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MSC - F. Surgery Answers 2025

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o Tenesmus (feeling of incomplete defecation).

oMucus in stool (rectal inflammation).

Systemic signs: Fever, malaise, chills.

Physical Examination

Abdominal exam:

oSuprapubic tenderness (may mimic cystitis).

Digital Rectal Exam (DRE):

oBulging, tender anterior rectal wall (key diagnostic sign).

Vaginal Exam (in women):

oTenderness in posterior fornix.

Diagnosis

1. Laboratory Findings:

o Leukocytosis (↑ WBC with left shift).

o↑ CRP, ESR.

2.Imaging:

oUltrasound (Transabdominal/Transrectal):

Hypoechoic fluid collection in the pelvis.

oCT (Gold Standard):

Fluid collection with rim enhancement in the rectovesical/rectouterine pouch.

oMRI (if CT unavailable or for better soft-tissue detail).

3.Diagnostic Puncture:

oTransrectal or transvaginal US-guided aspiration pus confirms abscess.

Treatment

1.Antibiotic Therapy

Empirical IV antibiotics (cover enteric Gram-negatives + anaerobes): o Ceftriaxone + Metronidazole (or Piperacillin-Tazobactam).

Adjust based on culture results.

2.Drainage (Definitive Treatment)

Indications: Abscess >3 cm or failed medical therapy.

Methods:

o Transrectal Drainage (most common):

US-guided needle puncture → insertion of drainage catheter.

oTransvaginal Drainage (in women):

Via posterior fornix puncture.

oPercutaneous CT-guided Drainage (if other routes inaccessible).

3.Surgical Drainage (Rarely Needed)

Laparoscopic/open drainage if:

oMultiloculated abscess.

oFailure of minimally invasive methods.

4.Delayed Appendectomy

Elective appendectomy after 6–8 weeks (if appendix not removed initially).

Complications

Sepsis (if untreated).

Fistula formation (rectal, vaginal, or bladder).

Recurrent abscess (incomplete drainage).

Key Points

1.Suspect pelvic abscess in appendicitis patients with persistent fever, rectal pain, or urinary symptoms.

2.DRE is crucial (bulging, tender anterior rectal wall).

3.CT confirms diagnosis; US-guided drainage is treatment of choice.

4.Antibiotics alone are insufficient if abscess >3 cm.

10.Surgical approaches for acute appendicitis. Indications and technique of retrograde appendectomy.

3. Differential Diagnosis

Acute appendicitis must be distinguished from:

1.Acute cholecystitis (RUQ pain, Murphy’s sign +).

2.Acute pancreatitis (epigastric pain, elevated amylase/lipase).

3.Perforated peptic ulcer (sudden severe pain, rigid abdomen).

4.Diverticulitis (left-sided in elderly, but right-sided cecal diverticulitis mimics appendicitis).

5.Renal colic (flank pain, hematuria).

6.Gynecological causes (ectopic pregnancy, ovarian torsion, PID).

Indications

Retrograde appendectomy is performed when:

1.The appendix is fixed to the retroperitoneum (severe adhesions, no mesentery).

2.Unable to mobilize the appendix into the surgical field (e.g., retrocecal position).

3.High risk of injury if attempting conventional antegrade removal.

Surgical Technique (Step-by-Step)

1.Access (Incision)

Volkovich-Dyakonov-McBurney incision (oblique, muscle-splitting in RLQ).

Alternative: Laparoscopic approach (if feasible).

2.Layer-by-Layer Dissection

Open external oblique aponeurosis, split internal oblique & transversus muscles.

Incise peritoneum to enter the abdominal cavity.

3.Identify & Expose the Cecum

Deliver the cecum into the wound.

Locate the base of the appendix (may require adhesiolysis).

4.Place Purse-String Suture

3-0 absorbable suture (e.g., Vicryl) around the appendix base on the cecum.

5.Ligate & Divide the Appendix Base

Ligate the base with absorbable suture (e.g., Catgut/Vicryl).

Clamp with Kocher forceps distal to the ligature.

Transect the appendix (leave a short stump).

Disinfect stump with iodine/povidone.

6.Invert the Stump

Tighten the purse-string suture to bury the stump.

Reinforce with a Z-stitch (Lembert suture) for secure closure.

7.Ligate the Mesoappendix (Retrograde)

Grasp the distal appendix with a clamp and apply traction.

Serially clamp, ligate, and divide the mesentery from base to tip.

8.Final Steps

Check hemostasis.

Irrigate the surgical site (if infection suspected).

Close the abdomen in layers:

o Peritoneum → muscle → fascia → skin.

Key Points

Advantage: Safer when the appendix is immobile or retroperitoneal.

Critical Steps:

Secure base ligation to prevent fecal fistula.

Complete mesentery ligation to avoid bleeding.

Post-Op: Monitor for stump leak or abscess (rare but serious).

11. Differential diagnosis of acute appendicitis and perforated gastric ulcer.

Key Clinical Differences

Feature

Acute Appendicitis

Perforated Gastric Ulcer

 

 

 

Onset of Pain

Gradual, starts periumbilical

Sudden, severe "dagger-

→ migrates to RLQ

like" pain in epigastrium

 

 

 

 

 

Maximal tenderness

Epigastric → may spread

Localization

to RLQ due to fluid

in RLQ (McBurney’s point)

 

tracking

 

 

 

 

 

Abdominal

Localized to RLQ (early) →

Board-like rigidity (entire

Rigidity

generalized (late)

abdomen)

 

 

 

Peritoneal Signs

+ Blumberg’s sign (RLQ)

+ Blumberg’s sign

(epigastrium → diffuse)

 

 

 

 

 

Temperature/Pulse

Low-grade fever (37.5–

Normal temp early (later

 

