1
- •5.4.2 Malignant Peripheral Nerve Sheath Tumors
- •5.4.3 Optic Pathway Gliomas
- •5.5 Genetics
- •5.5.1 The NF1 Gene
- •5.5.2 Overlapping NF1-Like Phenotype (SPRED1)
- •5.6.1 Introduction
- •5.7 Surgical Management of Orbitofacial Tumors in NF1
- •5.7.1 Introduction
- •5.7.2 Timing of Surgery
- •5.7.3 Periorbital Involvement
- •5.7.3.1 The Upper Eyelid
- •5.7.3.2 The Lower Eyelid and Midface
- •5.7.4 Orbital Involvement
- •5.7.4.1 Proptosis
- •5.7.4.3 Proptosis Due to Optic Nerve Glioma
- •5.7.4.4 Orbital Enlargement with Dystopia and Hypoglobus
- •5.8 The Natural History of NF1 Tumor Growth from Birth to Senescence
- •References
- •Chapter 6
- •6.1 Introduction
- •6.2 Surgical Anatomy of the Lacrimal Drainage System
- •6.3 Basic Diagnostics for Disorders of the Lacrimal Drainage System
- •6.4 Selective Lacrimal Sac Biopsy in External Dacryocystorhinostomy
- •6.5.1 Case A
- •6.5.2 Case B
- •6.5.3 Case C
- •6.5.4 Case D
- •6.5.5 Case E
- •6.5.6 Case F
- •6.5.7 Case G
- •References
- •Chapter 7
- •7.1 Introduction
- •7.2 Patients and Methods
- •7.2.1 Patients
- •7.2.2 Examination
- •7.3 Results
- •7.3.1 Patient Data
- •7.3.3 Family History
- •7.3.4 Pregnancy History
- •7.3.5 Birth
- •7.3.6 Associated Systemic and Ocular Diseases
- •7.3.8 Neuroradiological Findings (Brain MRI)
- •7.3.9 Nasolacrimal System Findings
- •7.4 Discussion
- •7.4.1 Patients
- •7.4.2 Obstetric and Family History
- •7.4.3 Associated Pathologies
- •7.4.3.1 Ophthalmological Findings in Unilateral Disease
- •7.4.3.2 Neuroradiological Findings
- •7.4.3.3 Systemic Diseases
- •7.4.3.4 Nasolacrimal Duct Findings
- •7.5 Conclusions
- •References
- •Chapter 8
- •8.1 Introduction
- •8.2 Evaluation of Complicated Ptosis
- •8.2.1 Compensatory Eyebrow Elevation
- •8.2.3 Innervation Patterns of the Frontalis Muscle
- •8.2.4 Checklist of Preoperative Evaluation of Complicated Ptosis
- •8.3 Surgical Technique of Levator Muscle Recession
- •8.3.1 Principle
- •8.3.2 Approach to the Levator
- •8.3.3 Partial Levator Recession
- •8.3.4 Total Levator Recession
- •8.3.6 Undercorrection and Overcorrection
- •8.4 Surgical Technique of Brow Suspension
- •8.4.1 Materials for Brow Suspension
- •8.4.1.1 Nonautogenous Materials
- •8.4.1.2 Autogenous Fascia Lata
- •8.4.2 Our Technique of Harvesting Autogenous Fascia Lata
- •8.4.3 Mechanical Principals of Brow Suspension
- •8.4.4 Upper Lid Approach
- •8.4.5 Fascia Implantation
- •References
- •Chapter 9
- •Modern Concepts in Orbital Imaging
- •9.1 Computerized Tomography
- •9.2 Three-Dimensional Imaging
- •9.3 Magnetic Resonance Imaging
- •9.3.1 The T1 Constant
- •9.3.2 The T2 Constant
- •9.3.3 Creating the MR Image
- •9.4 Imaging of Common Orbital Lesions
- •9.4.1 Adenoid Cystic Carcinoma
- •9.4.2 Cavernous Hemangioma
- •9.4.3 Dermoid Cyst
- •9.4.4 Fibrous Dysplasia
- •9.4.5 Lymphangioma
- •9.4.6 Lymphoma
- •9.4.7 Myositis
- •9.4.8 Optic Nerve Glioma
- •9.4.9 Pseudotumor
- •9.4.10 Rhabdomyosarcoma
- •9.6 Positron Emission Tomography
- •9.7 Orbital Ultrasound
- •9.7.1 Physics and Instrumentation
- •9.7.1.1 Topographic Echography
- •9.7.1.2 Quantitative Echography
