- •Chapter 1
- •Ocular Adnexal Lymphoproliferative
- •1.1 Pathogenesis
- •1.2 Chronic Antigen Stimulation
- •1.3 Immunosuppression
- •1.4 Pathology
- •1.5 Cytogenetics
- •1.6 Clinical Features
- •1.7 Imaging Findings
- •1.8 Staging
- •1.9 Positron Emission Tomography
- •1.10 Treatment
- •1.11 Follicular Lymphoma
- •1.12 Mantle Cell Lymphoma
- •1.13 Radiotherapy
- •1.14 Chemotherapy
- •1.15 Immunotherapy
- •1.16 Radioimmunotherapy
- •1.17 Outcome
- •1.18 The Future
- •References
- •Chapter 2
- •2.1 General Introduction
- •2.2 The Aging Process and Facial Analysis
- •2.3 Endoscopic Brow Lift
- •2.3.1 Introduction
- •2.3.2 Endoscopic Browlift Anesthesia Pearls
- •2.3.4 Endoscopic Browlift Postoperative Care Pearls
- •2.4 Upper Blepharoplasty
- •2.4.1 Introduction
- •2.4.2 Patient Evaluation
- •2.4.3 Upper Blepharoplasty Anesthesia Pearls
- •2.4.4 Upper Blepharoplasty Surgical Procedure Pearls
- •2.5 Lower Blepharoplasty, Fillers, and Midface Augmentation
- •2.5.1 Introduction
- •2.5.2 Patient Evaluation
- •2.5.3 Lower Blepharoplasty Anesthesia Pearls
- •2.5.4 Lower Blepharoplasty Surgical Procedure Pearls
- •References
- •Chapter 3
- •3.1 Introduction
- •3.2 What Is the Diagnosis?
- •3.2.1 Pitfalls of Diagnosis
- •3.2.2 A Diagnostic Corticosteroid Trial?
- •3.2.3 The Question of Biopsy
- •3.3 Treatment
- •3.3.1 Corticosteroids
- •3.3.2 Radiation
- •3.3.3 Other Agents
- •3.4 Special Circumstances
- •3.4.1 Pediatric IOIS
- •3.4.2 Sclerosing Pseudotumor
- •3.4.3 Tolosa–Hunt Syndrome
- •References
- •Chapter 4
- •4.1 Introduction
- •4.2 Embryology, Anatomy, Physiology, and Pathophysiology of the Canalicular System
- •4.3 Infective Causes
- •4.3.1 Periocular Herpes Simplex Infection
- •4.3.2 Bacterial Canaliculitis
- •4.4.1 Lichen Planus
- •4.4.2 Ocular Cicatricial Pemphigoid
- •4.5 Iatrogenic Causes
- •4.5.1 Systemic Drugs
- •4.5.1.2 Docetaxel (Taxotere)
- •4.5.2 Radiotherapy
- •4.5.3 Topical Ophthalmic Treatments
- •4.5.3.2 Mitomycin C (MMC) Therapy
- •4.5.4 Lacrimal Stents and Plugs
- •4.6 The Surgical Approach to Managing Canalicular Disease
- •4.6.1 Surgical Technique for Dacryocystorhinostomy with Retrograde Canaliculostomy
- •References
- •Chapter 5
- •5.1 Introduction
- •5.2 Nomenclature
- •5.3 Clinical Manifestations of NF1
- •5.4 Orbitofacial Tumors in NF1
- •5.4.2 Malignant Peripheral Nerve Sheath Tumors
- •5.4.3 Optic Pathway Gliomas
- •5.5 Genetics
- •5.5.1 The NF1 Gene
- •5.5.2 Overlapping NF1-Like Phenotype (SPRED1)
- •5.6.1 Introduction
- •5.7 Surgical Management of Orbitofacial Tumors in NF1
- •5.7.1 Introduction
- •5.7.2 Timing of Surgery
- •5.7.3 Periorbital Involvement
- •5.7.3.1 The Upper Eyelid
- •5.7.3.2 The Lower Eyelid and Midface
- •5.7.4 Orbital Involvement
- •5.7.4.1 Proptosis
- •5.7.4.3 Proptosis Due to Optic Nerve Glioma
- •5.7.4.4 Orbital Enlargement with Dystopia and Hypoglobus
- •5.8 The Natural History of NF1 Tumor Growth from Birth to Senescence
- •References
- •Chapter 6
- •6.1 Introduction
- •6.2 Surgical Anatomy of the Lacrimal Drainage System
- •6.3 Basic Diagnostics for Disorders of the Lacrimal Drainage System
