- •Chapter 1
- •Ocular Adnexal Lymphoproliferative
- •1.1 Pathogenesis
- •1.2 Chronic Antigen Stimulation
- •1.3 Immunosuppression
- •1.4 Pathology
- •1.5 Cytogenetics
- •1.6 Clinical Features
- •1.7 Imaging Findings
- •1.8 Staging
- •1.9 Positron Emission Tomography
- •1.10 Treatment
- •1.11 Follicular Lymphoma
- •1.12 Mantle Cell Lymphoma
- •1.13 Radiotherapy
- •1.14 Chemotherapy
- •1.15 Immunotherapy
- •1.16 Radioimmunotherapy
- •1.17 Outcome
- •1.18 The Future
- •References
- •Chapter 2
- •2.1 General Introduction
- •2.2 The Aging Process and Facial Analysis
- •2.3 Endoscopic Brow Lift
- •2.3.1 Introduction
- •2.3.2 Endoscopic Browlift Anesthesia Pearls
- •2.3.4 Endoscopic Browlift Postoperative Care Pearls
- •2.4 Upper Blepharoplasty
- •2.4.1 Introduction
- •2.4.2 Patient Evaluation
- •2.4.3 Upper Blepharoplasty Anesthesia Pearls
- •2.4.4 Upper Blepharoplasty Surgical Procedure Pearls
- •2.5 Lower Blepharoplasty, Fillers, and Midface Augmentation
- •2.5.1 Introduction
- •2.5.2 Patient Evaluation
- •2.5.3 Lower Blepharoplasty Anesthesia Pearls
- •2.5.4 Lower Blepharoplasty Surgical Procedure Pearls
- •References
- •Chapter 3
- •3.1 Introduction
- •3.2 What Is the Diagnosis?
- •3.2.1 Pitfalls of Diagnosis
- •3.2.2 A Diagnostic Corticosteroid Trial?
- •3.2.3 The Question of Biopsy
- •3.3 Treatment
- •3.3.1 Corticosteroids
- •3.3.2 Radiation
- •3.3.3 Other Agents
- •3.4 Special Circumstances
- •3.4.1 Pediatric IOIS
- •3.4.2 Sclerosing Pseudotumor
- •3.4.3 Tolosa–Hunt Syndrome
- •References
- •Chapter 4
- •4.1 Introduction
- •4.2 Embryology, Anatomy, Physiology, and Pathophysiology of the Canalicular System
- •4.3 Infective Causes
- •4.3.1 Periocular Herpes Simplex Infection
- •4.3.2 Bacterial Canaliculitis
- •4.4.1 Lichen Planus
- •4.4.2 Ocular Cicatricial Pemphigoid
- •4.5 Iatrogenic Causes
- •4.5.1 Systemic Drugs
- •4.5.1.2 Docetaxel (Taxotere)
- •4.5.2 Radiotherapy
- •4.5.3 Topical Ophthalmic Treatments
- •4.5.3.2 Mitomycin C (MMC) Therapy
- •4.5.4 Lacrimal Stents and Plugs
- •4.6 The Surgical Approach to Managing Canalicular Disease
- •4.6.1 Surgical Technique for Dacryocystorhinostomy with Retrograde Canaliculostomy
- •References
- •Chapter 5
- •5.1 Introduction
- •5.2 Nomenclature
- •5.3 Clinical Manifestations of NF1
- •5.4 Orbitofacial Tumors in NF1
- •5.4.2 Malignant Peripheral Nerve Sheath Tumors
- •5.4.3 Optic Pathway Gliomas
- •5.5 Genetics
- •5.5.1 The NF1 Gene
- •5.5.2 Overlapping NF1-Like Phenotype (SPRED1)
- •5.6.1 Introduction
- •5.7 Surgical Management of Orbitofacial Tumors in NF1
- •5.7.1 Introduction
- •5.7.2 Timing of Surgery
- •5.7.3 Periorbital Involvement
- •5.7.3.1 The Upper Eyelid
- •5.7.3.2 The Lower Eyelid and Midface
- •5.7.4 Orbital Involvement
- •5.7.4.1 Proptosis
- •5.7.4.3 Proptosis Due to Optic Nerve Glioma
- •5.7.4.4 Orbital Enlargement with Dystopia and Hypoglobus
- •5.8 The Natural History of NF1 Tumor Growth from Birth to Senescence
- •References
- •Chapter 6
- •6.1 Introduction