- •9.7.1.3 Kinetic Echography
- •9.7.2 Extraocular Muscles
- •9.7.3 Optic Nerves
- •References
- •Chapter 10
- •10.1 Introduction
- •10.3 Etiology
- •10.4 Microbiology
- •10.5 Changing Pathogens and Resistance
- •10.5.2 Orbital MRSA
- •10.6 Evaluation of Orbital Cellulitis
- •10.7 Medical Treatment of Orbital Cellulitis
- •10.8 Surgical Treatment of Orbital Cellulitis
- •10.9 Prevention of Orbital Cellulitis After Orbital Fracture
- •References
- •Chapter 11
- •11.1 Clinical Picture
- •11.1.1 Clinical Phases
- •11.2 Ocular Complications
- •11.3 Investigation
- •11.3.1 Angiography
- •11.4 Management
- •11.4.1 Active Nonintervention
- •11.4.2 Indications for Treatment
- •11.5 Modalities of Treatment
- •11.5.1 Steroids
- •11.5.1.1 Topical Steroids
- •11.5.1.2 Intralesional Corticosteroid Injection
- •11.5.1.3 Oral Corticosteroids
- •11.5.2 Interferon-Alfa
- •11.5.3 Vincristine
- •11.5.4 Laser
- •11.5.5 Embolization
- •11.5.6 Surgery
- •References
- •Chapter 12
- •12.1 Introduction
- •12.2 Epidemiology
- •12.3 Biological Behavior and Timing of Metastasis
- •12.4 Lateralization
- •12.5 Localization
- •12.6 Clinical Features
- •12.7 Imaging and Patterns of Orbital Metastatic Disease
- •12.8 Biopsy
- •12.9 Common Types of Orbital Metastases
- •12.9.1 Breast Carcinoma
- •12.9.2 Lung Carcinoma
- •12.9.3 Prostatic Cancer
- •12.9.4 Melanoma
- •12.9.5 Carcinoid Tumor
- •12.11 Treatment
- •12.11.1 Radiotherapy
- •12.11.2 Chemotherapy
- •12.11.3 Hormonal Therapy
- •12.11.4 Surgery
- •12.12 Prognosis and Survival
- •References
- •Chapter 13
- •13.1 Introduction
- •13.2 Rituximab
- •13.3 Yttrium-90-Labeled Ibritumomab Tiuxetan
- •13.4 Imatinib Mesylate
- •13.5 Cetuximab
- •References
- •Chapter 14
- •14.1 Introduction
- •14.2 Porous Orbital Implants
- •14.3 Orbital Implant Selection in Adults
- •14.4 Orbital Implant Selection in Children
- •14.5 Volume Considerations in Orbital Implant Selection
- •14.7 Which Wrap to Use
- •14.8 To Peg or Not to Peg Porous Implants
- •14.9 Summary
- •References
- •Chapter 15
- •15.1 Introduction
- •15.2 Etiology and Presentation
- •15.2.1 Etiology of Orbital Volume Loss
- •15.2.2 Etiology of Periorbital Volume Loss
- •15.2.3 Features of Orbital Volume Loss
- •15.2.4 Features of Periorbital Volume Loss
- •15.3 Background to Injectable Soft-Tissue Fillers
- •15.3.1 Historical Perspective on Volume Replacement
- •15.4 Types of Injectable Soft-Tissue Filler
- •15.4.1 Collagen Fillers
- •15.4.2 Hyaluronic acid Fillers
- •15.5 Treatment Areas
- •15.5.1 Orbit
- •15.5.2 Upper Eyelid and Brow
- •15.5.3 Tear Trough
- •15.5.4 Temple and Brow
- •15.6 Other Periorbital Uses of Injectable Soft-Tissue Fillers
- •15.6.1 Upper Eyelid Loading
- •15.6.2 Lower Eyelid Elevation
- •15.6.3 Treatment of Cicatricial Ectropion
- •15.7 Future Developments
- •References
2.5 Lower Blepharoplasty, Fillers, and Midface Augmentation |
33 |
a |
b |
Fig. 2.14 Preand postoperative internal ptosis repair. Note the improvement in the hollowness of the upper lid and improvement of the lid crease
Summary for the Clinician: Upper
Blepharoplasty
■Remember: The upper eyelid is a continuum of the forehead.
