
MSC - F. Surgery Answers 2025
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oFailure of conservative management
12.Cicatricial strictures of the esophagus. Causes, clinic, diagnosis, treatment. Types of bougie of the esophagus. Indications for surgical treatment.
Cicatricial Strictures of the Esophagus: Brief Overview
Definition:
Narrowing of the esophagus due to scar tissue formation after injury or inflammation, leading to lumen constriction and deformation.
Causes:
Most commonly chemical burns (caustic ingestion).
Iatrogenic injury, foreign bodies, peptic esophagitis, post-infectious causes (e.g., diphtheria).
Localization:
Pharyngoesophageal junction (behind cricoid)
Aortic arch level
Diaphragmatic hiatus
Types:
Membranous vs. tubular strictures
Single vs. multiple
Complete vs. incomplete
Clinical Features:
Progressive dysphagia (solids → liquids)
Regurgitation, retrosternal pain

Weight loss, aspiration in high strictures
Diagnosis:
Contrast esophagography: shows stricture shape, length, and dilation above.
Esophagoscopy: visualizes stricture, assesses mucosa, allows biopsy if malignancy suspected.
Treatment:
Primary: Bougienage (dilatation) using progressively larger bougies starting ~7 weeks post-injury.
Bougie types: blind, esophagoscope-guided, radiopaque with guidewire, retrograde (via gastrostomy).
Additional methods: balloon dilation, electrical dissection, stenting (temporary arthroplasty).
Indications for Surgery:
Complete esophageal lumen obstruction
Failed or recurrent strictures after multiple dilations
Fistulas with trachea/bronchi
Severe long segment strictures or repeated perforations
Surgical Options:
Segmental strictures → Partial esophageal plastic surgery.
Extensive strictures → Total esophageal replacement (esophagoplasty or esophagectomy).
Graft materials:
o Small or large intestine segments.

oGastric tube formed from stomach’s greater curvature.
Graft routes:
oAnterior mediastinum (behind sternum).
o Intrapleural.
oPosterior mediastinum.
13.Achalasia of the cardia. Etiology, classification, clinic, diagnosis, conservative treatment.
Esophageal achalasia is a rare, chronic esophageal motility disorder marked by:
Absence of normal peristalsis.
Failure of the lower esophageal sphincter (LES) to relax during swallowing.
Etiology & Pathogenesis:
Likely due to degeneration of Auerbach’s plexus (myenteric plexus).
Contributing theories: viral infections, autoimmune, neuropsychiatric disturbances, or congenital neural defects.
Secondary achalasia may be due to malignancies (pseudoachalasia).
Clinical Features:
Dysphagia: Initially for solids, then liquids. Paradoxical dysphagia may occur (liquids harder to swallow than solids).
Regurgitation: Especially at night; may cause aspiration.
Retrosternal pain: From esophageal spasm or overdistension.
Weight loss, halitosis, belching, and cough.
Esophageal vomiting brings relief.
Long-standing disease leads to megaesophagus (gross dilation).

Classification:
Petrovsky’s Staging (I–IV):
o I: Functional spasm only.
o II: Minimal esophageal dilation.
o III: Significant dilation and muscular changes.
oIV: Severe dilation, tortuosity, complications (esophagitis, ulcers).
Types (Radiologic):
oType I: Mild dilation, muscle hypertrophy.
o Type II: Marked dilation, muscle atrophy, S-shaped esophagus.
Diagnosis:
1.Barium swallow: “Bird-beak” tapering of distal esophagus; proximal dilation; S-shaped esophagus.
2.Endoscopy: Rules out cancer (especially pseudoachalasia); biopsy if needed.
3.Manometry (Gold standard):
o Incomplete LES relaxation.
o Absent peristalsis.
oElevated LES pressure (not always).
4.Pharmacological tests (e.g., carbachol): Hyperresponsiveness indicates denervation.
1.Non-Surgical:
Balloon Pneumatic Dilatation (first-line for many):
o Serial dilations under fluoroscopy.
oRisk: perforation (~2%), reflux, bleeding.
Pharmacotherapy (limited, adjunctive):
oNitrates, calcium channel blockers, antispasmodics, anxiolytics.

oBotulinum toxin injection into LES in non-surgical candidates
3.Lifestyle & Support:
Small, frequent meals.
Elevated head during sleep.
Avoid meals 3–4 hours before bedtime.
Psychological support as needed.
14. Achalasia of the cardia. Classification, clinic, diagnosis, differential diagnosis, surgical treatment
Differential Diagnosis:
Esophageal strictures (caustic, post-inflammatory)
Tumors (esophageal/gastric cardia cancer)
Diverticula
Benign motility disorders
Surgical:
Heller Myotomy (laparoscopic preferred):
o Longitudinal cut of LES muscle fibers.
oOften combined with anti-reflux procedure (e.g., Dor fundoplication).
Indications:
oFailed dilatation.
oAdvanced stage (III-IV), children, or complications like esophageal rupture
Prognosis:
Generally good with treatment.
Risk of recurrence exists.
Long-term risk: esophageal squamous cell carcinoma due to chronic stasis and irritation — warrants long-term surveillance in high-risk patients.

