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Ординатура / Офтальмология / Английские материалы / Clinical Medicine in Optometric Practice_Muchnick_2007

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The examiner should ask whether close family members have similar rashes. Patients should reveal whether this has happened before, if so, how it was treated, and whether it is currently being treated.

Overall skin color may change because of the presence of systemic disease, as in cases of jaundice, which produces a yellow skin tone caused by liver problems. Localized skin color changes around the head and neck may be to the result of aging, neoplastic changes, or medications.

Generalized itching, or pruritus, may be caused by biliary cirrhosis, or cancers, such as pancreatic or lymphoma. Localized pruritus is often associated with a contact dermatitis rash, as in cases of poison ivy, or viral infection, as in a herpes zoster outbreak. Changes in hair density and texture may be related to systemic diseases, diet and medications. Ovarian, adrenal, and pituitary tumors may cause significant changes in hair texture.

EXAMINATION OF THE EYE

After the skin evaluation, the clinician examines the eyes. Significant eye symptoms include loss of visual acuity, field loss, eye pain, redness, diplopia, tearing, discharge, or trauma. This examination includes a visual acuity measurement, confrontational visual field testing, pupil testing, extraocular muscle motility assessment, and fundus evaluation with an ophthalmoscope. The skin around the eyes is checked carefully for any lesions, such as basal cell carcinoma. Basal cell lesions can appear as small, red, or bleeding sores found especially around the eyes and bridge of the nose. Last, the cornea is evaluated for pterygium, and the lid is everted to look for conjunctival cysts, lumps, or changes in color. An in-depth discussion of the ocular portion of the physical examination is omitted in deference to the reader’s expertise.

EXAMINATION OF THE NOSE

Significant nasal symptoms include frequent nosebleeds, nasal discharge, nasal obstruction, sinus infection, and hay fever. Frequent nosebleeds, or epistaxis, are usually caused by nose picking, but malignancy must be considered. Examination of the nose requires inspection of its outer detail as well as its internal structures, which are viewed with the assistance of a nasal illuminator (Figure 2-10). Examination of the nasal mucosa indicates whether it is swollen and pale (as in allergic rhinitis), swollen and red (as in a viral rhinitis), and dry and cracking (from environmental changes such as dryness). By obtaining a nasal illuminator head for the ophthalmoscope, the optometrist can easily perform this examination. A practitioner can often discover clues to illicit drug use, such as punctate ulcerations from cocaine or ac-

THE PHYSICAL EXAMINATION

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FIGURE 2-10 The nasal illuminator. The nasal illuminator aids in the evaluation of the nose.

tual perforation of the nasal septum from a vasoconstricting illicit drug. Nasal tumors can be detected through careful observation.

A bloody discharge is caused by rupture of the superficial mucosal vessels. Known as epistaxis, this discharge may result from trauma, bleeding disorders, systemic hypertension, chronic sinusitis, or malignancy. Inflammation of the nasal mucosa, or rhinitis, may be allergic or non-allergic in etiology. Symptoms include nasal obstruction with itchiness and sneezing associated with a clear, watery nasal discharge.

EXAMINATION OF THE MOUTH

Significant complaints of oral cavity problems include bleeding gums, frequent sore throats, dysphonia (hoarseness, voice changes), and postnasal drip. Dysphagia, or difficulty swallowing, is a significant symptom of obstruction or neurologic problems. The oral cavity is examined with a light and a tongue depressor to allow visualization of all structures (Figure 2-11). The posterior pharynx and tonsils are examined for inflammation, infection, enlargement, and abnormal lesions that might be cancerous.

The underside of the tongue and the area between the teeth and the lips should be examined to identify premalignant or potentially malignant conditions. Normal variations, such as torus palatinus, a bony structure emanating from the hard palate, must be differentiated from more serious conditions. When anything abnormal is visualized—whether it is a lump, a bony prominence, a white plaque known as leukoplakia, or a red plaque known as erythroplakia—it should be palpated to provide a more complete description. Any abnormalities noted on the oral examination warrant a referral to the family physician or dentist.

18 DIAGNOSTIC PROCEDURES

FIGURE 2-11 Examination of the mouth. A penlight and optional tongue depressor are used to evaluate the teeth, gums, buccal mucosa, tongue, tonsils, uvula, and lips.

Pain in the teeth is most commonly a sign of underlying gum disease, or gingivitis. Patients who complain of tooth pain during physical exertion, however, may have chest pain from angina referred to their teeth. Oral ulceration may result from trauma, malignancy, or infections. For example, in all cases of uveitis the buccal mucosa must be explored for ulcers that may be related to Behçet’s disease.