- •6.2 Surgical Anatomy of the Lacrimal Drainage System
- •6.3 Basic Diagnostics for Disorders of the Lacrimal Drainage System
- •6.4 Selective Lacrimal Sac Biopsy in External Dacryocystorhinostomy
- •6.5.1 Case A
- •6.5.2 Case B
- •6.5.3 Case C
- •6.5.4 Case D
- •6.5.5 Case E
- •6.5.6 Case F
- •6.5.7 Case G
- •References
- •Chapter 7
- •7.1 Introduction
- •7.2 Patients and Methods
- •7.2.1 Patients
- •7.2.2 Examination
- •7.3 Results
- •7.3.1 Patient Data
- •7.3.3 Family History
- •7.3.4 Pregnancy History
- •7.3.5 Birth
- •7.3.6 Associated Systemic and Ocular Diseases
- •7.3.8 Neuroradiological Findings (Brain MRI)
- •7.3.9 Nasolacrimal System Findings
- •7.4 Discussion
- •7.4.1 Patients
- •7.4.2 Obstetric and Family History
- •7.4.3 Associated Pathologies
- •7.4.3.1 Ophthalmological Findings in Unilateral Disease
- •7.4.3.2 Neuroradiological Findings
- •7.4.3.3 Systemic Diseases
- •7.4.3.4 Nasolacrimal Duct Findings
- •7.5 Conclusions
- •References
- •Chapter 8
- •8.1 Introduction
- •8.2 Evaluation of Complicated Ptosis
- •8.2.1 Compensatory Eyebrow Elevation
- •8.2.3 Innervation Patterns of the Frontalis Muscle
- •8.2.4 Checklist of Preoperative Evaluation of Complicated Ptosis
- •8.3 Surgical Technique of Levator Muscle Recession
- •8.3.1 Principle
- •8.3.2 Approach to the Levator
- •8.3.3 Partial Levator Recession
- •8.3.4 Total Levator Recession
- •8.3.6 Undercorrection and Overcorrection
- •8.4 Surgical Technique of Brow Suspension
- •8.4.1 Materials for Brow Suspension
- •8.4.1.1 Nonautogenous Materials
- •8.4.1.2 Autogenous Fascia Lata
- •8.4.2 Our Technique of Harvesting Autogenous Fascia Lata
- •8.4.3 Mechanical Principals of Brow Suspension
- •8.4.4 Upper Lid Approach
- •8.4.5 Fascia Implantation
- •References
- •Chapter 9
- •Modern Concepts in Orbital Imaging
- •9.1 Computerized Tomography
- •9.2 Three-Dimensional Imaging
- •9.3 Magnetic Resonance Imaging
- •9.3.1 The T1 Constant
- •9.3.2 The T2 Constant
- •9.3.3 Creating the MR Image
- •9.4 Imaging of Common Orbital Lesions
- •9.4.1 Adenoid Cystic Carcinoma
- •9.4.2 Cavernous Hemangioma
- •9.4.3 Dermoid Cyst
- •9.4.4 Fibrous Dysplasia
- •9.4.5 Lymphangioma
- •9.4.6 Lymphoma
- •9.4.7 Myositis
- •9.4.8 Optic Nerve Glioma
- •9.4.9 Pseudotumor
- •9.4.10 Rhabdomyosarcoma
- •9.6 Positron Emission Tomography
- •9.7 Orbital Ultrasound
- •9.7.1 Physics and Instrumentation
- •9.7.1.1 Topographic Echography
- •9.7.1.2 Quantitative Echography
- •9.7.1.3 Kinetic Echography
- •9.7.2 Extraocular Muscles
- •9.7.3 Optic Nerves
- •References
- •Chapter 10
- •10.1 Introduction
- •10.3 Etiology
- •10.4 Microbiology
- •10.5 Changing Pathogens and Resistance
- •10.5.2 Orbital MRSA
- •10.6 Evaluation of Orbital Cellulitis
- •10.7 Medical Treatment of Orbital Cellulitis
- •10.8 Surgical Treatment of Orbital Cellulitis
- •10.9 Prevention of Orbital Cellulitis After Orbital Fracture
- •References
- •Chapter 11
- •11.1 Clinical Picture
- •11.1.1 Clinical Phases
- •11.2 Ocular Complications
- •11.3 Investigation