■You cannot cure brow ptosis with a blepharoplasty.
■Preserve the orbicularis.
■Leave at least 23 mm of skin.
■Add volume: Use fat to augment the subbrow region.
2.5Lower Blepharoplasty, Fillers, and Midface Augmentation
2.5.1Introduction
The lower eyelid forms the upper portion of the eyelid/ midface continuum. The midface contributes to the overall contour and shape of the eyelids. Ideally, the lower eyelid follows the contour of the globe until it reaches the inferior orbital rim. At this point, there is a slight concavity. As we follow the lid inferiorly, it becomes more convex. As we age, however, the lower eyelid “lengthens,” and the concavities and convexities change [15].
The goal of lower blepharoplasty is to restore the youthful contour of the lower eyelid and midface [20, 21] (Fig. 2.15). Lower blepharoplasty is not about removing
fat, skin, or muscle. Removal of this tissue may be a means to achieve this goal but not its primary purpose. The lower eyelids are unique in that they are regarded in both their aesthetic and functional senses. It is imperative that the surgeon prioritize function over beauty.
2.5.2Patient Evaluation
Patients presenting for blepharoplasty are evaluated in a similar manner to those patients presenting for all other facial procedures. The eyelids are evaluated in layers: skin, muscle, fat, and bone. The skin of the eyelid is extremely thin and stretches over time. When considering the amount of skin that is “redundant,” it is important to keep in mind that there is a minimum amount of skin that is necessary to ensure proper eyelid function. Patients are evaluated by putting the lower eyelid on maximal stretch. This involves having the patient open his or her mouth and looking up.
The degree of skin excess is then assessed. Again, patients with prominent eyes will require more skin since the eyelid has to cover a greater convex surface. It is important to differentiate between loss of skin elasticity and actual skin excess. Loss of skin elasticity will result in rhytids, and skin may appear redundant. It is tempting to remove this skin. However, removal of this skin may result in lid retraction if there is truly no skin excess. It is often better to laser or peel the skin to improve the elasticity than to remove the skin (Fig. 2.16).
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2 Pearls in Cosmetic Oculofacial Plastic Surgery |
|
|
a |
b |
2
c |
d |
Fig. 2.15 (a, b) The youthful eyelid/midface complex involves a short eyelid and a single midfacial convexity. (c, d) The aged midface involves a long lower eyelid and a double convexity of the midface
|
2.5 |
Lower Blepharoplasty, Fillers, and Midface Augmentation |
35 |
a |
b |
c |
|
Fig. 2.16 (a) Patient appears to have extra lower eyelid skin (b) Excess lower eyelid skin determination: maximal skin stretch can be achieved by having patients open their mouth while looking up. (c) When the skin is in maximal stretch there is little excess noted
Laxity of the orbicularis results in sagging of the lower eyelid and a generalized aged appearance. The pretarsal and preseptal orbicularis are vital for proper eyelid function and position. Laxity of the orbicularis results in bowing of the lower eyelid, lid retraction, and festoon formation [12]. Traditional lower blepharoplasty involved removal of preseptal orbicularis and skin. The removal of this vital muscle invariably resulted in lid retraction to some degree. In some cases, the lids became cicatrized to the orbital rim, resulting in severe retraction and ectropion (Fig. 2.17). Once the muscle was removed, it was difficult to attain proper lid height and contour without extensive surgery, such as midface lifting or insertion of hard palate grafts [38].
