
MSC - F. Surgery Answers 2025
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o Tenesmus (feeling of incomplete defecation).
oMucus in stool (rectal inflammation).
Systemic signs: Fever, malaise, chills.
Physical Examination
Abdominal exam:
oSuprapubic tenderness (may mimic cystitis).
Digital Rectal Exam (DRE):
oBulging, tender anterior rectal wall (key diagnostic sign).
Vaginal Exam (in women):
oTenderness in posterior fornix.
Diagnosis
1. Laboratory Findings:
o Leukocytosis (↑ WBC with left shift).
o↑ CRP, ESR.
2.Imaging:
oUltrasound (Transabdominal/Transrectal):
Hypoechoic fluid collection in the pelvis.
oCT (Gold Standard):
Fluid collection with rim enhancement in the rectovesical/rectouterine pouch.
oMRI (if CT unavailable or for better soft-tissue detail).
3.Diagnostic Puncture:
oTransrectal or transvaginal US-guided aspiration → pus confirms abscess.

Treatment
1.Antibiotic Therapy
Empirical IV antibiotics (cover enteric Gram-negatives + anaerobes): o Ceftriaxone + Metronidazole (or Piperacillin-Tazobactam).
Adjust based on culture results.
2.Drainage (Definitive Treatment)
Indications: Abscess >3 cm or failed medical therapy.
Methods:
o Transrectal Drainage (most common):
US-guided needle puncture → insertion of drainage catheter.
oTransvaginal Drainage (in women):
Via posterior fornix puncture.
oPercutaneous CT-guided Drainage (if other routes inaccessible).
3.Surgical Drainage (Rarely Needed)
Laparoscopic/open drainage if:
oMultiloculated abscess.
oFailure of minimally invasive methods.
4.Delayed Appendectomy
Elective appendectomy after 6–8 weeks (if appendix not removed initially).
Complications
Sepsis (if untreated).
Fistula formation (rectal, vaginal, or bladder).
Recurrent abscess (incomplete drainage).

Key Points
1.Suspect pelvic abscess in appendicitis patients with persistent fever, rectal pain, or urinary symptoms.
2.DRE is crucial (bulging, tender anterior rectal wall).
3.CT confirms diagnosis; US-guided drainage is treatment of choice.
4.Antibiotics alone are insufficient if abscess >3 cm.
10.Surgical approaches for acute appendicitis. Indications and technique of retrograde appendectomy.
3. Differential Diagnosis
Acute appendicitis must be distinguished from:
1.Acute cholecystitis (RUQ pain, Murphy’s sign +).
2.Acute pancreatitis (epigastric pain, elevated amylase/lipase).
3.Perforated peptic ulcer (sudden severe pain, rigid abdomen).
4.Diverticulitis (left-sided in elderly, but right-sided cecal diverticulitis mimics appendicitis).
5.Renal colic (flank pain, hematuria).
6.Gynecological causes (ectopic pregnancy, ovarian torsion, PID).
Indications
Retrograde appendectomy is performed when:
1.The appendix is fixed to the retroperitoneum (severe adhesions, no mesentery).
2.Unable to mobilize the appendix into the surgical field (e.g., retrocecal position).
3.High risk of injury if attempting conventional antegrade removal.
Surgical Technique (Step-by-Step)

1.Access (Incision)
Volkovich-Dyakonov-McBurney incision (oblique, muscle-splitting in RLQ).
Alternative: Laparoscopic approach (if feasible).
2.Layer-by-Layer Dissection
Open external oblique aponeurosis, split internal oblique & transversus muscles.
Incise peritoneum to enter the abdominal cavity.
3.Identify & Expose the Cecum
Deliver the cecum into the wound.
Locate the base of the appendix (may require adhesiolysis).
4.Place Purse-String Suture
3-0 absorbable suture (e.g., Vicryl) around the appendix base on the cecum.
5.Ligate & Divide the Appendix Base
Ligate the base with absorbable suture (e.g., Catgut/Vicryl).
Clamp with Kocher forceps distal to the ligature.
Transect the appendix (leave a short stump).
Disinfect stump with iodine/povidone.
6.Invert the Stump
Tighten the purse-string suture to bury the stump.
Reinforce with a Z-stitch (Lembert suture) for secure closure.
7.Ligate the Mesoappendix (Retrograde)
Grasp the distal appendix with a clamp and apply traction.
Serially clamp, ligate, and divide the mesentery from base to tip.
8.Final Steps
Check hemostasis.