- •11.3.1 Angiography
- •11.4 Management
- •11.4.1 Active Nonintervention
- •11.4.2 Indications for Treatment
- •11.5 Modalities of Treatment
- •11.5.1 Steroids
- •11.5.1.1 Topical Steroids
- •11.5.1.2 Intralesional Corticosteroid Injection
- •11.5.1.3 Oral Corticosteroids
- •11.5.2 Interferon-Alfa
- •11.5.3 Vincristine
- •11.5.4 Laser
- •11.5.5 Embolization
- •11.5.6 Surgery
- •References
- •Chapter 12
- •12.1 Introduction
- •12.2 Epidemiology
- •12.3 Biological Behavior and Timing of Metastasis
- •12.4 Lateralization
- •12.5 Localization
- •12.6 Clinical Features
- •12.7 Imaging and Patterns of Orbital Metastatic Disease
- •12.8 Biopsy
- •12.9 Common Types of Orbital Metastases
- •12.9.1 Breast Carcinoma
- •12.9.2 Lung Carcinoma
- •12.9.3 Prostatic Cancer
- •12.9.4 Melanoma
- •12.9.5 Carcinoid Tumor
- •12.11 Treatment
- •12.11.1 Radiotherapy
- •12.11.2 Chemotherapy
- •12.11.3 Hormonal Therapy
- •12.11.4 Surgery
- •12.12 Prognosis and Survival
- •References
- •Chapter 13
- •13.1 Introduction
- •13.2 Rituximab
- •13.3 Yttrium-90-Labeled Ibritumomab Tiuxetan
- •13.4 Imatinib Mesylate
- •13.5 Cetuximab
- •References
- •Chapter 14
- •14.1 Introduction
- •14.2 Porous Orbital Implants
- •14.3 Orbital Implant Selection in Adults
- •14.4 Orbital Implant Selection in Children
- •14.5 Volume Considerations in Orbital Implant Selection
- •14.7 Which Wrap to Use
- •14.8 To Peg or Not to Peg Porous Implants
- •14.9 Summary
- •References
- •Chapter 15
- •15.1 Introduction
- •15.2 Etiology and Presentation
- •15.2.1 Etiology of Orbital Volume Loss
- •15.2.2 Etiology of Periorbital Volume Loss
- •15.2.3 Features of Orbital Volume Loss
- •15.2.4 Features of Periorbital Volume Loss
- •15.3 Background to Injectable Soft-Tissue Fillers
- •15.3.1 Historical Perspective on Volume Replacement
- •15.4 Types of Injectable Soft-Tissue Filler
- •15.4.1 Collagen Fillers
- •15.4.2 Hyaluronic acid Fillers
- •15.5 Treatment Areas
- •15.5.1 Orbit
- •15.5.2 Upper Eyelid and Brow
- •15.5.3 Tear Trough
- •15.5.4 Temple and Brow
- •15.6 Other Periorbital Uses of Injectable Soft-Tissue Fillers
- •15.6.1 Upper Eyelid Loading
- •15.6.2 Lower Eyelid Elevation
- •15.6.3 Treatment of Cicatricial Ectropion
- •15.7 Future Developments
- •References
2.3 Endoscopic Brow Lift |
23 |
a |
b |
Fig. 2.3 (a) Overall loss of facial fat, especially in the temples, buccal space, and jawline. (b) Loss of fat in the periorbital region with gain of fat in the submentum
the face: We analyze the bones, muscles, fat, and skin. We all understand the concept that rebuilding the foundation of the house does little for the outside paint. Similarly, removing protruding eyelid fat in a patient with significant skin changes does little to improve the skin. I find it helpful to have workup sheets available for the evaluation of the patient. This forces the surgeon to analyze the various layers and come up with specific solutions. Table 2.1 is an example of my facial analysis workup sheet.