As oculoplastic surgeons, we manage many of the complications of lower blepharoplasty. In the 1980s, the trend toward limited skin and muscle excision emerged, and transconjunctival blepharoplasty became popular [3] (Fig. 2.18). The transconjunctival blepharoplasty, however, only addressed the orbital fat and neglected the orbicularis laxity. The addition of pinch skin removal or laser resurfacing of the skin improved the skin but still did not address the underlying orbicularis [5]. Currently, in patients with skin and orbicularis issues, an orbicularis oculi plication blepharoplasty is performed. This procedure is described in Section 2.5.4.
Protrusion of the orbital fat results in fullness of the eyelids above the inferior orbital rim. This fullness is exacerbated by concavities of the midface, where loss of the suborbicularis oculi fat (SOOF) is common. I like to give the following analogy to the patients: The eyelid/
Fig. 2.17 Severe lid retraction and ectropion resulting from aggressive removal of skin and orbicularis during transcutaneous blepharoplasty
midface is similar to a hill and a valley. If one is standing in a deep valley next to a hill, then the hill will appear higher. Once the valley is filled, the hill appears less high. Thus, removal of protruding fat (hill) in the face of a concavity inferior to the fat (valley) will create an even larger
36 |
2 Pearls in Cosmetic Oculofacial Plastic Surgery |
a
2
b
Fig. 2.18 (a and b) Transconjunctival blepharoplasty combined with laser resurfacing: Pre and Postoperative photos
concavity (greater valley). It is imperative that the blepharoplasty surgeon evaluate the contours of the lower eyelid and decide if fat removal, fat addition, or fat repositioning is indicated (Fig. 2.19).
The inferior orbital rim and bony midface are evaluated. The contours of these bones significantly contribute to eyelid position [13]. The relationship between the anterior corneal surface and the inferior orbital rim is evaluated in the sagittal plane. Patients are grouped into three categories: negative vector (the globe projects anterior to the rim), neutral vector (the globe is in line with the inferior rim), and positive vector (the globe projects posterior to the rim) [11] (Fig. 2.20).
Patients with a negative vector configuration have less midfacial support and are more at risk for lid malpositions and contour irregularities after transconjunctival fat removal or transcutaneous blepharoplasty. Patients with prominent eyes and severe negative vector configuration may benefit from midface implants and a midface lift (Fig. 2.21).
The question of fat removal versus fat preservation and repositioning is daunting [17]. I have abided by the
Fig. 2.19 T his patient has mild fat protrusion and significant infraorbital hollowness. The concavity extends from the medial to lateralmost portion of the eyelid. Removal of protruding orbital fat in this patient will make the hollowness worse
following general principals and have managed to get consistent results: In patients with a prominent eye, negative vector, and orbital fat protrusion, a fat-repositioning procedure is performed [42]. In patients with a deeperset eye, positive vector, and orbital fat protrusion, fat removal is acceptable (Table 2.2).
The lower eyelids are probably the most difficult region of the face to rejuvenate. Attaining excellent and consistent results in lower eyelid surgery is dependent on proper evaluation of the anatomic problems and proper solutions to address these problems. Since there are so many anatomic variations among patients, there are multiple procedures that are available for lower eyelid rejuvenation. Often, a patient may require multiple procedures within the context of “blepharoplasty” to address each anatomic abnormality. For example, lower lid fat repositioning may be combined with orbicularis suspension and skin excision. Transconjunctival blepharoplasty may be combined with hyaluronic fillers and a chemical peel [22].
The pearls of lower blepharoplasty are separated into anesthesia and procedure sections.