- •6.4 Selective Lacrimal Sac Biopsy in External Dacryocystorhinostomy
- •6.5.1 Case A
- •6.5.2 Case B
- •6.5.3 Case C
- •6.5.4 Case D
- •6.5.5 Case E
- •6.5.6 Case F
- •6.5.7 Case G
- •References
- •Chapter 7
- •7.1 Introduction
- •7.2 Patients and Methods
- •7.2.1 Patients
- •7.2.2 Examination
- •7.3 Results
- •7.3.1 Patient Data
- •7.3.3 Family History
- •7.3.4 Pregnancy History
- •7.3.5 Birth
- •7.3.6 Associated Systemic and Ocular Diseases
- •7.3.8 Neuroradiological Findings (Brain MRI)
- •7.3.9 Nasolacrimal System Findings
- •7.4 Discussion
- •7.4.1 Patients
- •7.4.2 Obstetric and Family History
- •7.4.3 Associated Pathologies
- •7.4.3.1 Ophthalmological Findings in Unilateral Disease
- •7.4.3.2 Neuroradiological Findings
- •7.4.3.3 Systemic Diseases
- •7.4.3.4 Nasolacrimal Duct Findings
- •7.5 Conclusions
- •References
- •Chapter 8
- •8.1 Introduction
- •8.2 Evaluation of Complicated Ptosis
- •8.2.1 Compensatory Eyebrow Elevation
- •8.2.3 Innervation Patterns of the Frontalis Muscle
- •8.2.4 Checklist of Preoperative Evaluation of Complicated Ptosis
- •8.3 Surgical Technique of Levator Muscle Recession
- •8.3.1 Principle
- •8.3.2 Approach to the Levator
- •8.3.3 Partial Levator Recession
- •8.3.4 Total Levator Recession
- •8.3.6 Undercorrection and Overcorrection
- •8.4 Surgical Technique of Brow Suspension
- •8.4.1 Materials for Brow Suspension
- •8.4.1.1 Nonautogenous Materials
- •8.4.1.2 Autogenous Fascia Lata
- •8.4.2 Our Technique of Harvesting Autogenous Fascia Lata
- •8.4.3 Mechanical Principals of Brow Suspension
- •8.4.4 Upper Lid Approach
- •8.4.5 Fascia Implantation
- •References
- •Chapter 9
- •Modern Concepts in Orbital Imaging
- •9.1 Computerized Tomography
- •9.2 Three-Dimensional Imaging
- •9.3 Magnetic Resonance Imaging
- •9.3.1 The T1 Constant
- •9.3.2 The T2 Constant
- •9.3.3 Creating the MR Image
- •9.4 Imaging of Common Orbital Lesions
- •9.4.1 Adenoid Cystic Carcinoma
- •9.4.2 Cavernous Hemangioma
- •9.4.3 Dermoid Cyst
- •9.4.4 Fibrous Dysplasia
- •9.4.5 Lymphangioma
- •9.4.6 Lymphoma
- •9.4.7 Myositis
- •9.4.8 Optic Nerve Glioma
- •9.4.9 Pseudotumor
- •9.4.10 Rhabdomyosarcoma
- •9.6 Positron Emission Tomography
- •9.7 Orbital Ultrasound
- •9.7.1 Physics and Instrumentation
- •9.7.1.1 Topographic Echography
- •9.7.1.2 Quantitative Echography
- •9.7.1.3 Kinetic Echography
- •9.7.2 Extraocular Muscles
- •9.7.3 Optic Nerves
- •References
- •Chapter 10
- •10.1 Introduction
- •10.3 Etiology
- •10.4 Microbiology
- •10.5 Changing Pathogens and Resistance
- •10.5.2 Orbital MRSA
- •10.6 Evaluation of Orbital Cellulitis
- •10.7 Medical Treatment of Orbital Cellulitis
- •10.8 Surgical Treatment of Orbital Cellulitis
- •10.9 Prevention of Orbital Cellulitis After Orbital Fracture
- •References
- •Chapter 11
- •11.1 Clinical Picture
- •11.1.1 Clinical Phases
- •11.2 Ocular Complications
- •11.3 Investigation
- •11.3.1 Angiography
- •11.4 Management
- •11.4.1 Active Nonintervention
- •11.4.2 Indications for Treatment
- •11.5 Modalities of Treatment
- •11.5.1 Steroids
- •11.5.1.1 Topical Steroids