Summary for the Clinician
■Analyze the face in layers: skin, muscle fat, and bone. Remember, aging occurs in all of these layers.
■A prominent underlying skeleton acts as a scaffold to support the soft tissue.
■Define the problem.
■Design a solution that addresses the specific anatomic abnormality.
2.3Endoscopic Brow Lift
2.3.1Introduction
The reestablishment of the structural integrity of the eyebrow is fundamental to achieving an aesthetically acceptable surgical result for cosmetic and functional periocular surgery [41]. Patients often present to the aesthetic
surgeon complaining of excess upper eyelid skin and request blepharoplasty. However, when the eyebrows are raised to their normal position, there is often less redundant upper eyelid skin than anticipated, and the required amount of skin removal during blepharoplasty is significantly reduced [40, 47]. Malposition of the eyebrows can often be overlooked. Many patients reflexively raise the eyebrows with their frontalis muscles to lift the eyebrow and eyelid tissue out of the visual axis. These patients develop furrows in the forehead region due to the constant contraction of the frontalis muscles (Fig. 2.4). It is the surgeon’s task to ensure that the patient’s frontalis muscles are completely relaxed prior to assessing the eyebrow position and excess upper eyelid skin.
The eyebrow region ages by deflation as well as descent. As we age, we lose volume in the subbrow fat pad known as the ROOF (retro orbicularis oculi fat) [29]. This deflation contributes to the hooding that occurs in the brow and upper eyelid region (Fig. 2.5). The forehead and eyebrows are also under the constant influence of downward forces of both gravity and the periorbital protractor muscles (orbicularis oculi, procerus, and corrugator and depressor supercilii). These downward forces are opposed by the elevating action of the frontalis muscle. In time, this constant “tug-of-war” between the downward and upward forces leads to a series of wrinkles in the forehead and downward displacement of the eyebrows and eyelids. The lateral portion of the brow tends to descend in an inferomedial vector due in part to the lack of frontalis muscle in this
24 |
2 Pearls in Cosmetic Oculofacial Plastic Surgery |
Table 2.1. Jonathan Hoenig, M.D.
New Patient Consultation
2CHIEF COMPLAINT
HISTORY OF PRESENT ILLNESS:
MAJOR CONCERNS:
PHYSICAL EXAMINATION
Scalp: |
|
|
|
|
|
|
|
Hair Density |
|
|
Length of Forehead |
|
|
CM |
|
|
|
|
|
||||
Forehead: |
|
|
|
|
|
|
|
Brow Ptosis |
0 1 2 3 4 |
Forehead Rhytids: |
0 1 2 3 4 |
|
|||
Glabella Rhytids |
0 1 2 3 4 |
Solar Damage: |
0 1 2 3 4 |
|
|||
Bony Contour: |
Normal |
Abnormal |
|
|
|
|
|
|
|
|
|
||||
EYELIDS: |
|
|
|
|
|
Ptosis: |
None |
OD |
OS |
PF |
LF |
Lower Eyelids: |
|
|
MRD1 |
Responds to Neosynephrine |
|
|
|
|
|
|
|
Laxity: |
|
None |
OD _____ |
|
OS _____ |
Retraction: |
|
None |
OD _____ mm |
|
OS_____mm |
Fat Herniation: |
None |
OD 1 2 3 4 |
|
OS 1 2 3 4 |
|
Orbicularis Strength: |
Upper Eyelid: |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
|
|
|
Lower Eyelid |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
Periorbital Rhytids: |