All patients with dry-eye syndrome should be asked about the presence of a dry mouth. This may occur in aging, Sjögren’s syndrome, and in the use of certain medications. Oral examination may help to confirm trauma to the orbital floor with subsequent blow-out fracture. Because the inferior orbital contents prolapse downward through the fracture, an entrapped branch of the fifth cranial nerve may cause reduced sensation to part of the upper gum. First, the wooden end of a sterile cotton-tipped applicator is gently pressed against the gum behind the incisor (“eye tooth”) on the uninvolved side. Next, the applicator is pressed into the gum behind the incisor tooth of the involved side. The patient is asked to compare the sensation on either side. If the gum on the side of the orbital trauma is numb when compared with the sensation on the uninvolved side, then a blow-out fracture is likely.

If the patient reveals an oral mass or bleeding in the history, he or she should be referred to his or her physician to evaluate for a possible trauma.

EXAMINATION OF THE EAR

Significant ear complaints include hearing loss, otorrhea (ear discharge), vertigo (dizziness), otalgia (pain), tinnitus (ringing in the ears) and ear infection. Examination of the ear requires observation of its external structures for abnormalities such as squamous cell

carcinoma. The examiner then inspects the ear canal and drum with the assistance of an otoscope. Significant signs of inflammation include redness or swelling of the canal and drum and a loss of the drum light reflex, suggestive of fluid behind the drum. An excessive accumulation of cerumen (ear wax) can block the canal or press against the tympanic membrane and produce hearing loss.

Two hearing tests can easily be performed by the optometrist in the office. Both tests make use of a 512-Hz tuning fork. The Rinne test involves placement of a vibrating tuning fork on the mastoid sinus of the patient (Figure 2-12). The patient hears the sound by bone conduction. When the sound subsides, the patient signals the examiner, who then removes the fork and brings it to the opening of the patient’s ear. The sound should still

A

B

FIGURE 2-12 The Rinne auditory test. A, A vibrating tuning fork is placed on the mastoid process. B, When the patient no longer hears a sound, the tuning fork is removed and brought to the ear opening. The sound should continue to be audible to the patient.

THE PHYSICAL EXAMINATION

19

Lumps along the sides of the neck are suggestive of enlarged lymph nodes. Enlarged salivary glands can be observed and palpated under the chin. Dilation of the veins of the neck may suggest congestive heart failure.

Palpation of the neck is the most critical part of the examination. The examiner should palpate the thyroid and, if an enlarged thyroid is detected, ask the patient to swallow a sip of water. The act of swallowing allows the thyroid to move up and down, making it easier to palpate (Figure 2-14). The size, symmetry, and consistency

FIGURE 2-13 The Weber test. A vibrating tuning fork is placed in the center of the patient’s forehead. Normally the vibration sounds are equally loud to both ears. A conduction deficit lateralizes the sound to the affected side. A sensorineural deficit lateralizes the sound to the unaffected side.

be audible, because air conduction is superior to bone conduction. If the patient has a conductive hearing loss, then bone conduction, which bypasses the obstruction, will be superior to air conduction.

If the patient has a sensorineural deficit, then air conduction will be better than bone conduction be- A cause the middle ear amplifies the sound the same amount in both positions.

A second method, the Weber test, also makes use of a vibrating 512-Hz tuning fork. This time the tuning fork is placed on the center of the patient’s forehead. The Weber test evaluates bone conduction between the ears. Normally, the patient hears a sound equally in both ears. If the sound is louder in one ear, that ear may have a conduction deficit. To prove this principle, plug one ear with a finger to produce a conduction deficit and hum softly. The sound seems louder in the occluded ear. Of course, if a sensorineural deficit is present in one ear, the sound is lateralized to the unaffected side (Figure 2-13).

EXAMINATION OF THE NECK

After examination of the head, eyes, ears, nose, and mouth, which requires primarily observational skills with the assistance of instruments, the clinician should focus on the neck. Significant presenting symptoms of neck problems include pain, goiter, tenderness, neck stiffness, and anomalous lumps. The multiple structures in the neck should be observed and carefully palpated.

First, the midline position of the trachea is confirmed by observation. Next, the neck is inspected for any asymmetry or visible abnormal lumps. An enlarged central lump is suggestive of an enlarged thyroid, or goiter.