Irrigate the surgical site (if infection suspected).
Close the abdomen in layers:
o Peritoneum → muscle → fascia → skin.
Key Points
Advantage: Safer when the appendix is immobile or retroperitoneal.
Critical Steps:
Secure base ligation to prevent fecal fistula.
Complete mesentery ligation to avoid bleeding.
Post-Op: Monitor for stump leak or abscess (rare but serious).
11. Differential diagnosis of acute appendicitis and perforated gastric ulcer.
Key Clinical Differences
Feature |
Acute Appendicitis |
Perforated Gastric Ulcer |
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Onset of Pain |
Gradual, starts periumbilical |
Sudden, severe "dagger- |
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→ migrates to RLQ |
like" pain in epigastrium |
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Maximal tenderness |
Epigastric → may spread |
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Localization |
to RLQ due to fluid |
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in RLQ (McBurney’s point) |
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tracking |
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Abdominal |
Localized to RLQ (early) → |
Board-like rigidity (entire |
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Rigidity |
generalized (late) |
abdomen) |
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Peritoneal Signs |
+ Blumberg’s sign (RLQ) |
+ Blumberg’s sign |
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(epigastrium → diffuse) |
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Temperature/Pulse |
Low-grade fever (37.5– |
Normal temp early (later |
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Feature |
Acute Appendicitis |
Perforated Gastric Ulcer |
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38.5°C), ↑ pulse |
fever if peritonitis) |
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Vomiting |
1–2 episodes (reflexive) |
Rare (unless gastric outlet |
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obstruction) |
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"Ulcerative |
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History |
No prior ulcer history |
history" (chronic |
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dyspepsia, melena) |
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Imaging Findings |
- Thickened appendix, fat |
- Free air under |
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stranding (CT/US) |
diaphragm (X-ray/CT) |
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Laboratory |
Leukocytosis (12,000– |
Normal WBC early (later |
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18,000/µL) |
leukocytosis) |
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Critical Diagnostic Clues
1. Pain Migration
o Appendicitis: Periumbilical → RLQ (Kocher’s sign).
oPerforated ulcer: Epigastric → diffuse (due to chemical peritonitis).
2.Abdominal Rigidity
oAppendicitis: Localized RLQ guarding.
oPerforated ulcer: Global rigidity (entire abdomen "like a board").
3.Imaging
oX-ray/CT: Free subdiaphragmatic air (pathognomonic for perforation).
oUltrasound: RLQ appendix dilation (>6 mm), periappendiceal fluid.
4.Ancillary Signs

o Obturator/Psoas signs (appendicitis).
o Liver dullness loss (perforation, but late sign).
Pitfalls & Mimics
Late Presentation: Both conditions → generalized peritonitis (obscures origin).
Atypical Anatomy:
o Retrocecal appendicitis → flank/back pain (mimics renal colic).
oPelvic appendix → urinary symptoms (dysuria, tenesmus).
Perforated Duodenal Ulcer: Fluid tracks to RLQ (mimics appendicitis).
Management Approach
1.Uncertain Diagnosis → CT abdomen/pelvis (gold standard).
2.Surgical Planning:
o Appendicitis: RLQ incision (McBurney) or laparoscopy.
oPerforated ulcer: Upper midline laparotomy (for repair + washout).
3.Intraoperative Surprise:
oIf RLQ incision reveals no appendicitis → convert to midline laparotomy.
Takeaway
Sudden epigastric pain + rigidity → Think perforation (image for free air).
Migrating RLQ pain + fever → Think appendicitis.
Always consider ulcer perforation in RLQ peritonitis (especially with prior dyspepsia).

External abdominal hernias
1. External abdominal hernias. The concept, elements of hernia and classification.
Definition
An external abdominal hernia is the abnormal protrusion of intra-abdominal organs (covered by parietal peritoneum) through a weak spot in the abdominal or pelvic wall.
Internal hernias occur when organs enter peritoneal recesses (e.g., paraduodenal) or the thoracic cavity (e.g., diaphragmatic hernia).
Elements of a Hernia
1. Hernial Orifice
oThe defect in the musculoaponeurotic layer (e.g., inguinal canal, femoral ring).
2.Hernial Sac
oA pouch of parietal peritoneum protruding through the orifice.
oParts:
Neck (narrow part at the orifice).
Body (main expanded portion).
Fundus (distal end).
3.Hernial Contents
oCommon: Omentum, small bowel loops.
oSliding Hernia: Organs partially covered by peritoneum (e.g., bladder, cecum, ascending colon).
Classification of External Abdominal Hernias
1. By Anatomical Location

Type |
Description |
Gender Predilection |
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Inguinal |
Through inguinal canal (indirect/direct) |
♂ > ♀ (10:3) |
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Femoral |
Below inguinal ligament (femoral ring) |
♀ > ♂ (8:1) |
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Umbilical |
Through umbilical ring |
♀ > ♂ (10:1) |
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Epigastric |
Midline above umbilicus (linea alba) |
♂ > ♀ |
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Spigelian |
Along semilunar line (lateral rectus sheath) |
Rare |
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Lumbar |
Through lumbar triangles (Grynfelt/Lesgaft) |
♂ > ♀ |
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Obturator |
Through obturator canal (pelvic floor) |
♀ > ♂ |
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Sciatic |
Through greater/lesser sciatic foramen |
Rare |
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Incisional |
At site of prior surgery |
♀ > ♂ (4:1) |
2.By Etiology
Congenital (e.g., patent processus vaginalis → indirect inguinal hernia).
Acquired (e.g., obesity, aging, trauma).
3.By Clinical Status
Type Characteristics
Reducible Hernia contents can be manually pushed back.
Irreducible Contents cannot be reduced (no strangulation).
Obstructed Bowel obstruction (no ischemia).

Type Characteristics
Strangulated Ischemic necrosis (surgical emergency!).
Incarcerated Irreducible + tender (early strangulation risk).
2. Etiopathogenesis of external abdominal hernias and their prevention.
Core Concept
Hernias develop due to an imbalance between intra-abdominal pressure (IAP) and abdominal wall resistance.
Etiopathogenesis
1. Weakness of the Abdominal Wall
A.Congenital Weakness
Persistent processus vaginalis → Indirect inguinal hernia (males).
Patent canal of Nuck → Indirect inguinal hernia (females).
Incomplete umbilical ring closure → Infantile umbilical hernia.
B.Acquired Weakness
Muscle atrophy (aging, malnutrition, cachexia).
Obesity (fat infiltration weakens muscle fibers).
Post-surgical (nerve injury, poor wound healing → incisional hernia).
Pregnancy (repeated stretching → diastasis recti, umbilical hernia).
2. Increased Intra-Abdominal Pressure (IAP)
Cause |
Example |
Chronic cough |
COPD, tuberculosis, whooping cough (kids). |
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