Static: |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
|
|
|
Dynamic: |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
EYELID cont. |
|
|
|
|
|
Skin Excess: |
|
Upper |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
|
|
Lower |
OD 0 1 2 3 4 |
|
OS 0 1 2 3 4 |
Other: |
|
Ectropion |
Entropion |
|
|
Lesions: |
|
|
|
|
|
PHOTOS/DRAWINGS
|
|
|
|
2.3 |
Endoscopic Brow Lift |
25 |
Table 2.1. (continued) |
|
|
|
|
|
|
|
|
|
|
|
|
|
MIDFACE |
|
|
|
|
|
|
Infraorbital bony status: |
Normal |
Hypoplasia |
|
|
|
|
Infraorbital Soft Tissue Dent |
None |
|
1 2 3 4 |
|
|
|
Central Midfacial Contour |
-4 -3 -2 -1 0 1 2 3 4 |
|
|
|
|
|
Lateral Midfacial Contour |
-4 -3 -2 -1 0 1 2 3 4 |
|
|
|
|
|
Festoons: |
None |
|
Preseptal |
Orbital |
Malar |
Mounds |
NASOLABIAL FOLD |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
JOWLS |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
PARACHUTE |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
NECK LIPOMATOSIS |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
PLATYSMAL BANDS |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
SKIN LAXITY |
Right: |
0 1 2 3 4 |
Left: 0 1 2 3 4 |
|
|
|
MIMETIC MUSCLE STRENGTH/NEURO |
|
SENSORY |
|
|
|
|
Frontal: R 0 1 2 3 4 |
L 0 1 2 3 4 |
V-1 |
Normal |
Abnormal |
|
|
Zygomatic: R 0 1 2 3 4 |
L 0 1 2 3 4 |
V-2 |
Normal |
Abnormal |
|
|
Buccal |
R 0 1 2 3 4 |
L 0 1 2 3 4 |
V-3 |
Normal |
Abnormal |
|
Mandibular R 0 1 2 3 4 |
L 0 1 2 3 4 |
G Auricular |
Normal |
Abnormal |
|
|
SKIN |
|
|
|
|
|
|
Fitzpatrick Classification |
1 2 3 4 5 6 |
|
|
|
|
|
Glagou Classification |
1 2 3 4 |
|
|
|
|
|
Lentigines |
Actinic Keratoses: |
|
Acne Vulgaris: |
|
||
Acne Rosacea |
Scars: |
|
|
Pigmentary Disturbances |
|
|
IMPRESSION: |
|
|
|
|
|
|
PLAN: |
|
|
|
|
|
|
Common Risks |
|
|
|
|
|
|
Bleeding |
|
|
Persistent Droopiness |
|
|
|
Bruising |
|
|
Loss of Fat |
|
|
|
Scarring |
|
|
Infection |
|
|
|
Submandibular Gland Ptosis |
|
|
Contour Irregularities |
|
|
|
Change of Vision |
|
|
Need for Additional fat |
|
|
|
Loss of Vision |
|
|
Skin Necrosis |
|
|
|
Eyelid Malposition |
|
|
Persistent Neck Prominence |
|
||
Tearing |
|
|
Visible Submandibular Glands |
|
||
Persistent Wrinkles |
|
|
Extrusion or Infection of Implant |
|
||
Persistent Swelling |
|
|
Sensory and Motor Nerve Damage |
|
||
Changes in Skin Color |
|
|
Loss of Hair |
|
|
|
Asymmetry |
|
|
Persistent Droopiness |
|
|
|
Change in Shape of Eyes or Face |
|
|
Unrealized Expectations |
|
|
|
|
|
|
|
|
|
|
portion of the brow as well as the sphincter action of the orbicularis oculi muscles [14].
There are many methods to raise the ptotic brow [19]. However, it is important for the surgeon to understand that the purpose of a brow lift is partly to raise the brow but more important to reshape the contour of the brow. Most
female patients benefit from elevation of the lateral half of the brow only, thereby restoring the youthful arch of the brow. Men, on the other hand, require a straight brow that is less arched and sits lower than the female brow.
The endoscopic forehead lift has now become the most popular method of raising the eyebrows and forehead.