 

 

 

Feature

Acute Appendicitis

Perforated Gastric Ulcer

 

 

 

 

 

38.5°C), ↑ pulse

fever if peritonitis)

 

 

 

 

Vomiting

1–2 episodes (reflexive)

Rare (unless gastric outlet

obstruction)

 

 

 

 

 

 

 

 

"Ulcerative

History

No prior ulcer history

history" (chronic

 

 

dyspepsia, melena)

 

 

 

 

Imaging Findings

- Thickened appendix, fat

- Free air under

stranding (CT/US)

diaphragm (X-ray/CT)

 

 

 

 

 

Laboratory

Leukocytosis (12,000–

Normal WBC early (later

18,000/µL)

leukocytosis)

 

 

 

 

 

 

 

 

 

Critical Diagnostic Clues

1. Pain Migration

o Appendicitis: Periumbilical → RLQ (Kocher’s sign).

oPerforated ulcer: Epigastric → diffuse (due to chemical peritonitis).

2.Abdominal Rigidity

oAppendicitis: Localized RLQ guarding.

oPerforated ulcer: Global rigidity (entire abdomen "like a board").

3.Imaging

oX-ray/CT: Free subdiaphragmatic air (pathognomonic for perforation).

oUltrasound: RLQ appendix dilation (>6 mm), periappendiceal fluid.

4.Ancillary Signs

o Obturator/Psoas signs (appendicitis).

o Liver dullness loss (perforation, but late sign).

Pitfalls & Mimics

Late Presentation: Both conditions → generalized peritonitis (obscures origin).

Atypical Anatomy:

o Retrocecal appendicitis → flank/back pain (mimics renal colic).

oPelvic appendix → urinary symptoms (dysuria, tenesmus).

Perforated Duodenal Ulcer: Fluid tracks to RLQ (mimics appendicitis).

Management Approach

1.Uncertain Diagnosis CT abdomen/pelvis (gold standard).

2.Surgical Planning:

o Appendicitis: RLQ incision (McBurney) or laparoscopy.

oPerforated ulcer: Upper midline laparotomy (for repair + washout).

3.Intraoperative Surprise:

oIf RLQ incision reveals no appendicitis → convert to midline laparotomy.

Takeaway

Sudden epigastric pain + rigidity → Think perforation (image for free air).

Migrating RLQ pain + fever → Think appendicitis.

Always consider ulcer perforation in RLQ peritonitis (especially with prior dyspepsia).

External abdominal hernias

1. External abdominal hernias. The concept, elements of hernia and classification.

Definition

An external abdominal hernia is the abnormal protrusion of intra-abdominal organs (covered by parietal peritoneum) through a weak spot in the abdominal or pelvic wall.

Internal hernias occur when organs enter peritoneal recesses (e.g., paraduodenal) or the thoracic cavity (e.g., diaphragmatic hernia).

Elements of a Hernia

1. Hernial Orifice

oThe defect in the musculoaponeurotic layer (e.g., inguinal canal, femoral ring).

2.Hernial Sac

oA pouch of parietal peritoneum protruding through the orifice.

oParts:

Neck (narrow part at the orifice).

Body (main expanded portion).

Fundus (distal end).

3.Hernial Contents

oCommon: Omentum, small bowel loops.

oSliding Hernia: Organs partially covered by peritoneum (e.g., bladder, cecum, ascending colon).

Classification of External Abdominal Hernias

1. By Anatomical Location

Type

Description

Gender Predilection

 

 

 

Inguinal

Through inguinal canal (indirect/direct)

♂ > ♀ (10:3)

 

 

 

Femoral

Below inguinal ligament (femoral ring)

♀ > ♂ (8:1)

 

 

 

Umbilical

Through umbilical ring

♀ > ♂ (10:1)

 

 

 

Epigastric

Midline above umbilicus (linea alba)

♂ > ♀

 

 

 

Spigelian

Along semilunar line (lateral rectus sheath)

Rare

 

 

 

Lumbar

Through lumbar triangles (Grynfelt/Lesgaft)

♂ > ♀

 

 

 

Obturator

Through obturator canal (pelvic floor)

♀ > ♂

 

 

 

Sciatic

Through greater/lesser sciatic foramen

Rare

 

 

 

Incisional

At site of prior surgery

♀ > ♂ (4:1)

2.By Etiology

Congenital (e.g., patent processus vaginalis → indirect inguinal hernia).

Acquired (e.g., obesity, aging, trauma).

3.By Clinical Status

Type Characteristics

Reducible Hernia contents can be manually pushed back.

Irreducible Contents cannot be reduced (no strangulation).

Obstructed Bowel obstruction (no ischemia).

Type Characteristics

Strangulated Ischemic necrosis (surgical emergency!).

Incarcerated Irreducible + tender (early strangulation risk).

2. Etiopathogenesis of external abdominal hernias and their prevention.

Core Concept

Hernias develop due to an imbalance between intra-abdominal pressure (IAP) and abdominal wall resistance.

Etiopathogenesis

1. Weakness of the Abdominal Wall

A.Congenital Weakness

Persistent processus vaginalis → Indirect inguinal hernia (males).

Patent canal of Nuck → Indirect inguinal hernia (females).

Incomplete umbilical ring closure → Infantile umbilical hernia.

B.Acquired Weakness

Muscle atrophy (aging, malnutrition, cachexia).

Obesity (fat infiltration weakens muscle fibers).

Post-surgical (nerve injury, poor wound healing → incisional hernia).

Pregnancy (repeated stretching → diastasis recti, umbilical hernia).

2. Increased Intra-Abdominal Pressure (IAP)

Cause

Example

Chronic cough

COPD, tuberculosis, whooping cough (kids).