15. Diaphragmatic hernias. Etiology, classification, diagnostic methods. Traumatic hernias of the diaphragm, clinic, diagnosis, treatment.
A diaphragmatic hernia is the protrusion of abdominal organs into the thoracic cavity through a defect in the diaphragm, which may be congenital, acquired, traumatic, or iatrogenic.
Etiology and Pathogenesis
Predisposing factors:
Congenital diaphragmatic defects (e.g., failure of pleuroperitoneal canal closure)
Connective tissue weakness (congenital or acquired)
Muscular atrophy due to aging
Iatrogenic (postoperative weakening)
Producing factors:
Increased intra-abdominal pressure due to:
o Heavy lifting
o Chronic cough
o Obesity
o Constipation
o Pregnancy
o Abundant meals
Classification
A.By Etiology
1.Congenital Diaphragmatic Hernias (CDH) o Bochdalek hernia (posterolateral)
o Morgagni hernia (retrosternal/parasternal)

oCentral tendon defects
2.Acquired (Non-traumatic)
oHiatal hernia (esophageal hiatus hernia)
oIatrogenic (surgical or procedural injury)
3.Traumatic
oBlunt trauma (e.g., MVCs, falls)
oPenetrating injuries (e.g., stab wounds)
B.By Presence of Hernial Sac
True hernia: with peritoneal sac
False hernia: without sac
C.By Anatomical Site (Petrovsky Classification)
False congenital hernias
True hernias of weak zones (e.g., Larrey’s triangle, Bochdalek’s triangle)
True hernias of atypical localization
Natural opening hernias (e.g., esophageal, caval, aortic hiatus)
Clinical Presentation
Symptoms result from mechanical compression, organ displacement, and/or infringement:
Gastrointestinal Symptoms
Epigastric or chest pain (worse after meals)
Postprandial vomiting (relieves symptoms)
Bloating, dysphagia
"Gurgling" sounds in the chest
Respiratory/Cardiovascular Symptoms
Dyspnea (especially postprandial or supine)

Palpitations
Mediastinal shift signs
Decreased breath sounds
Physical Examination
Decreased breath sounds over affected hemithorax
Tympanic note or dullness on percussion
Audible bowel sounds in chest
Scaphoid or sunken abdomen (large hernias)
Displaced apex beat or cardiac dullness
Diagnostic Methods
1.Chest X-ray (Initial)
Air-fluid levels in thoracic cavity
Mediastinal shift
Intestinal loops in chest
Absent diaphragmatic contour
2.Contrast Radiography
Barium swallow: visualizes herniation of stomach/intestine
"Flow sign" or "hernial gate" symptom
3.CT or MRI
Defines content and site of herniation
Rules out associated injuries in trauma
4.Endoscopy
Useful for evaluating sliding hiatal hernias

5.Diagnostic Pneumoperitoneum (rare)
Outlines the diaphragmatic contour for difficult cases
Complications
Strangulation/Incarceration (common and dangerous)
Bowel obstruction
Volvulus (especially stomach)
Respiratory compromise
Mediastinal shift → hemodynamic instability
Differential Diagnosis
Diaphragm relaxation (eventration)
Myocardial infarction
Acute pancreatitis
Mesenteric ischemia
Pericardial cyst
Lung cyst/tumor
Traumatic Diaphragmatic Hernias
Etiology
Penetrating or blunt trauma to the lower thoracoabdominal region
Often associated with polytrauma (e.g., spleen, liver, lung)
Clinical Features
Initially asymptomatic or misdiagnosed
Progressive dyspnea, chest pain, or gastrointestinal obstruction

Often found late due to delayed diagnosis
Diagnosis
Chest X-ray: Elevated hemidiaphragm, air-fluid levels
CT thorax and abdomen: diagnostic gold standard
Nasogastric tube coiling in chest (if stomach involved)
Laparoscopy/thoracoscopy: diagnostic and therapeutic
Treatment
Surgical repair is mandatory
o Laparotomy (preferred in acute trauma)
oThoracotomy (for chronic cases or large defects)
Hernia contents reduced
Diaphragmatic defect closed with non-absorbable sutures ± mesh
Treatment of Diaphragmatic Hernia (General)
Indications: All symptomatic or large hernias; most asymptomatic congenital hernias
Surgical Approaches:
Transthoracic (4th or 7th intercostal space)
Transabdominal (especially for Morgagni or hiatal hernia)
Laparoscopic or thoracoscopic repair in stable cases
Procedure:
Reduction of herniated viscera
Excision of hernial sac (if present)
Primary closure or use of synthetic mesh for large defects
Emergency Surgery: Needed for strangulated hernias with signs of obstruction or ischemia