262 Pearls in Cosmetic Oculofacial Plastic Surgery
2.3.2Endoscopic Browlift Anesthesia Pearls
|
|
Many facial surgical procedures can be performed under |
|
|
|
local anesthesia. The endoscopic brow lift, however, is |
|
2 |
|
||
|
typically performed with intravenous sedation due to the |
||
|
|
difficulty in anesthetizing the glabella region. |
|
|
|
||
|
|
(a) |
Avoid Bleeding: Use 50–100 ml of tumescent anes- |
|
|
|
thesia [24] to balloon up the scalp and forehead tis- |
|
|
|
sues. This compresses the blood vessels, separates out |
|
|
|
the layers, and reduces the chances of bleeding and |
|
|
|
nerve damage. The anesthetic is placed in a ring pat- |
|
|
|
tern starting from just above the ears, across the coro- |
|
|
|
nal line, and across the brows. |
|
|
(b) |
Tractional Nerve Pain: As the brow is released and |
|
Fig. 2.4 T his patient has significant dermatochalasis, eyelid |
|
elevated, there is traction on the sensory nerves. The |
|
ptosis, and brow ptosis. The brow ptosis can be overlooked since |
|
traction extends to the posterior orbit, and the patient |
|
the patient reflexively raises his eyebrows to pull the skin out of |
|
will feel pain and will be quite uncomfortable. To |
|
his visual axis. Note the deep wrinkles of his forehead. The hori- |
|
avoid the pain, perform supraorbital, supratrochlear, |
|
zontal lines are due to frontalis contraction. The vertical lines are |
|
zygomaticotemporal, and zygomaticofacial nerve |
|
sleep lines. These lines form when a pillow pushes his forehead |
|
|
|
|
blocks with Septocaine® or Marcaine®. Extend the |
|
|
tissues medially when sleeping face down |
|
|
|
|
|
supraorbital block into the anterior, superior orbit. |
|
|
(c) |
Decrease the Amount of Sedatives: Have the anesthe- |
|
|
|
siologist sedate the patient with propofol and then |
|
|
|
add 20–30 mg ketamine IV [2]. The ketamine will act |
|
|
|
synergistically with the propofol and allow the anes- |
|
|
|
thesiologist to use less sedative. Giving the propofol |
|
|
|
first will reduce the potential ketamine side effect of |
|
|
|
bad dreams. |
|
|
|
|
|
|
2.3.3 Endoscopic Browlift Surgical |
|
|
|
|
Procedure Pearls |
|
|
|
|
|
|
(a) |
Vertical Incisions: Make all the incisions vertical and |
|
|
|
not horizontal. Vertical incisions, unlike horizontal |
|
|
|
ones, are almost never visible [19] (Fig. 2.6). |
|
|
(b) |
Lacrimal Retractors: Use a lacrimal retractor to keep |
|
|
|
the incisions open. The retractors will compress the |
|
|
|
edge of the incisions, thereby reducing the bleeding. |
|
Fig. 2.5 Deflation of the sub-brow fat pad (ROOF) contributes |
|
The retractors make it easy to insert the endoscope |
|
to the aging appearance of the brow. Reinflation of this region |
|
and prevent blood from getting on the lens (Fig. 2.7). |
|
restores the more youthful three-dimensional contour of the |
(c) |
Blind Dissection: 90% of the surgery can be per- |
|
eyebrow |
|
formed without the use of an endoscope. Blind, sub- |
|
|
|
periosteal dissection can be performed centrally until |
|
This procedure can achieve elevation of the eyebrows and |
|
2 cm above the brow. Laterally, dissection along the |
|
reduction of forehead furrows and glabellar folds. The |
|
deep temporalis fascia can be performed easily with |
|
aesthetic success of the endoscopic approach is similar to |
|
just headlight illumination. |
|
a coronal lift without the need for a large incision. |
(d) |
Periosteal Elevators: A suction elevator with a “lip- |
|
Pearls of the endoscopic lift are separated into three |
|
down” configuration is an excellent tool to dissect |
|
sections: anesthesia, surgical procedure, and postopera- |
|
along the deep temporalis fascia and for the blind |
|
tive care. |
|
subperiosteal dissection (Fig. 2.8). |