B

FIGURE 2-14 Palpation of the thyroid. A, Anterior approach. The patient tilts his chin slightly to the right as the examiner grasps the right sternocleidomastoid muscle and displaces the larynx to the patient’s right side by use of the examiner’s left hand. The patient swallows and the right lobe of the thyroid is felt moving up and down against the examiner’s right thumb and fingers. The process is reversed for the left lobe. B, Posterior approach. The examiner places both hands on the patient’s sternocleidomastoid muscles. The left hand pushes the trachea to the right while the patient swallows. The right lobe is felt against the sternocleidomastoid muscle with the right hand. The process is reversed for the left lobe.

20 DIAGNOSTIC PROCEDURES

FIGURE 2-15 Palpation of preauricular lymph node. The preauricular lymph node is found just in front of the ear where the jaw articulates with the cranium. This node may become enlarged and painful with viral conjunctivitis.

of the thyroid should be described. The optometrist should palpate the thyroid of any patient exhibiting signs of Graves’ disease (see Chapter 12).

The lymph nodes should be described in the entire neck and head area. If inflammation of the eye is present, the optometrist should examine the preauricular lymph nodes, which are the major area of lymph drainage (Figure 2-15). Lymph nodes occur along the jugular vein in the neck, across the back (the posterior cervical lymph nodes), behind the ear, under the jawbone (the submandibular lymph nodes), and under the chin (the submental lymph nodes).

Any lymph node enlargement must be explained, because it is a key sign in many diseases. Such enlargement is usually the result of inflammation or infection, but lymph nodes that grow and become firm, hard, or irregular may indicate a lymph node tumor known as lymphoma. In other situations, a tumor arising elsewhere in the body can metastasize and cause enlarged and hardened lymph nodes.

Lymph nodes must be palpated in the supraclavicular area just above the clavicle. Lymph nodes below the clavicle are within the purview of the patient’s PCP and if noted on history to be painful or palpable, a referral is mandatory. The patient’s PCP will then palpate the lymph nodes in the axilla (armpit), and in the inguinal (groin) area. Although painful lymph nodes are often associated with inflammation or infection, painless lymph nodes can also be associated with malignant change.

sounds, called bruits, sometimes requires a carotid work-up (Figure 2-16). Causes of carotid bruits include excessive blood flow (in a young person), transmission of a murmur from the heart, or narrowing of the carotid arteries. Any patient seen with the ocular symptoms of a transient ischemic attack warrants a carotid auscultation, because carotid plaques may occlude the ocular blood flow, causing sudden loss of vision. Usually these changes are monocular when they originate in the carotid artery. Whether or not the optometrist hears a bruit, the patient requires a carotid work-up by a vascular specialist as long as all other causes of the transient attack have been eliminated (see Chapter 6).

The carotid arteries are located lateral to the thyroid cartilage between the trachea and sternocleidomastoid muscle. Fatty accumulations of lipid, fibrin, and cholesterol may develop on the inner walls of the carotid artery. The formation of this thrombus may grow to eventually cause complete blockage, or stenosis, of the artery. Reduced blood perfusion to the brain leads to cerebral ischemia and possible stroke.

Alternatively, pieces of the carotid thrombus may break off and travel downstream to the vascular system of the eye or brain. These emboli may temporarily lodge in a blood vessel in the eye, causing transient, unilateral blackouts of vision. Likewise, the embolus may lodge in a cerebral vessel causing episodic and variable loss of neurological function. These TIAs, whether ocular or cerebral, indicate the increased risk of stroke. Even in the absence of symptoms, an ocular examination of the fundus may uncover Hollenhorst plaques located in the retinal arterial tree. These

AUSCULTATION OF THE CAROTID ARTERY

An important examination, auscultation of the carotid arteries involves listening for abnormal sounds suggestive of narrowing of the artery. Detection of these

FIGURE 2-16 Auscultation for carotid bruit. Thrombus development in the carotid artery causes the normally smooth and laminar blood flow to become disturbed and produce a “whooshing” sound heard between the two quick beats of the carotid pulse. Instead of the normal “blub-dup, blub-dup” the sound becomes “blub-woosh-dup, blub-woosh-dup.

emboli, composed of fibrin, calcium, or cholesterol, may have originated in the carotid system.

A patient with a history of ocular or cerebral TIAs, or in whom plaques are found, must therefore have an evaluation of their carotid arteries. Normal blood flow through the carotid artery is laminar in nature and therefore fairly silent when auscultated. The sound generated by auscultation of the normal carotid artery consists of the beat of the heart as transmitted up the vascular tree. The sound is best mimicked as “Blubdup, blub-dup, blub-dup.”

The presence of a thrombus creates turbulence in the smooth flow of blood. This sound, or bruit, is heard between the quick beats of the heart. The sound thus auscultated is best mimicked as “Blub-whoosh- dup, blub-whoosh-dup, blub-whoosh-dup.”