- •11.5.1.2 Intralesional Corticosteroid Injection
- •11.5.1.3 Oral Corticosteroids
- •11.5.2 Interferon-Alfa
- •11.5.3 Vincristine
- •11.5.4 Laser
- •11.5.5 Embolization
- •11.5.6 Surgery
- •References
- •Chapter 12
- •12.1 Introduction
- •12.2 Epidemiology
- •12.3 Biological Behavior and Timing of Metastasis
- •12.4 Lateralization
- •12.5 Localization
- •12.6 Clinical Features
- •12.7 Imaging and Patterns of Orbital Metastatic Disease
- •12.8 Biopsy
- •12.9 Common Types of Orbital Metastases
- •12.9.1 Breast Carcinoma
- •12.9.2 Lung Carcinoma
- •12.9.3 Prostatic Cancer
- •12.9.4 Melanoma
- •12.9.5 Carcinoid Tumor
- •12.11 Treatment
- •12.11.1 Radiotherapy
- •12.11.2 Chemotherapy
- •12.11.3 Hormonal Therapy
- •12.11.4 Surgery
- •12.12 Prognosis and Survival
- •References
- •Chapter 13
- •13.1 Introduction
- •13.2 Rituximab
- •13.3 Yttrium-90-Labeled Ibritumomab Tiuxetan
- •13.4 Imatinib Mesylate
- •13.5 Cetuximab
- •References
- •Chapter 14
- •14.1 Introduction
- •14.2 Porous Orbital Implants
- •14.3 Orbital Implant Selection in Adults
- •14.4 Orbital Implant Selection in Children
- •14.5 Volume Considerations in Orbital Implant Selection
- •14.7 Which Wrap to Use
- •14.8 To Peg or Not to Peg Porous Implants
- •14.9 Summary
- •References
- •Chapter 15
- •15.1 Introduction
- •15.2 Etiology and Presentation
- •15.2.1 Etiology of Orbital Volume Loss
- •15.2.2 Etiology of Periorbital Volume Loss
- •15.2.3 Features of Orbital Volume Loss
- •15.2.4 Features of Periorbital Volume Loss
- •15.3 Background to Injectable Soft-Tissue Fillers
- •15.3.1 Historical Perspective on Volume Replacement
- •15.4 Types of Injectable Soft-Tissue Filler
- •15.4.1 Collagen Fillers
- •15.4.2 Hyaluronic acid Fillers
- •15.5 Treatment Areas
- •15.5.1 Orbit
- •15.5.2 Upper Eyelid and Brow
- •15.5.3 Tear Trough
- •15.5.4 Temple and Brow
- •15.6 Other Periorbital Uses of Injectable Soft-Tissue Fillers
- •15.6.1 Upper Eyelid Loading
- •15.6.2 Lower Eyelid Elevation
- •15.6.3 Treatment of Cicatricial Ectropion
- •15.7 Future Developments
- •References
Chapter 2 |
|
Pearls in Cosmetic Oculofacial |
2 |
Plastic Surgery |
Jonathan A. Hoenig
Core Messages
■Analyze the Face in Layers: Aging occurs in the skin, muscle, fat, and bony layers. The key for successful and consistent results is to define the anatomic problem and select procedures that address these problems.
■Do not sacrifice function for beauty. It is imperative that the patient and surgeon come to an understanding that function always supersedes beauty. The eyelids are unique in that they serve a vital function: to protect the eye. It is sometimes difficult for the patient to understand that, for example, the wrinkles that are still present after surgery are in actuality the skin necessary for eyelid closure.
■Endoscopic Brow Lift: The goal of a brow lift is less about lifting and more about contouring the shape of the brow. In women, the brow is elevated in a superior/medial vector. In men, the brow is elevated superiorly so that the brow becomes
straight and forms a T configuration with the nose.