For auscultation of the carotid artery, the patient should ideally lie supine, but can be seated comfortably, and facing the examiner. Tight collars should be loosened. The patient should be instructed to turn his or her head slightly to the left, thus exposing the right carotid artery to the examiners’ stethoscope. Because a bruit sounds like a rush of air, the patient should be asked to hold his or her breath during the test.

Either the flat head or the bell of the stethoscope can be used for examination. The head of the stethoscope is placed over the carotid artery just superior to the clavicle. The examiner asks the patient to hold his or her breath, and the carotid is auscultated during 4 or 5 beats of the heart. Next, the patient is asked to breathe and the head of the stethoscope is brought up to the middle of the neck. The process is then repeated. Finally, the head of the stethoscope is brought to the angle of the jaw and the carotid is once again auscultated. The process is repeated on the other carotid.

Detection of a carotid bruit mandates a carotid artery and cardiovascular work-up to search for thrombus and cardiovascular disease. On finding a bruit, the examiner should take a pen or indelible ink and circle the area of the bruit on the neck of the patient. Referral should be made within days to the patient’s PCP for confirmation of the bruit and initiation of a vascular work-up. The circled area on the patient’s neck should last until they see their PCP and serves as a visual queue as to the location of the bruit and will help the PCP localize the involved area. If the stethoscope is offset by just a short distance from the bruit, it may not be auscultated and the carotid thrombus may be missed.

Any patient with TIAs should have a carotid workup even in the absence of a bruit. The work-up should be done because auscultation of the carotid arteries is at best a screening technique, and a carotid thrombus may not result in a bruit. In addition, the carotid artery must be more than 50% occluded for the bruit to be produced. But if the carotid is occluded by more than about 85%, the artery may be too narrow to pro-

THE PHYSICAL EXAMINATION

21

duce a bruit. This circumstance yields a false-negative result.

The detection of a bruit also mandates a diligent search for Hollenhorst plaques in the retinal arterial trees of both eyes. This search is best conducted by a pupil dilation and use of a hand-held stereoscopic fundus lens at the slit lamp. The retinal arterial plaque will appear as a bright white, yellow, or gray mass lodged most commonly at the bifurcation of a retinal artery or arteriole. Blood may be seen distal to the plaque within the retinal vascular tree. When visualized through the binocular indirect ophthalmoscope head loop, the plaque will appear as a glistening white dot within a retinal arteriole.

The carotid artery should be palpated to gain information regarding poor blood flow and changes in the heart valves, but it is important never to palpate a carotid artery in which a bruit or thrombus has been discovered. Physical pressure on the plaque may cause it to dislodge and embolize.

EXAMINATION OF THE LUNGS

Patients with lung problems (see Chapter 9) may complaint of cough, chest pain, dyspnea (difficulty breathing or shortness of breath), sputum production, hemoptysis (coughing up blood), or a history of lung disease, such as asthma, emphysema, tuberculosis, or sarcoidosis. The clinician should examine the lungs by inspecting for abnormalities in symmetry, scoliosis (lateral curvature of the spine), and audible wheezes during inspiration or expiration.

After this evaluation, the examiner percusses the lung by pressing on the patient’s chest or back with the middle finger of one hand and striking it with the middle finger of the other hand. A resonant note indicates hollowness produced by the presence of air. A dull note suggests fluid, which can accumulate in a patient with pneumonia or heart failure. An abnormal accumulation of fluid in the pleural sac is known as pleural effusion.

The stethoscope is then used to listen for abnormal lung sounds. In the healthy patient, the practitioner should be able to hear air coming into the lungs on inspiration and going out on expiration. If spasm of the bronchial tubes is present, the clinician may hear a wheeze suggestive of asthma. Fluid in the lungs produces a crackle, or rale, which is sometimes likened to the sound made by rubbing hair between two fingers. Louder variations of that sound are known as wheezes, or rhonchi. Rales and rhonchi suggest fluid, which may be caused by congestive heart failure or infection such as pneumonia. Sometimes sounds are heard that can be cleared by asking the patient to cough. The disappearance of adventitious sounds with coughing probably indicates a benign condition.

22 DIAGNOSTIC PROCEDURES

Monitoring the length of inspiration versus expiration can be helpful. Normal vesicular breathing produces a long inspiration and shorter expiration. Bronchial breathing has a prolonged expiration indicative of early obstructive changes.

The optometrist should be reminded that betablockers used for the topical treatment of glaucoma can exacerbate asthma. Some patients may be unaware that they have asthma. It is therefore recommended that the optometrist contact the patient’s physician to determine whether a pulmonary problem has been diagnosed before prescribing a betablocker.

EXAMINATION OF THE HEART

The most frequent cardiac complaints include chest pain, coughing, syncope, fatigue, palpitations, high blood pressure, dyspnea while lying flat or asleep, peripheral edema (fluid in the legs), and a history of heart attack or abnormal electrocardiograms (see Chapter 9). Before the advent of echocardiography, the clinical examination of the heart was the most elegant and most challenging in internal medicine. It was incumbent on the clinician to determine by clinical means whether a patient had a narrowing of the aortic valve, known as aortic stenosis, or a leaking valve causing aortic insufficiency or aortic regurgitation.

The clinician can hear systolic ejection sounds suggestive of an atrial septal defect, which is a hole in the superior aspect of the heart. The clinician might hear a click and a late systolic murmur associated with the most common valve abnormality, known as mitral valve prolapse.

The heart is examined in the context of its sounds, their radiation into the axilla or the neck, and the effect they have on the carotid pulse. Palpation is used to detect a displaced primary impulse, suggesting a change in the heart’s size. Palpation can also detect abnormal movement of the blood through the heart, known as a thrill. The heart is percussed to determine whether it is enlarged.

Auscultation is carefully performed by listening for murmurs indicative of heart valve dysfunction. These murmurs are described in terms of their intensity, duration, location, and transmission. Many murmurs, if not most, are physiologic or functional. A murmur is a sound indicative of a relative increase in blood flow through the valve opening, and it suggests either a narrowing, a leak, or simply a normal variant. Other heart sounds that indicate malfunction, such as S4 gallops, precede the first heart sound and are indicative of a noncompliant ventricle. An S3 gallop follows the second heart sound and indicates a dilated or poorly functioning heart muscle.

The echocardiogram uses sound waves to measure the heart sound, as well as the function of the valve. It establishes whether a disease is present.

It is important for the optometrist to realize that beta-blockers may exacerbate bradycardia and other significant heart conditions. Before the optometrist treats glaucoma with topical beta-blockers, the patient’s family physician should be consulted to verify that the drug will not exacerbate an underlying heart condition. Some heart conditions are treated with betablockers, and the patient may already be on such oral medications. Topical application of an additional betablocker may cause overdosage. To reduce the chance of systemic absorption of a topical beta-blocker, the patient must be taught the technique of punctal occlusion when an eye drop is applied.

EXAMINATION OF THE BREAST

Patients may have breast complaints, including lumps, discharge from the nipple, pain and tenderness, and abnormal findings from a self-examination. Breasts in both men and women should be examined in the routine medical examination. In men, palpation is used to detect the abnormal accumulation of breast tissue, known as gynecomastia, or the rare case of male breast cancer.

Examination of the female breasts involves visual inspection, followed by palpation. The skin of the breast is inspected for changes, lumps, retractions, and asymmetries. Palpation can determine whether dominant masses are present. Radiographs of the breasts (mammograms) or ultrasound, or both, are recommended to supplement the physical examination.

The breasts should be examined regularly, and a good physical examination should include education about the importance of monthly self-examination by the patient. Breast examination is not part of the routine optometric examination, and any patient with a breast complaint should be referred to a family physician or surgeon. A patient with a history of breast carcinoma requires a dilated fundus examination to rule out metastasis to the retina.

EXAMINATION OF THE ABDOMEN

Abdominal complaints include pain, nausea, vomiting, rectal bleeding, jaundice, and pruritus. The clinician should begin examination of the abdomen by inspecting for the obvious signs of obesity or muscular laxity. Scars, engorged veins, abdominal contours, and visible masses should be noted. The abdomen is then percussed to detect any enlargement of the liver or spleen, and any dullness suggestive of an underlying mass. The abdomen is then palpated, feeling for the liver’s edge on the right side of the body and the

spleen’s edge on the left. Sometimes abdominal palpation requires that the patient be placed in a left lateral position to ascertain enlargement. The examiner should palpate the midabdomen carefully, particularly in older patients, looking for signs of dilatation of the aorta, known as an aortic aneurysm.

The abdomen is then auscultated for bruits, which can indicate narrowing of the renal artery or the presence of atherosclerosis in the abdominal aorta. Auscultation is particularly important in the assessment of patients with unusual blood pressure elevations, because such elevations can be the result of narrowing, or stenosis, of the renal artery.