■Upper Blepharoplasty: An upper blepharoplasty procedure cannot cure brow ptosis. If the brow is ptotic, correct the brow. The goal of upper blepharoplasty is definition and subbrow fullness. Blepharoplasty is not about how much skin and fat you take out but how much tissue you leave in. Preserving the pretarsal and preseptal orbicularis is necessary for proper eyelid function.
■Lower Blepharoplasty: The goal of lower blepharoplasty is restoration of the youthful contour of the eyelid and midface. This consists of a vertically short lower eyelid and a full, convex midface. Analysis of the lower eyelid involves the relationship between the globe and inferior orbital rim projection, orbital fat protrusion, and midfacial fat loss and skin elasticity versus excess.
2.1General Introduction
Twenty years ago, life for the oculoplastic surgeon was relatively simple. If a patient presented with a problem, there were limited options to address the problem. As surgeons, we were good at removing tissue: fat, muscle, and skin. For example, if a patient presented with lower eyelid “bags,” the fat was always removed, while the skin would either be removed or be resurfaced. Most patients were treated in a similar manner despite significant anatomic differences. Most surgeons and patients were relatively happy with the surgical results. The reality is that we did not know any better. Then came the Internet.
The Internet allowed patients and physicians to share and gather information. We all became more sophisticated. A variety of injectable fillers came on the market. Our whole philosophy of the aging process suddenly changed.
Today, we are blessed with an array of options to rejuvenate the periocular region. There are multiple surgical and nonsurgical options. However, with all the various options, confusion ensues. What technique do we use to rejuvenate the lower eyelids? Do we use filler or fat? Who needs a transcutaneous blepharoplasty, and who is a better candidate for a transconjunctival incision? I often hear, “What about a midface lift, doctor?” “My friend read on the Internet that to fix lower eyelid bags you need to put in an orbital rim implant.” “How about a laser or a peel?” The combination of solutions can get dizzying.
This chapter summarizes my personal experience and philosophy on what techniques seem to be the most effective. Many of these ideas are based on work described by others. The purpose of this chapter is to show the reader and surgeon my path through the mountain of options that are now available. The chapter is organized into sections for brow lifts, upper blepharoplasty, and lower
22 |
2 Pearls in Cosmetic Oculofacial Plastic Surgery |
blepharoplasty (which also includes information on fillers and midface augmentation).
22.2 The Aging Process and Facial Analysis
The face is created in layers. The sturdiest of these layers is the skeletal bones. The contour and configuration of these bones truly defines the facial shape and soft tissue positions. In many instances, prominent bony contours act as a scaffold and prevent soft tissue descent. This is an important concept since augmenting the bones and deep tissues with implants, fat, or fillers will lift the face to some degree (Fig. 2.1).
The outermost layer of the face is the skin, which bears the brunt of environmental exposure. Between the bones and skin lay fat and muscular layers. In general, there are superficial and deep muscular layers known as the SMAS (superficial musculoaponeurotic system) and DMAS (deep musculoaponeurotic system) [32, 31, 46].
Aging occurs in all four layers: bone, muscle, fat, and skin. As we age, we lose bone around the eyes and mouth [30]. The vertical height of the orbit elongates, and the inferior orbital rim and maxillary face retrude [35]. There is also loss of bone in the mandible, and it also has a loss of its vertical dimension (Fig. 2.2). The muscles of the face stretch and become ptotic, resulting in jowls and neck laxity. There is loss of fat in the eyelid region, cheeks, and buccal space (Fig. 2.3). Some patients gain fat in the jowl region and neck. Finally, the skin undergoes significant changes [25]. The skin thins by losing dermal thickness.
a
Fig. 2.1 (a and b) Two patients who are of the same age. Patients with prominent bony contour have less soft tissue ptosis. The heart-shaped face is considered more youthful than the rectangular-shaped face
The dermal collagen becomes irregular and disorganized. Clinically, the skin has an increase in pigmented spots and wrinkles.
Analyzing the face in layers and defining the problem in each layer is paramount to designing a specific individualized solution for each patient. This approach allows the surgeon to decide which procedure and treatment are needed for each patient. I like to compare facial surgery to building a house. When a house is constructed or repaired, the contractor looks at the foundation, internal beams, walls, and finally the paint. This is analogous to
Fig. 2.2 Loss of bone in the mandible contributes to the appearance of a jowl (arrow). This prejowl sulcus is often due to loss of bone in this region
b