When abdominal distension is present, the clinician needs to look for the signs of ascites, which is fluid in the abdomen. Ascites can occur as a result of a generalized accumulation of fluid secondary to heart failure, liver failure, or tumors. Because fluid produces a change in the percussion sound, one clinical sign of ascites is a change in the distribution of dullness. The examiner can induce shifting of the dullness by placing a hand in the middle and tapping the side of the abdomen while turning the patient. In this way a fluid wave can be produced.

In addition to these physical signs, the clinician can use an ultrasound examination to confirm a clinical suspicion of ascites. The patient who is seen by the optometrist with symptoms of nausea, vomiting, and intestinal cramps, along with the ocular signs of uveitis, must be evaluated for inflammatory bowel disease (see Chapter 11).

EXAMINATION OF THE GENITOURINARY TRACT AND PELVIS

Typical pelvic complaints include pain, dysuria (pain on urination), incontinence, hematuria (blood in the urine), discharge, lesions, impotence, or dyspareunia (painful intercourse), infertility, and dysmenorrhea (painful menstruation).

A pelvic examination of the female patient begins with an inspection of the external structures for signs of abnormalities. The pelvic examination involves all of the physical examination skills and also a scraping of cells from the cervix, known as a Pap smear. Abnormal cervical cells may be the first sign of an underlying cervical or uterine malignancy. Next, a speculum is inserted into the vaginal canal to visualize the vaginal wall and cervix. The speculum is kept in place while a sample of the cervical and uterine cells are obtained for the Pap smear. The speculum is then removed.

Palpation is used to evaluate the size of the uterus, uterine irregularities, enlargement of the ovaries, or abnormal masses in the area surrounding the uterus. The clinician performs uterine palpation by inserting one finger into the vaginal canal while another finger

THE PHYSICAL EXAMINATION

23

deeply palpates the surface of the abdomen. A rectovaginal examination takes place to ascertain whether a fullness that is felt in the patient’s side is caused by the bowel or to the pelvic organs. A pelvic examination is critical for early detection of cervical, uterine, or ovarian cancer.

The male genitals are inspected for changes in size of the external genitalia, particularly the testicles, and for lesions on the scrotum or penis. Palpation of the testicles is essential for determining symmetry, changes in abnormal lumps or bumps, and the presence of hardness. Testicular tumors are common tumors occurring in men younger than 30 years. Although female breast examination and screening have been given considerable media attention, the need to instruct men in routine genital self-examination is often overlooked.

Another common abnormality of the male genitalia is the varicocele, which is an abnormal dilatation of the vein of the spermatic cord. Varicoceles are usually asymptomatic, but they are sometimes implicated in cases of subfertility. Enlargements of the epididymis, known as spermatoceles, also may be noted and may require corroboration by a urologic specialist.

Coughing or straining is necessary to elicit hernias, which result from breaks or discontinuities in the abdominal wall, allowing herniation of bowel. This condition requires surgical repair.

EXAMINATION OF THE RECTUM

Constipation, diarrhea, and abdominal pain are complaints that necessitate a detailed rectal exam. In male patients, the rectal examination includes observation and palpation to determine the size and consistency of the prostate and to detect any signs of prostate cancer, early polyps, or tumors of the rectal area.

Examination of the rectum is improved by the use of an endoscope. A small anoscope is used to look for rectal abnormalities. Rigid sigmoidoscopes allow examination of 25 cm of the bowel. A flexible sigmoidoscope may extend to 60 cm.

The detail with which one approaches the examination depends on the age of the patient. Significant risk factors for colon cancer include the age of the patient and a family history of colon cancer (see Chapter 11).

EXAMINATION OF THE MUSCULOSKELETAL SYSTEM

Musculoskeletal complaints include paresis (weakness) or paralysis, muscle stiffness, joint pain, limited movement, and a history of gout, arthritis, or deformity. The clinician must perform the orthopedic examination in a disciplined fashion, looking at every joint and assessing the integrity of the joint for both

24 DIAGNOSTIC PROCEDURES

disease and function. Inspect for signs of inflammation, swelling, warmth, and redness, and examine each joint through its full range of motion. Whether it be the shoulder, the elbow, the knee, or the hip, each joint has a specific range of functional capabilities. Restrictions in function and elicitation of pain are important findings to record. They may be indicative of disease intrinsic to the joint, such as degenerative change or rheumatoid arthritis, or extrinsic to the joint, involving the tendons, muscles, or ligaments that surround it. The term arthritis is often used to describe musculoskeletal pain, but that diagnosis is appropriate only when inflammation of a joint is involved (see Chapter 13).

The clinician begins by examining the small joints of the hands and feet and then moves to the shoulders, elbows, wrists, hips, knees, and ankles. Next, the neck, midback, and lower back are examined. The neck has a prescribed range of motion, which includes flexion forward, extension backward, lateral rotation to the right and left, and flexion to each side. The measurement of each movement should be recorded in degrees, and any limitation, pain, or spasm should be noted.

The midback is inspected for lateral curvature, known as scoliosis, palpated for pain, and guided through a routine set of movements that indicate whether function is normal.

The same examination is performed on the lower back. Lower back evaluation is complex because the most common back afflictions involve irritation to the nerves that emanate from the spine and continue down the leg. For example, patients with knee pain may have an intrinsic disease of the knee, such as arthritis, bursitis, tendinitis, or torn cartilage. Alternatively, the pain may reflect irritation of the nerves that supply the knee from the level of the back.

The spinal examination also involves palpation of the sacroiliac joint and the sciatic notch to rule out irritation of the sciatic nerve at the level of the back. Even the examination of the legs is directed at determining dysfunction in the back. This examination includes maneuvers such as straight-leg raising, reflex examination, and determination of strength and sensation in the independently innervated areas of the foot.

The dorsal web of the foot is supplied by the nerves of the L5-S1 spinal disc and the medial aspect of the foot by those of the L4-L5 disc. Examination of these is more appropriate to the neurologic examination, but they are essential in examination of the back as well.

The optometrist must rule out significant collagenvascular disease in all cases of anterior uveitis. Ankylosing spondylitis is typically found in a male uveitis patient, aged 20 to 40 years, with chronic lower back pain. A male patient in the same age group with arthritic joint pain and dysuria may have Reiter’s

syndrome. Young children who present with uveitis, particularly in a white, quiet eye, and who have posterior synechiae, cataract, and corneal edema or keratopathy, should be referred to a pediatric rheumatologist to rule out juvenile rheumatoid arthritis (see Chapters 13 and 21).

EXAMINATION OF THE LOWER EXTREMITIES

After the neurologic examination (see Chapter 2), the lower extremities are examined. The feet and legs are inspected for changes in color. A lack of hair growth may be indicative of poor arterial supply.

The pulses in the legs are assessed, starting with the femoral pulse in the groin, the popliteal pulse behind the knees, the posterior tibial pulse behind the ankle, and the dorsalis pedis pulse on the dorsum of the foot (approximately between the first and second toes).

Evaluation of arterial insufficiency in the legs is extremely important. To test the feet for swelling, place pressure on the skin and note whether a residual indentation remains, indicating peripheral edema. Palpate the calf for any abnormalities that might suggest deep vein thrombophlebitis.

In diabetics the skin of the feet should be inspected with special care, looking particularly between the toes for signs of cracking or fungal infection. The skin is also assessed for lesions, especially for evidence of dryness.

THE PHILOSOPHY OF THE PHYSICAL EXAMINATION

There are three categories of physical examination: the general physical, the focused examination, and the evaluation of a specific problem. The general physical provides an overall assessment of the patient. In the focused examination, a particular area of the body is inspected in great detail, and the entire examination is directed at that organ system or the related areas that might produce the patient’s chief symptom. This is true, for example, in the assessment of the common problem of back pain. In that case, the back is inspected in greater detail, and a careful neurologic examination is made of the lower extremity to determine possible nerve dysfunction emanating from the back. The third category of physical examination focuses on the evaluation of a specific medical problem, such as headache (see Chapter 18).

SUMMARY

Medical technology has overshadowed the physical examination for many physicians. The care and detail once given to the cardiac examination are sometimes

dismissed, and the patient is simply referred for an expensive echocardiogram. The painstaking detail of a neurologic assessment is replaced by a magnetic resonance scan. The importance of palpating the prostate to detect early prostatic carcinoma has at times been supplanted by a screening blood test known as a pros- tate-specific antigen.

The physical examination suggests whether or not medical technology should be applied. The careful physical examination is the essence of the doctor’s contribution to the patient’s medical care and the basis for subsequent treatment.

It is hoped that this chapter provides a useful introduction and guide, a basis for continued learning, and a new attitude of respect for and interest in the elegance and significance of the physical examination.

BIBLIOGRAPHY

Bates B: A guide to physical examination and history taking, ed 5, Philadelphia, 1991, JB Lippincott.

Braunwald E: Heart disease: a textbook of cardiovascular medicine, ed 4, Philadelphia, 1992,WB Saunders Co.

Burnside JW, McGlynn TJ: Physical diagnosis, ed 17, Baltimore, 1987,Williams & Wilkins.

Cassel El: Talking with patients: clinical technique, Cambridge, MA, MIT Press, 1985.

Cattau EL Jr, Benjamin SB, Knuff TE, et al: The accuracy of the physical examination in the diagnosis of suspected ascites, JAMA 247:1164, 1982.

THE PHYSICAL EXAMINATION

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D’Ambrosia RD: Musculoskeletal disorders: regional examination and differential diagnosis, ed 2, Philadelphia, 1986, JB Lippincott.

DeGowin R: DeGowin and DeGowin’s bedside diagnostic examination, ed 6, New York, 1994, MacMillan.

DeWeese DD: Otolaryngology: head and neck surgery, ed 8, St. Louis, 1993, Mosby-Year Book.

Fitzpatrick TB, Freeberg, IM: Dermatology in general medicine, New York, 1987, McGraw-Hill.

Fletcher RH, Fletcher SW: Has medicine outgrown physical diagnosis? (editorial), Ann Intern Med 117:786,1992.

Gorrol, May L, Mulley AG: Primary care medicine, Philadelphia, 1994, JB Lippincott.

Gorlin R, Zucker HD: Physician’s reaction to patients: a key to teaching humanistic medicine, N Engl JMed 308:1057, 1983.

Lookingbill DP, Marks JG Jr: Principles of dermatology, Philadelphia, 1986,WB Saunders Co.

Sackett DL: The science of the art of the clinical examination, JAMA 267:2650, 1992.

Schwartz MH: Textbook of physical diagnosis, history and examination, Philadelphia, 1989,WB Saunders Co.

Weinberger SE: Principles of pulmonary medicine, Philadelphia, 1992,WB Saunders Co.

Werner SC, Ingbar SCH, editors: The thyroid: a fundamental and clinical text, New York, 1991, Harper and Row.

Wilson JM, Foster DW: Williams’ textbook of endocrinology,

Philadelphia, 1992,WB Saunders Co.

C H A P T E R 3

The Neurologic Examination

C H A P T E R O U T L I N E

THE PRINCIPLES OF NEUROLOGIC DIAGNOSIS

Third Cranial Nerve—Oculomotor Nerve

Symptoms Without Clinical Signs

Fourth Cranial Nerve—Trochlear Nerve

Visual Defects With No Obvious Neurologic

Fifth Cranial Nerve—Trigeminal Nerve

Problems

Sixth Cranial Nerve—Abducens Nerve

 

Ocular and Neurologic Defects

Seventh Cranial Nerve—Facial Nerve

 

THE NEUROLOGIC SCREENING

Eighth Cranial Nerve—Vestibulocochlear Nerve

 

Evaluation of Mental Status

Ninth Cranial Nerve—Glossopharyngeal Nerve

 

Evaluation of Motor Functions

Tenth Cranial Nerve—Vagus Nerve

 

Evaluation of Sensory Functions

Eleventh Cranial Nerve—Accessory Nerve

 

Evaluation of Cerebellar Functions

Twelfth Cranial Nerve—Hypoglossal Nerve

 

Evaluation of Reflexes

 

CRANIAL NERVE TESTING

 

First Cranial Nerve—Olfactory Nerve

 

Second Cranial Nerve—Optic Nerve

 

 

 

The visual pathway from the retina to the occipital lobe is so lengthy that intracranial lesions are likely to affect some part of the functional visual system. The

key to diagnosis of the location and nature of such a lesion is the determination of its effect on the visual system and on other neurologic structures. The site of the lesion can be inferred from neurologic deficits identified by the practitioner. The lesion’s location is confirmed by radiography, computed tomography (CT scan), or magnetic resonance (MR) imaging (see Chapter 5). Finally, the nature of the lesion is tentatively deduced from the radiographic results along with the patient’s history, symptoms, and clinical signs.

This chapter describes a basic neurologic evaluation for the optometrist. The chapter first describes the evaluation of the patient’s mental status, reflexes, sensory and motor functions, and cerebellar functions. The remainder of the chapter is devoted to evaluation of the cranial nerves.

THE PRINCIPLES OF NEUROLOGIC DIAGNOSIS

Patients may be seen by the optometrist with no complaints at all or with ocular or neurologic symptoms. Neurologic symptoms are clues that help the practitioner establish the presence of a lesion; these symptoms must be differentiated from a psychogenic problem or malingering.

The eye examination may reveal the presence of a visual pathway disturbance as evidenced by a decrease in visual acuity, pupillary defects, visual field changes on perimetry testing, color vision distortions, or optic nerve head problems.

Whether or not a defect is discovered in the visual pathway, a neurologic screening should be performed on every patient. If no ocular or neurologic deficits are discovered, the optometrist must determine the cause of any symptoms. If no symptoms or signs are present,

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